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Deficiency in the repair of DNA damage by homologous recombination and sensitivity to poly(ADP-ribose) polymerase inhibition

Cancer research (Chicago, Ill.), 2006-08, Vol.66 (16), p.8109-8115

2006

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The DNA helicase BRIP1 is defective in Fanconi anemia complementation group J

Nature genetics, 2005-09, Vol.37 (9), p.934-935

2005

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Warsaw Breakage Syndrome, a Cohesinopathy Associated with Mutations in the XPD Helicase Family Member DDX11/ChlR1

American journal of human genetics, 2010-02, Vol.86 (2), p.262-266

2010

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FANCD1/BRCA2 plays predominant role in the repair of DNA damage induced by ACNU or TMZ

PloS one, 2011-05, Vol.6 (5), p.e19659

2011

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The XRCC genes: expanding roles in DNA double-strand break repair

DNA Repair, 2004-08, Vol.3 (8), p.1081-1090

2004

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Repair pathways independent of the Fanconi anemia nuclear core complex play a predominant role in mitigating formaldehyde-induced DNA damage

Biochemical and biophysical research communications, 2011-01, Vol.404 (1), p.206-210

2011

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Suppression of the DNA Repair Defects of BRCA2-Deficient Cells with Heterologous Protein Fusions

Proceedings of the National Academy of Sciences - PNAS, 2006-06, Vol.103 (23), p.8768-8773

2006

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Brca2 (XRCC11) Deficiency Results in Radioresistant DNA Synthesis and a Higher Frequency of Spontaneous Deletions

Molecular and Cellular Biology, 2002-01, Vol.22 (2), p.669-679

2002

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Inducibility of nuclear Rad51 foci after DNA damage distinguishes all Fanconi anemia complementation groups from D1/BRCA2

Mutation research, 2006-02, Vol.594 (1), p.39-48

2006

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Artesunate derived from traditional Chinese medicine induces DNA damage and repair

Cancer research (Chicago, Ill.), 2008-06, Vol.68 (11), p.4347-4351

2008

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Cellular characterization of cells from the Fanconi anemia complementation group, FA-D1/BRCA2

Mutation research, 2006-10, Vol.601 (1), p.191-201

2006

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The role of the Fanconi anemia pathway in DNA repair and maintenance of genome stability

Postȩpy higieny i medycyny doświadczalnej, 2014-05, Vol.68, p.459-472

2014

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Synthetic lethal targeting of DNA double-strand break repair deficient cells by human apurinic/apyrimidinic endonuclease inhibitors

International journal of cancer, 2012-11, Vol.131 (10), p.2433-2444

2012

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Chinese hamster cell mutant, V-C8, a model for analysis of Brca2 function

Mutation research, 2006-08, Vol.600 (1), p.79-88

2006

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Warsaw Breakage Syndrome, a Cohesinopathy Associated with Mutations in the XPD Helicase Family Member DDX11/ChIR1

American journal of human genetics, 2010-02, Vol.86 (2), p.262-266

2010

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Brca2/Xrcc2 dependent HR, but not NHEJ, is required for protection against O 6-methylguanine triggered apoptosis, DSBs and chromosomal aberrations by a process leading to SCEs

DNA repair, 2009, Vol.8 (1), p.72-86

2009

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The mammalian XRCC genes: their roles in DNA repair and genetic stability

DNA Repair, 2003-06, Vol.2 (6), p.655-672

2003

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Functional link between ataxia-telangiectasia and Nijmegen breakage syndrome gene products

Nature (London), 2000-05, Vol.405 (6785), p.473-477

2000

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homologous recombination defect affects replication-fork progression in mammalian cells

Journal of cell science, 2008-01, Vol.121 (2), p.162-166

2008

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Different types of V(D)J recombination and end‐joining defects in DNA double‐strand break repair mutant mammalian cells

European journal of immunology, 2002-03, Vol.32 (3), p.701-709

2002

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Fanconi Anemia Complementation Group D2 (FANCD2) Functions Independently of BRCA2- and RAD51-associated Homologous Recombination in Response to DNA Damage

The Journal of biological chemistry, 2005-04, Vol.280 (15), p.14877-14883

2005

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Correction: FANCD1/BRCA2 Plays Predominant Role in the Repair of DNA Damage Induced by ACNU or TMZ

PloS one, 2011-06, Vol.6 (6)

2011

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Impaired DNA damage-induced nuclear Rad51 foci formation uniquely characterizes Fanconi anemia group D1

Oncogene, 2002-07, Vol.21 (32), p.5002-5005

2002

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Ionizing radiation-induced foci formation of mammalian Rad51 and Rad54 depends on the Rad51 paralogs, but not on Rad52

Mutation Research-Fundamental and Molecular Mechanisms of Mutagenesis, 2005-07, Vol.574 (1), p.34-49

2005

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Rad51C is essential for embryonic development and haploinsufficiency causes increased DNA damage sensitivity and genomic instability

Mutation research, 2010-07, Vol.689 (1), p.50-58

2010

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Deficiency in the repair of DNA damage by homologous recombination and sensitivity to poly(ADP-ribose) polymerase inhibition

The DNA helicase BRIP1 is defective in Fanconi anemia complementation group J

Warsaw Breakage Syndrome, a Cohesinopathy Associated with Mutations in the XPD Helicase Family Member DDX11/ChlR1

FANCD1/BRCA2 plays predominant role in the repair of DNA damage induced by ACNU or TMZ

The XRCC genes: expanding roles in DNA double-strand break repair

Repair pathways independent of the Fanconi anemia nuclear core complex play a predominant role in mitigating formaldehyde-induced DNA damage

Suppression of the DNA Repair Defects of BRCA2-Deficient Cells with Heterologous Protein Fusions

Brca2 (XRCC11) Deficiency Results in Radioresistant DNA Synthesis and a Higher Frequency of Spontaneous Deletions

Inducibility of nuclear Rad51 foci after DNA damage distinguishes all Fanconi anemia complementation groups from D1/BRCA2

Artesunate derived from traditional Chinese medicine induces DNA damage and repair

Cellular characterization of cells from the Fanconi anemia complementation group, FA-D1/BRCA2

The role of the Fanconi anemia pathway in DNA repair and maintenance of genome stability

Synthetic lethal targeting of DNA double-strand break repair deficient cells by human apurinic/apyrimidinic endonuclease inhibitors

Chinese hamster cell mutant, V-C8, a model for analysis of Brca2 function

Warsaw Breakage Syndrome, a Cohesinopathy Associated with Mutations in the XPD Helicase Family Member DDX11/ChIR1

Brca2/Xrcc2 dependent HR, but not NHEJ, is required for protection against O 6-methylguanine triggered apoptosis, DSBs and chromosomal aberrations by a process leading to SCEs

The mammalian XRCC genes: their roles in DNA repair and genetic stability

Functional link between ataxia-telangiectasia and Nijmegen breakage syndrome gene products

homologous recombination defect affects replication-fork progression in mammalian cells

Different types of V(D)J recombination and end‐joining defects in DNA double‐strand break repair mutant mammalian cells

Fanconi Anemia Complementation Group D2 (FANCD2) Functions Independently of BRCA2- and RAD51-associated Homologous Recombination in Response to DNA Damage

Correction: FANCD1/BRCA2 Plays Predominant Role in the Repair of DNA Damage Induced by ACNU or TMZ

Impaired DNA damage-induced nuclear Rad51 foci formation uniquely characterizes Fanconi anemia group D1

Ionizing radiation-induced foci formation of mammalian Rad51 and Rad54 depends on the Rad51 paralogs, but not on Rad52

Rad51C is essential for embryonic development and haploinsufficiency causes increased DNA damage sensitivity and genomic instability

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