UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Unequal crossing over between CYP11B2 and CYP11B1 causes 11 β -hydroxylase deficiency in a consanguineous family

The Journal of steroid biochemistry and molecular biology, 2023-10, Vol.233, p.106375-106375, Article 106375

2023

Volltextzugriff (PDF)

Blockade of a novel MAP4K4-LATS2-SASH1-YAP1 cascade inhibits tumorigenesis and metastasis in luminal breast cancer

The Journal of biological chemistry, 2024-06, Vol.300 (6), p.107309, Article 107309

2024

Volltextzugriff (PDF)

The circadian clock gene, BMAL1, promotes radiosensitization in nasopharyngeal carcinoma by inhibiting the epithelial-to-mesenchymal transition via the TGF-β1/Smads/Snail1 axis

Oral oncology, 2024-05, Vol.152, p.106798, Article 106798

2024

Volltextzugriff (PDF)

The PER3rs772027021 SNP induces pigmentation phenotypes of dyschromatosis universalis hereditaria

Journal of molecular medicine (Berlin, Germany), 2023-03, Vol.101 (3), p.279-294

2023

Volltextzugriff (PDF)

Association between platelets and in-hospital mortality in critically ill patients with tumours: a retrospective cohort study

BMJ open, 2022-04, Vol.12 (4), p.e053691-e053691

2022

Volltextzugriff (PDF)

Dickkopf-1 is an immune infiltration-related prognostic biomarker of head and neck squamous cell carcinoma

Aging (Albany, NY.), 2024-02, Vol.16 (4), p.3837-3855

2024

Volltextzugriff (PDF)

Circadian clock gene BMAL1 inhibits the proliferation and tumor-formation ability of nasopharyngeal carcinoma cells and increases the sensitivity of radiotherapy

Chronobiology international, 2022-10, Vol.39 (10), p.1340-1351

2022

Volltextzugriff (PDF)

Effects of biological clock gene BMAL1 and hypoxia-inducible factor HIF-1α on proliferation, migration and radiotherapy sensitivity of nasopharyngeal carcinoma cells HONE1

Holistic Integrative Oncology, 2023-07, Vol.2 (1), p.26, Article 26

2023

Volltextzugriff (PDF)

Retyping and molecular pathology diagnosis of dyschromatosis universalis hereditaria

Experimental dermatology, 2023-09, Vol.32 (9), p.1334-1343

2023

Volltextzugriff (PDF)

Aberrant production of soluble inducible T-cell co-stimulator (sICOS) and soluble programmed cell death protein 1 (sPD-1) in patients with chronic hepatitis C

Molecular medicine reports, 2013-04, Vol.7 (4), p.1197-1202

2013

Volltextzugriff (PDF)

A novel insertional mutation in the connexin 46 (gap junction alpha 3) gene associated with autosomal dominant congenital cataract in a Chinese family

Molecular vision, 2013-04, Vol.19, p.789-795

2013

Volltextzugriff (PDF)

A novel genomic rearrangement on chr19q13.42 leads to PRPF31‐associated retinitis pigmentosa

Clinical & experimental ophthalmology, 2024-07, Vol.52 (5), p.595-598

2024

Volltextzugriff (PDF)

Three mutations in SASH1 cause the pathogenesis of dyschromatosis universalis hereditaria (DUH)

Nature precedings, 2011-02

2011

Volltextzugriff (PDF)

Combination of gemcitabine and erlotinib inhibits recurrent pancreatic cancer growth in mice via the JAK-STAT pathway

Oncology reports, 2018-03, Vol.39 (3), p.1081-1089

2018

Volltextzugriff (PDF)

A novel P53/POMC/Gαs/SASH1 autoregulatory feedback loop activates mutated SASH1 to cause pathologic hyperpigmentation

Journal of cellular and molecular medicine, 2017-04, Vol.21 (4), p.802-815

2017

Volltextzugriff (PDF)

Mutated SASH1 promotes Mitf expression in a heterozygous mutated SASH1 knock‑in mouse model

International journal of molecular medicine, 2020-09, Vol.46 (3), p.1118-1134

2020

Volltextzugriff (PDF)

SASH1 regulates melanocyte transepithelial migration through a novel Gαs–SASH1–IQGAP1–E-Cadherin dependent pathway

Cellular signalling, 2013-06, Vol.25 (6), p.1526-1538

2013

Volltextzugriff (PDF)

p53 regulates ERK1/2/CREB cascade via a novel SASH1/MAP2K2 crosstalk to induce hyperpigmentation

Journal of cellular and molecular medicine, 2017-10, Vol.21 (10), p.2465-2480

2017

Volltextzugriff (PDF)

The PER3 rs772027021 SNP induces pigmentation phenotypes of dyschromatosis universalis hereditaria

Journal of molecular medicine (Berlin, Germany), 2023-03, Vol.101 (3), p.279

2023

Volltextzugriff (PDF)

p53 regulates ERK 1/2/ CREB cascade via a novel SASH 1/ MAP 2K2 crosstalk to induce hyperpigmentation

Journal of cellular and molecular medicine, 2017-10, Vol.21 (10), p.2465-2480

2017

Volltextzugriff (PDF)

A novel P53/ POMC /Gαs/ SASH 1 autoregulatory feedback loop activates mutated SASH 1 to cause pathologic hyperpigmentation

Journal of cellular and molecular medicine, 2017-04, Vol.21 (4), p.802-815

2017

Volltextzugriff (PDF)

SASH1 regulates melanocyte transepithelial migration through a novel GI-saSASH1aIQGAP1aE-Cadherin dependent pathway

Cellular signalling, 2013-06, Vol.25 (6), p.1526-1538

2013

Volltextzugriff (PDF)

Aspect ratio and interference effects on flow over two rectangular cylinders in tandem arrangement

Ocean engineering, 2023-03, Vol.271, p.113731, Article 113731

2023

Volltextzugriff (PDF)

Determination of Risk Perception of Drivers Using Fuzzy-Clustering Analysis for Road Safety

IEEE access, 2020, Vol.8, p.125501-125512

2020

Volltextzugriff (PDF)

Identifying hepatocellular carcinoma patients with survival benefits from surgery combined with chemotherapy: based on machine learning model

World journal of surgical oncology, 2022-12, Vol.20 (1), p.1-377, Article 377

2022

Volltextzugriff (PDF)

1 – 25 von 38

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(31)
Mit Volltextzugriff
(34)

Publikationsform

Artikel
(38)

Sprache

Englisch
(38)
Japanisch
(1)

Erscheinungsjahr

n.n

Thema

Science & Technology
(24)
Life Sciences & Biomedicine
(20)
Humans
(12)
Hyperpigmentation
(6)
Male
(6)
Mutation
(6)
Oncology
(6)
Female
(4)
Medicine, Research & Experimental
(4)
Research & Experimental Medicine
(4)
Tumor Suppressor Proteins - Genetics
(4)
Tumors
(4)
Animals
(3)
Blood Pressure
(3)
Cancer Therapies
(3)
Cell Biology
(3)
Driver Behavior
(3)
Feedback
(3)
Hyperpigmentation - Genetics
(3)
Machine Learning
(3)

15 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

Unequal crossing over between CYP11B2 and CYP11B1 causes 11 β -hydroxylase deficiency in a consanguineous family

Blockade of a novel MAP4K4-LATS2-SASH1-YAP1 cascade inhibits tumorigenesis and metastasis in luminal breast cancer

The circadian clock gene, BMAL1, promotes radiosensitization in nasopharyngeal carcinoma by inhibiting the epithelial-to-mesenchymal transition via the TGF-β1/Smads/Snail1 axis

The PER3rs772027021 SNP induces pigmentation phenotypes of dyschromatosis universalis hereditaria

Association between platelets and in-hospital mortality in critically ill patients with tumours: a retrospective cohort study

Dickkopf-1 is an immune infiltration-related prognostic biomarker of head and neck squamous cell carcinoma

Circadian clock gene BMAL1 inhibits the proliferation and tumor-formation ability of nasopharyngeal carcinoma cells and increases the sensitivity of radiotherapy

Effects of biological clock gene BMAL1 and hypoxia-inducible factor HIF-1α on proliferation, migration and radiotherapy sensitivity of nasopharyngeal carcinoma cells HONE1

Retyping and molecular pathology diagnosis of dyschromatosis universalis hereditaria

Aberrant production of soluble inducible T-cell co-stimulator (sICOS) and soluble programmed cell death protein 1 (sPD-1) in patients with chronic hepatitis C

A novel insertional mutation in the connexin 46 (gap junction alpha 3) gene associated with autosomal dominant congenital cataract in a Chinese family

A novel genomic rearrangement on chr19q13.42 leads to PRPF31‐associated retinitis pigmentosa

Three mutations in SASH1 cause the pathogenesis of dyschromatosis universalis hereditaria (DUH)

Combination of gemcitabine and erlotinib inhibits recurrent pancreatic cancer growth in mice via the JAK-STAT pathway

A novel P53/POMC/Gαs/SASH1 autoregulatory feedback loop activates mutated SASH1 to cause pathologic hyperpigmentation

Mutated SASH1 promotes Mitf expression in a heterozygous mutated SASH1 knock‑in mouse model

SASH1 regulates melanocyte transepithelial migration through a novel Gαs–SASH1–IQGAP1–E-Cadherin dependent pathway

p53 regulates ERK1/2/CREB cascade via a novel SASH1/MAP2K2 crosstalk to induce hyperpigmentation

The PER3 rs772027021 SNP induces pigmentation phenotypes of dyschromatosis universalis hereditaria

p53 regulates ERK 1/2/ CREB cascade via a novel SASH 1/ MAP 2K2 crosstalk to induce hyperpigmentation

A novel P53/ POMC /Gαs/ SASH 1 autoregulatory feedback loop activates mutated SASH 1 to cause pathologic hyperpigmentation

SASH1 regulates melanocyte transepithelial migration through a novel GI-saSASH1aIQGAP1aE-Cadherin dependent pathway

Aspect ratio and interference effects on flow over two rectangular cylinders in tandem arrangement

Determination of Risk Perception of Drivers Using Fuzzy-Clustering Analysis for Road Safety

Identifying hepatocellular carcinoma patients with survival benefits from surgery combined with chemotherapy: based on machine learning model

Suchergebnisse filtern