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Regulation of human ornithine decarboxylase expression by the c-Myc.Max protein complex

The Journal of biological chemistry, 1993-12, Vol.268 (36), p.27277-27285

1993

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Insulin-Like Growth Factor II Stimulates Cell Proliferation through the Insulin Receptor

Proceedings of the National Academy of Sciences - PNAS, 1997-04, Vol.94 (8), p.3777-3782

1997

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Hepatic growth hormone resistance during sepsis is associated with increased suppressors of cytokine signaling expression and impaired growth hormone signaling

Critical care medicine, 2006-05, Vol.34 (5), p.1420-1427

2006

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Interleukin-1 inhibits the induction of insulin-like growth factor-I by growth hormone in CWSV-1 hepatocytes

American journal of physiology: Gastrointestinal and liver physiology, 2005-08, Vol.289 (2), p.G227-G239

2005

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Tumor necrosis factor mediates hepatic growth hormone resistance during sepsis

American journal of physiology: endocrinology and metabolism, 2002-09, Vol.283 (3), p.E472-E481

2002

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Inhibition of tumorigenesis and induction of apoptosis in human tumor cells by the stable expression of a myristylated COOH terminus of the insulin-like growth factor I receptor

Cancer research (Chicago, Ill.), 1998-06, Vol.58 (11), p.2477-2484

1998

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PERVANADATE BLOCKS THE INHIBITORY EFFECTS OF TNF ON GH SIGNALING: 35

Shock (Augusta, Ga.), 2004-06, Vol.21, p.12

2004

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Cytokine signaling in sepsis: Redundancy, crosstalk, and regulatory mechanisms

Critical care medicine, 2002-01, Vol.30 (1), p.262-263

2002

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Regulation of human ornithine decarboxylase expression by the c-Myc-Max protein complex

The Journal of biological chemistry, 1993-12, Vol.268 (36), p.27277-27285

1993

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IL-1 INHIBITS THE INDUCTION OF IGF-I BY GH IN CWSV-1 HEPATOCYTES: 68

Shock (Augusta, Ga.), 2003-06, Vol.19 (Supplement), p.23-24

2003

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Capsaicin-sensitive nerves regulate the metabolic response to abdominal sepsis

The Journal of surgical research, 2003-06, Vol.112 (2), p.152-161

2003

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MECHANISMS OF GROWTH HORMONE RESISTANCE DURING ABDOMINAL SEPSIS: 48

Shock (Augusta, Ga.), 2003-06, Vol.19 (Supplement), p.17

2003

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Synthetic peptide sequence from the C-terminus of the insulin-like growth factor-I receptor that induces apoptosis and inhibition of tumor growth

Journal of cellular physiology, 1999-10, Vol.181 (1), p.124-135

1999

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TUMOR NECROSIS FACTOR MEDIATES HEPATIC GROWTH HORMONE RESISTANCE DURING SEPSIS: 34

Shock (Augusta, Ga.), 2002-06, Vol.17 (Supplement), p.12

2002

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Characterization of an Antibody That Can Detect an Activated IGF-I Receptor in Human Cancers

Experimental cell research, 1999-08, Vol.251 (1), p.22-32

1999

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Stability of manipulated plasmid DNA in aquatic environments

Environmental toxicology and water quality, 1996, Vol.11 (2), p.129-135

1996

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In situ survival of genetically engineered microorganisms in a tropical aquatic environment[]

Environmental toxicology and water quality, 1996, Vol.11 (1), p.21-25

1996

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Induction of c-fos and c-jun mRNA at the M/G1 border is required for cell cycle progression

Journal of cellular biochemistry, 1994-08, Vol.55 (4), p.503-512

1994

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Transcriptional regulation of c-Jun expression during late G1/S in normal human cells is lost in human tumor cells

Oncogene, 1994-09, Vol.9 (9), p.2675-2682

1994

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Endothelin-1 induces gene expression through stimulation of endothelin type A receptors in normal rat kidney cells

Journal of cellular physiology, 1995-09, Vol.164 (3), p.491-498

1995

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Transcriptional regulation of c-Jun expression during late G sub(1)/S in normal human cells is lost in human tumor cells

Oncogene, 1994-01, Vol.9 (9), p.2675-2682

1994

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Use of Antisense Oligomers to Study the Role of c-jun in G1 Progression

Annals of the New York Academy of Sciences, 1992-10, Vol.660 (1), p.231-239

1992

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Induction of c-fos and c-jun mRNA at the M/G sub(1) border is required for cell cycle progression

Journal of cellular biochemistry, 1994-01, Vol.55 (4), p.503-512

1994

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Regulation of human ornithine decarboxylase expression by the c-Myc.Max protein complex

Insulin-Like Growth Factor II Stimulates Cell Proliferation through the Insulin Receptor

Hepatic growth hormone resistance during sepsis is associated with increased suppressors of cytokine signaling expression and impaired growth hormone signaling

Interleukin-1 inhibits the induction of insulin-like growth factor-I by growth hormone in CWSV-1 hepatocytes

Tumor necrosis factor mediates hepatic growth hormone resistance during sepsis

Inhibition of tumorigenesis and induction of apoptosis in human tumor cells by the stable expression of a myristylated COOH terminus of the insulin-like growth factor I receptor

PERVANADATE BLOCKS THE INHIBITORY EFFECTS OF TNF ON GH SIGNALING: 35

Cytokine signaling in sepsis: Redundancy, crosstalk, and regulatory mechanisms

Regulation of human ornithine decarboxylase expression by the c-Myc-Max protein complex

IL-1 INHIBITS THE INDUCTION OF IGF-I BY GH IN CWSV-1 HEPATOCYTES: 68

Capsaicin-sensitive nerves regulate the metabolic response to abdominal sepsis

MECHANISMS OF GROWTH HORMONE RESISTANCE DURING ABDOMINAL SEPSIS: 48

Synthetic peptide sequence from the C-terminus of the insulin-like growth factor-I receptor that induces apoptosis and inhibition of tumor growth

TUMOR NECROSIS FACTOR MEDIATES HEPATIC GROWTH HORMONE RESISTANCE DURING SEPSIS: 34

Characterization of an Antibody That Can Detect an Activated IGF-I Receptor in Human Cancers

Stability of manipulated plasmid DNA in aquatic environments

In situ survival of genetically engineered microorganisms in a tropical aquatic environment[]

Induction of c-fos and c-jun mRNA at the M/G1 border is required for cell cycle progression

Transcriptional regulation of c-Jun expression during late G1/S in normal human cells is lost in human tumor cells

Endothelin-1 induces gene expression through stimulation of endothelin type A receptors in normal rat kidney cells

Transcriptional regulation of c-Jun expression during late G sub(1)/S in normal human cells is lost in human tumor cells

Use of Antisense Oligomers to Study the Role of c-jun in G1 Progression

Induction of c-fos and c-jun mRNA at the M/G sub(1) border is required for cell cycle progression

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