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Nintedanib overcomes drug resistance from upregulation of FGFR signalling and imatinib‐induced KIT mutations in gastrointestinal stromal tumours

Molecular oncology, 2022-04, Vol.16 (8), p.1761-1774

2022

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Discovery of a novel and highly selective CDK9 kinase inhibitor (JSH-009) with potent antitumor efficacy in preclinical acute myeloid leukemia models

Investigational new drugs, 2020-10, Vol.38 (5), p.1272-1281

2020

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Selectively targeting FLT3-ITD mutants over FLT3-wt by a novel inhibitor for acute myeloid leukemia

Haematologica (Roma), 2021-02, Vol.106 (2), p.605-609

2021

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Discovery of a highly selective VEGFR2 kinase inhibitor CHMFL-VEGFR2-002 as a novel anti-angiogenesis agent

Acta pharmaceutica Sinica. B, 2020-03, Vol.10 (3), p.488-497

2020

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Irreversible inhibition of BTK kinase by a novel highly selective inhibitor CHMFL-BTK-11 suppresses inflammatory response in rheumatoid arthritis model

Scientific reports, 2017-03, Vol.7 (1), p.466-466, Article 466

2017

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Ibrutinib selectively and irreversibly targets EGFR (L858R, Del19) mutant but is moderately resistant to EGFR (T790M) mutant NSCLC Cells

Oncotarget, 2015-10, Vol.6 (31), p.31313-31322

2015

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Axitinib overcomes multiple imatinib resistant cKIT mutations including the gatekeeper mutation T670I in gastrointestinal stromal tumors

Therapeutic advances in medical oncology, 2019-01, Vol.11, p.1758835919849757-1758835919849757

2019

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The paradigm of tumor shrinkage and rapid liver remnant hypertrophy for conversion of initially unresectable colorectal liver metastasis: a case report and literature review

World journal of surgical oncology, 2017-08, Vol.15 (1), p.148-148, Article 148

2017

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Discovery of a highly potent and selective Bruton's tyrosine kinase inhibitor avoiding impairment of ADCC effects for B-cell non-Hodgkin lymphoma

Signal transduction and targeted therapy, 2020-09, Vol.5 (1), p.200-200, Article 200

2020

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Discovery of a highly selective KIT kinase primary V559D mutant inhibitor for gastrointestinal stromal tumors (GISTs)

Oncotarget, 2017-12, Vol.8 (67), p.111110-111118

2017

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Abstract 21: Similarity in the Changes of Brain Myeloid Transcriptome Signature is Shared Between Ischemic Stroke and Chronic Inflammation Caused by Diabetes Mellitus

Stroke (1970), 2024-02, Vol.55 (Suppl_1)

2024

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LC-MS/MS for Assessing the Incorporation and Repair of N2-Alkyl-2′-deoxyguanosine in Genomic DNA

Chemical research in toxicology, 2022-10, Vol.35 (10), p.1814-1820

2022

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LC-MS/MS for Assessing the Incorporation and Repair of N 2‑Alkyl-2′-deoxyguanosine in Genomic DNA

Chemical research in toxicology, 2022-10, Vol.35 (10), p.1814-1820

2022

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Abstract WP327: Altered Bone Marrow Microenvironment Impacts Endothelial Precursor Cell Development and Egress in Mice With Type 2 Diabetes

Stroke (1970), 2024-02, Vol.55 (Suppl_1)

2024

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Targeted Quantitative Profiling of Epitranscriptomic Reader, Writer, and Eraser Proteins Using Stable Isotope-Labeled Peptides

Analytical chemistry (Washington), 2022-09, Vol.94 (37), p.12559-12564

2022

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Modulation of N-terminal methyltransferase 1 by an N 6 -methyladenosine-based epitranscriptomic mechanism

Biochemical and biophysical research communications, 2021-03, Vol.546, p.54

2021

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Proteome-wide Interrogation of Small GTPases Regulated by N 6‑Methyladenosine Modulators

Analytical chemistry (Washington), 2020-07, Vol.92 (14), p.10145-10152

2020

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Streptococcus bovis Contributes to the Development of Colorectal Cancer via Recruiting CD11b⁺TLR-4⁺ Cells

Medical science monitor, 2020-08, Vol.26, p.e921886-e921886-9

2020

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Quantitative proteomics revealed new functions of ALKBH4

Proteomics (Weinheim), 2022-04, Vol.22 (7), p.e2100231-n/a

2022

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Modulation of N-terminal methyltransferase 1 by an N6-methyladenosine-based epitranscriptomic mechanism

Biochemical and biophysical research communications, 2021-03, Vol.546, p.54-58

2021

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Proteome-wide Interrogation of Small GTPases Regulated by N6-Methyladenosine Modulators

Analytical chemistry (Washington), 2020-07, Vol.92 (14), p.10145-10152

2020

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Chemical Proteomic Profiling of the Interacting Proteins of Isoprenoid Pyrophosphates

Analytical chemistry (Washington), 2020-06, Vol.92 (12), p.8031-8036

2020

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All-trans retinoic acid exerts selective anti-FLT3-ITD acute myeloid leukemia efficacy through downregulating Chk1 kinase

Cancer letters, 2020-03, Vol.473, p.130-138

2020

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Discovery of (E)‑N 1‑(3-Fluorophenyl)‑N 3‑(3-(2-(pyridin-2-yl)vinyl)‑1H‑indazol-6-yl)malonamide (CHMFL-KIT-033) as a Novel c‑KIT T670I Mutant Selective Kinase Inhibitor for Gastrointestinal Stromal Tumors (GISTs)

Journal of medicinal chemistry, 2019-05, Vol.62 (10), p.5006-5024

2019

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Discovery of 1‑(4-(4-Amino-3-(4-(2-morpholinoethoxy)phenyl)‑1H‑pyrazolo[3,4‑d]pyrimidin-1-yl)phenyl)-3-(5‑(tert-butyl)isoxazol-3-yl)urea (CHMFL-FLT3-213) as a Highly Potent Type II FLT3 Kinase Inhibitor Capable of Overcoming a Variety of FLT3 Kinase Mutants in FLT3-ITD Positive AML

Journal of medicinal chemistry, 2017-10, Vol.60 (20), p.8407-8424

2017

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Nintedanib overcomes drug resistance from upregulation of FGFR signalling and imatinib‐induced KIT mutations in gastrointestinal stromal tumours

Discovery of a novel and highly selective CDK9 kinase inhibitor (JSH-009) with potent antitumor efficacy in preclinical acute myeloid leukemia models

Selectively targeting FLT3-ITD mutants over FLT3-wt by a novel inhibitor for acute myeloid leukemia

Discovery of a highly selective VEGFR2 kinase inhibitor CHMFL-VEGFR2-002 as a novel anti-angiogenesis agent

Irreversible inhibition of BTK kinase by a novel highly selective inhibitor CHMFL-BTK-11 suppresses inflammatory response in rheumatoid arthritis model

Ibrutinib selectively and irreversibly targets EGFR (L858R, Del19) mutant but is moderately resistant to EGFR (T790M) mutant NSCLC Cells

Axitinib overcomes multiple imatinib resistant cKIT mutations including the gatekeeper mutation T670I in gastrointestinal stromal tumors

The paradigm of tumor shrinkage and rapid liver remnant hypertrophy for conversion of initially unresectable colorectal liver metastasis: a case report and literature review

Discovery of a highly potent and selective Bruton's tyrosine kinase inhibitor avoiding impairment of ADCC effects for B-cell non-Hodgkin lymphoma

Discovery of a highly selective KIT kinase primary V559D mutant inhibitor for gastrointestinal stromal tumors (GISTs)

Abstract 21: Similarity in the Changes of Brain Myeloid Transcriptome Signature is Shared Between Ischemic Stroke and Chronic Inflammation Caused by Diabetes Mellitus

LC-MS/MS for Assessing the Incorporation and Repair of N2-Alkyl-2′-deoxyguanosine in Genomic DNA

LC-MS/MS for Assessing the Incorporation and Repair of N 2‑Alkyl-2′-deoxyguanosine in Genomic DNA

Abstract WP327: Altered Bone Marrow Microenvironment Impacts Endothelial Precursor Cell Development and Egress in Mice With Type 2 Diabetes

Targeted Quantitative Profiling of Epitranscriptomic Reader, Writer, and Eraser Proteins Using Stable Isotope-Labeled Peptides

Modulation of N-terminal methyltransferase 1 by an N 6 -methyladenosine-based epitranscriptomic mechanism

Proteome-wide Interrogation of Small GTPases Regulated by N 6‑Methyladenosine Modulators

Streptococcus bovis Contributes to the Development of Colorectal Cancer via Recruiting CD11b⁺TLR-4⁺ Cells

Quantitative proteomics revealed new functions of ALKBH4

Modulation of N-terminal methyltransferase 1 by an N6-methyladenosine-based epitranscriptomic mechanism

Proteome-wide Interrogation of Small GTPases Regulated by N6-Methyladenosine Modulators

Chemical Proteomic Profiling of the Interacting Proteins of Isoprenoid Pyrophosphates

All-trans retinoic acid exerts selective anti-FLT3-ITD acute myeloid leukemia efficacy through downregulating Chk1 kinase

Discovery of (E)‑N 1‑(3-Fluorophenyl)‑N 3‑(3-(2-(pyridin-2-yl)vinyl)‑1H‑indazol-6-yl)malonamide (CHMFL-KIT-033) as a Novel c‑KIT T670I Mutant Selective Kinase Inhibitor for Gastrointestinal Stromal Tumors (GISTs)

Discovery of 1‑(4-(4-Amino-3-(4-(2-morpholinoethoxy)phenyl)‑1H‑pyrazolo[3,4‑d]pyrimidin-1-yl)phenyl)-3-(5‑(tert-butyl)isoxazol-3-yl)urea (CHMFL-FLT3-213) as a Highly Potent Type II FLT3 Kinase Inhibitor Capable of Overcoming a Variety of FLT3 Kinase Mutants in FLT3-ITD Positive AML

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