UB Paderborn / Katalog / Suche / in CDI

1 – 25 von 392

Sortiert nach

Neuregulin-1/ErbB4 Signaling Regulates Visual Cortical Plasticity

Neuron (Cambridge, Mass.), 2016-10, Vol.92 (1), p.160-173

2016

Volltextzugriff (PDF)

The Antinociceptive Properties of the Corydalis yanhusuo Extract

PloS one, 2016-09, Vol.11 (9), p.e0162875-e0162875

2016

Volltextzugriff (PDF)

Epileptiform activity and behavioral arrests in mice overexpressing the calcium channel subunit α2δ-1

Neurobiology of disease, 2017-06, Vol.102, p.70-80

2017

Volltextzugriff (PDF)

Calcium channel alpha-2-delta-1 protein upregulation in dorsal spinal cord mediates spinal cord injury-induced neuropathic pain states

Pain (Amsterdam), 2011-03, Vol.152 (3), p.649-655

2011

Volltextzugriff (PDF)

A Novel Analgesic Isolated from a Traditional Chinese Medicine

Current biology, 2014-01, Vol.24 (2), p.117-123

2014

Volltextzugriff (PDF)

Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states

The Journal of neuroscience, 2012-06, Vol.32 (26), p.8977-8987

2012

Volltextzugriff (PDF)

Profiling of dynamically changed gene expression in dorsal root ganglia post peripheral nerve injury and a critical role of injury-induced glial fibrillary acetic protein in maintenance of pain behaviors

Pain (Amsterdam), 2009-05, Vol.143 (1), p.114-122

2009

Volltextzugriff (PDF)

Central Mechanisms Mediating Thrombospondin-4-induced Pain States

The Journal of biological chemistry, 2016-06, Vol.291 (25), p.13335-13348

2016

Volltextzugriff (PDF)

The EGF-LIKE domain of thrombospondin-4 is a key determinant in the development of pain states due to increased excitatory synaptogenesis

The Journal of biological chemistry, 2018-10, Vol.293 (42), p.16453-16463

2018

Volltextzugriff (PDF)

Calcium channel functions in pain processing

Channels (Austin, Tex.), 2010-11, Vol.4 (6), p.510-517

2010

Volltextzugriff (PDF)

Thrombospondin-4 divergently regulates voltage-gated Ca2+ channel subtypes in sensory neurons after nerve injury

Pain (Amsterdam), 2016-09, Vol.157 (9), p.2068-2080

2016

Volltextzugriff (PDF)

Pathophysiological roles of thrombospondin-4 in disease development

Seminars in cell & developmental biology, 2024-03, Vol.155 (Pt B), p.66-73

2024

Volltextzugriff (PDF)

Persistent inflammation alters the density and distribution of voltage-activated calcium channels in subpopulations of rat cutaneous DRG neurons

Pain (Amsterdam), 2010-12, Vol.151 (3), p.633-643

2010

Volltextzugriff (PDF)

Injury-induced maladaptation and dysregulation of calcium channel α 2 δ subunit proteins and its contribution to neuropathic pain development

British journal of pharmacology, 2018-06, Vol.175 (12), p.2231-2243

2018

Volltextzugriff (PDF)

Injury discharges regulate calcium channel alpha-2-delta-1 subunit upregulation in the dorsal horn that contributes to initiation of neuropathic pain

Pain (Amsterdam), 2008-10, Vol.139 (2), p.358-366

2008

Volltextzugriff (PDF)

Gabapentin Receptor α2δ-1 Is a Neuronal Thrombospondin Receptor Responsible for Excitatory CNS Synaptogenesis

Cell, 2009-10, Vol.139 (2), p.380-392

2009

Volltextzugriff (PDF)

Enhanced Pre-Synaptic Glutamate Release in Deep-Dorsal Horn Contributes to Calcium Channel Alpha-2-Delta-1 Protein-Mediated Spinal Sensitization and Behavioral Hypersensitivity

Molecular pain, 2009-02, Vol.5 (1), p.6-6

2009

Volltextzugriff (PDF)

Impaired Pain Sensation in Mice Lacking Prokineticin 2

Molecular pain, 2006-11, Vol.2 (1), p.35-35

2006

Volltextzugriff (PDF)

Targeting Voltage-Gated Calcium Channels for Neuropathic Pain Management

Neurotherapeutics, 2009-10, Vol.6 (4), p.679-692

2009

Volltextzugriff (PDF)

Painful nerve injury upregulates thrombospondin-4 expression in dorsal root ganglia

Journal of neuroscience research, 2015-03, Vol.93 (3), p.443-453

2015

Volltextzugriff (PDF)

Thrombospondin-4 and excitatory synaptogenesis promote spinal sensitization after painful mechanical joint injury

Experimental neurology, 2015-02, Vol.264, p.111-120

2015

Volltextzugriff (PDF)

Calcium Channel α2δ1 Proteins Mediate Trigeminal Neuropathic Pain States Associated with Aberrant Excitatory Synaptogenesis

The Journal of biological chemistry, 2014-03, Vol.289 (10), p.7025-7037

2014

Volltextzugriff (PDF)

Increased thrombospondin-4 after nerve injury mediates disruption of intracellular calcium signaling in primary sensory neurons

Neuropharmacology, 2017-05, Vol.117, p.292-304

2017

Volltextzugriff (PDF)

Spinal Dorsal Horn Calcium Channel α2δ-1 Subunit Upregulation Contributes to Peripheral Nerve Injury-Induced Tactile Allodynia

The Journal of neuroscience, 2004-09, Vol.24 (39), p.8494-8499

2004

Volltextzugriff (PDF)

Synaptic ultrastructure changes in trigeminocervical complex posttrigeminal nerve injury

Journal of comparative neurology (1911), 2016-02, Vol.524 (2), p.309-322

2016

Volltextzugriff (PDF)

1 – 25 von 392

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(328)
Mit Volltextzugriff
(334)

Publikationsform

Sprache

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 7 Tagen
(1)

Menü

Weitere Dienste

Einstellungen

Neuregulin-1/ErbB4 Signaling Regulates Visual Cortical Plasticity

The Antinociceptive Properties of the Corydalis yanhusuo Extract

Epileptiform activity and behavioral arrests in mice overexpressing the calcium channel subunit α2δ-1

Calcium channel alpha-2-delta-1 protein upregulation in dorsal spinal cord mediates spinal cord injury-induced neuropathic pain states

A Novel Analgesic Isolated from a Traditional Chinese Medicine

Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states

Profiling of dynamically changed gene expression in dorsal root ganglia post peripheral nerve injury and a critical role of injury-induced glial fibrillary acetic protein in maintenance of pain behaviors

Central Mechanisms Mediating Thrombospondin-4-induced Pain States

The EGF-LIKE domain of thrombospondin-4 is a key determinant in the development of pain states due to increased excitatory synaptogenesis

Calcium channel functions in pain processing

Thrombospondin-4 divergently regulates voltage-gated Ca2+ channel subtypes in sensory neurons after nerve injury

Pathophysiological roles of thrombospondin-4 in disease development

Persistent inflammation alters the density and distribution of voltage-activated calcium channels in subpopulations of rat cutaneous DRG neurons

Injury-induced maladaptation and dysregulation of calcium channel α 2 δ subunit proteins and its contribution to neuropathic pain development

Injury discharges regulate calcium channel alpha-2-delta-1 subunit upregulation in the dorsal horn that contributes to initiation of neuropathic pain

Gabapentin Receptor α2δ-1 Is a Neuronal Thrombospondin Receptor Responsible for Excitatory CNS Synaptogenesis

Enhanced Pre-Synaptic Glutamate Release in Deep-Dorsal Horn Contributes to Calcium Channel Alpha-2-Delta-1 Protein-Mediated Spinal Sensitization and Behavioral Hypersensitivity

Impaired Pain Sensation in Mice Lacking Prokineticin 2

Targeting Voltage-Gated Calcium Channels for Neuropathic Pain Management

Painful nerve injury upregulates thrombospondin-4 expression in dorsal root ganglia

Thrombospondin-4 and excitatory synaptogenesis promote spinal sensitization after painful mechanical joint injury

Calcium Channel α2δ1 Proteins Mediate Trigeminal Neuropathic Pain States Associated with Aberrant Excitatory Synaptogenesis

Increased thrombospondin-4 after nerve injury mediates disruption of intracellular calcium signaling in primary sensory neurons

Spinal Dorsal Horn Calcium Channel α2δ-1 Subunit Upregulation Contributes to Peripheral Nerve Injury-Induced Tactile Allodynia

Synaptic ultrastructure changes in trigeminocervical complex posttrigeminal nerve injury

Suchergebnisse filtern