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A Breath of Fresh AIRE Surrounds Paneth Cells and Defensins

Gastroenterology (New York, N.Y. 1943), 2015-07, Vol.149 (1), p.22-25

2015

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Interferon-γ protects against chronic viral myocarditis by reducing mast cell degranulation, fibrosis, and the profibrotic cytokines transforming growth Factor-β1, interleukin-1β, and interleukin-4 in the heart

The American journal of pathology, 2004-12, Vol.165 (6), p.1883-1894

2004

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IL-12 protects against coxsackievirus B3-induced myocarditis by increasing IFN-gamma and macrophage and neutrophil populations in the heart

The Journal of immunology (1950), 2005-01, Vol.174 (1), p.261-269

2005

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Complement Receptor 1 and 2 Deficiency Increases Coxsackievirus B3-Induced Myocarditis, Dilated Cardiomyopathy, and Heart Failure by Increasing Macrophages, IL-1β, and Immune Complex Deposition in the Heart

Journal of Immunology, 2006-03, Vol.176 (6), p.3516-3524

2006

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Interferon-γ Protects against Chronic Viral Myocarditis by Reducing Mast Cell Degranulation, Fibrosis, and the Profibrotic Cytokines Transforming Growth Factor-β 1, Interleukin-1β, and Interleukin-4 in the Heart

The American journal of pathology, 2004-12, Vol.165 (6), p.1883-1894

2004

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Complement Receptor 1 and 2 Deficiency Increases Coxsackievirus B3-Induced Myocarditis, Dilated Cardiomyopathy, and Heart Failure by Increasing Macrophages, IL-1beta, and Immune Complex Deposition in the Heart

The Journal of immunology (1950), 2006-03, Vol.176 (6), p.3516-3524

2006

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IL-12 Protects against Coxsackievirus B3-Induced Myocarditis by Increasing IFN-{gamma} and Macrophage and Neutrophil Populations in the Heart

The Journal of immunology (1950), 2005-01, Vol.174 (1), p.261

2005

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Interferon-{gamma} Protects against Chronic Viral Myocarditis by Reducing Mast Cell Degranulation, Fibrosis, and the Profibrotic Cytokines Transforming Growth Factor-{beta}1, Interleukin-1{beta}, and Interleukin-4 in the Heart

The American journal of pathology, 2004-12, Vol.165 (6), p.1883

2004

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Molecular mimicry, inflammatory bowel disease, and the vaccine safety debate

BMC medicine, 2014-09, Vol.12 (1), p.166-166, Article 166

2014

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Environmental Interaction of Resolved Human Cytomegalovirus Infection With Crohn’s Disease Location

Inflammatory bowel diseases, 2023-02, Vol.29 (2), p.328-331

2023

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NK Cells are Biologic and Biochemical Targets of 6-Mercaptopurine in Crohn's Disease patients

Clinical immunology (Orlando, Fla.), 2017-02, Vol.175, p.82-90

2017

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Su1860 Cytomegalovirus Infection Enhances Susceptibility to Crohn's Disease in Individuals With KIR and HLA Pairs Enabling NK Cell Licensing

Gastroenterology (New York, N.Y. 1943), 2016-04, Vol.150 (4), p.S572-S573

2016

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Sa1868 NK Cells Are Biologic and Biochemical Target of 6-Mercaptopurine in Crohn's Disease Patients

Gastroenterology (New York, N.Y. 1943), 2016-04, Vol.150 (4), p.S385-S385

2016

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Su1988 ENVIRONMENTAL EFFECT OF RESOLVED HUMAN CMV INFECTION AND NK RECEPTOR GENETICS IN PEDIATRIC CROHN'S DISEASE SUSCEPTIBILITY AND PHENOTYPE

Gastroenterology (New York, N.Y. 1943), 2020-05, Vol.158 (6), p.S-735-S-735

2020

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Sa1796 Genetic Licensing Uncovers a Role for NK Cells in Immune Colitis by Co-Activation of CD4+ T Cells

Gastroenterology (New York, N.Y. 1943), 2013-05, Vol.144 (5), p.S-308-S-308

2013

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Human NK cells licensed by killer Ig receptor genes have an altered cytokine program that modifies CD4+ T cell function

The Journal of immunology (1950), 2014-07, Vol.193 (2), p.940-949

2014

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Genetically-determined cytokine programs of human NK cells uncover a novel mechanism of NK cells and Killer cell IgG-like Receptor genes in Crohn’s Disease susceptibility (P3262)

The Journal of immunology (1950), 2013-05, Vol.190 (1_Supplement), p.136-136.13

2013

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Human Natural Killer Cells Licensed by KIR Genes Have an Altered Cytokine Program That Modifies CD4+ T Cell Function1

The Journal of immunology (1950), 2014-06, Vol.193 (2), p.940-949

2014

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Sa1778 Genetically-Determined Cytokine Programs of Human Natural Killer (NK) Cells Uncover a Novel Mechanism of NK Cells and Kir Genes in Crohn's Disease Susceptibility

Gastroenterology (New York, N.Y. 1943), 2013-05, Vol.144 (5), p.S-304-S-304

2013

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A Breath of Fresh AIRE Surrounds Paneth Cells and Defensins

Interferon-γ protects against chronic viral myocarditis by reducing mast cell degranulation, fibrosis, and the profibrotic cytokines transforming growth Factor-β1, interleukin-1β, and interleukin-4 in the heart

IL-12 protects against coxsackievirus B3-induced myocarditis by increasing IFN-gamma and macrophage and neutrophil populations in the heart

Complement Receptor 1 and 2 Deficiency Increases Coxsackievirus B3-Induced Myocarditis, Dilated Cardiomyopathy, and Heart Failure by Increasing Macrophages, IL-1β, and Immune Complex Deposition in the Heart

Interferon-γ Protects against Chronic Viral Myocarditis by Reducing Mast Cell Degranulation, Fibrosis, and the Profibrotic Cytokines Transforming Growth Factor-β 1, Interleukin-1β, and Interleukin-4 in the Heart

Complement Receptor 1 and 2 Deficiency Increases Coxsackievirus B3-Induced Myocarditis, Dilated Cardiomyopathy, and Heart Failure by Increasing Macrophages, IL-1beta, and Immune Complex Deposition in the Heart

IL-12 Protects against Coxsackievirus B3-Induced Myocarditis by Increasing IFN-{gamma} and Macrophage and Neutrophil Populations in the Heart

Interferon-{gamma} Protects against Chronic Viral Myocarditis by Reducing Mast Cell Degranulation, Fibrosis, and the Profibrotic Cytokines Transforming Growth Factor-{beta}1, Interleukin-1{beta}, and Interleukin-4 in the Heart

Molecular mimicry, inflammatory bowel disease, and the vaccine safety debate

Environmental Interaction of Resolved Human Cytomegalovirus Infection With Crohn’s Disease Location

NK Cells are Biologic and Biochemical Targets of 6-Mercaptopurine in Crohn's Disease patients

Su1860 Cytomegalovirus Infection Enhances Susceptibility to Crohn's Disease in Individuals With KIR and HLA Pairs Enabling NK Cell Licensing

Sa1868 NK Cells Are Biologic and Biochemical Target of 6-Mercaptopurine in Crohn's Disease Patients

Su1988 ENVIRONMENTAL EFFECT OF RESOLVED HUMAN CMV INFECTION AND NK RECEPTOR GENETICS IN PEDIATRIC CROHN'S DISEASE SUSCEPTIBILITY AND PHENOTYPE

Sa1796 Genetic Licensing Uncovers a Role for NK Cells in Immune Colitis by Co-Activation of CD4+ T Cells

Human NK cells licensed by killer Ig receptor genes have an altered cytokine program that modifies CD4+ T cell function

Genetically-determined cytokine programs of human NK cells uncover a novel mechanism of NK cells and Killer cell IgG-like Receptor genes in Crohn’s Disease susceptibility (P3262)

Human Natural Killer Cells Licensed by KIR Genes Have an Altered Cytokine Program That Modifies CD4+ T Cell Function1

Sa1778 Genetically-Determined Cytokine Programs of Human Natural Killer (NK) Cells Uncover a Novel Mechanism of NK Cells and Kir Genes in Crohn's Disease Susceptibility

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