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Transactivation of ErbB Family of Receptor Tyrosine Kinases Is Inhibited by Angiotensin-(1-7) via Its Mas Receptor

PloS one, 2015-11, Vol.10 (11), p.e0141657

2015

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Activation of EGFR/ERBB2 via pathways involving ERK1/2, P38 MAPK, AKT and FOXO enhances recovery of diabetic hearts from ischemia-reperfusion injury

PloS one, 2012-06, Vol.7 (6), p.e39066

2012

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Cationic Polyamidoamine Dendrimers as Modulators of EGFR Signaling In Vitro and In Vivo

PloS one, 2015-07, Vol.10 (7), p.e0132215-e0132215

2015

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Angiotensin-(1-7) prevents diabetes-induced cardiovascular dysfunction

American journal of physiology. Heart and circulatory physiology, 2007-01, Vol.292 (1), p.H666-H672

2007

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Angiotensin‐(1‐7) inhibits epidermal growth factor receptor transactivation via a Mas receptor‐dependent pathway

British journal of pharmacology, 2012-03, Vol.165 (5), p.1390-1400

2012

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Vasodilator Effect of Trace Amines, 3-Iodothyronamine, and RO5263397 in the Rat Perfused Kidney: Comparison with Tryptamine

Pharmacology, 2023-07, Vol.108 (4), p.368-378

2023

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Angiotensin-(1-7) prevents development of severe hypertension and end-organ damage in spontaneously hypertensive rats treated with L-NAME

American journal of physiology. Heart and circulatory physiology, 2006-02, Vol.290 (2), p.H684-H691

2006

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Activation of ErbB2 and Downstream Signalling via Rho Kinases and ERK1/2 Contributes to Diabetes-Induced Vascular Dysfunction

PloS one, 2013-06, Vol.8 (6), p.e67813

2013

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Angiotensin-(1–7) prevents diabetes-induced attenuation in PPAR-γ and catalase activities

European journal of pharmacology, 2010-07, Vol.638 (1), p.108-114

2010

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Hydrogen sulfide donor GYY4137 attenuates vascular complications in mesenteric bed of streptozotocin-induced diabetic rats

European journal of pharmacology, 2022-10, Vol.933, p.175265-175265, Article 175265

2022

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Chronic treatment with LY294002, an inhibitor of phosphatidylinositol 3-kinase, attenuates ischemia/reperfusion-induced cardiac dysfunction in normotensive and hypertensive diabetic animals

Molecular and cellular biochemistry, 2013-01, Vol.373 (1-2), p.259-264

2013

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Chronic Treatment with Ang-(1-7) Reverses Abnormal Reactivity in the Corpus Cavernosum and Normalizes Diabetes-Induced Changes in the Protein Levels of ACE, ACE2, ROCK1, ROCK2 and Omega-Hydroxylase in a Rat Model of Type 1 Diabetes

Journal of Diabetes Research, 2014-01, Vol.2014 (2014), p.1-10

2014

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Chronic Treatment With Hydrogen Sulfide Donor GYY4137 Mitigates Microglial and Astrocyte Activation in the Spinal Cord of Streptozotocin-Induced Diabetic Rats

Journal of neuropathology and experimental neurology, 2020-12, Vol.79 (12), p.1320-1343

2020

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Alleviation of impaired reactivity in the corpus cavernosum of STZ-diabetic rats by slow-release H2S donor GYY4137

International journal of impotence research, 2019-03, Vol.31 (2), p.111-118

2019

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On the nanotoxicity of PAMAM dendrimers: Superfect® stimulates the EGFR–ERK1/2 signal transduction pathway via an oxidative stress-dependent mechanism in HEK 293 cells

International journal of pharmaceutics, 2013-05, Vol.448 (1), p.239-246

2013

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Characterization of Angiotensin-(1–7) effects on the cardiovascular system in an experimental model of Type-1 diabetes

Pharmacological research, 2012-09, Vol.66 (3), p.269-275

2012

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Angiotensin-(1–7) Prevents Activation of NADPH Oxidase and Renal Vascular Dysfunction in Diabetic Hypertensive Rats

American journal of nephrology, 2008-01, Vol.28 (1), p.25-33

2008

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Phosphoinositide 3-kinase contributes to diabetes-induced abnormal vascular reactivity in rat perfused mesenteric bed

Cell biochemistry and function, 2008-06, Vol.26 (4), p.451-458

Yousif, Mariam H. M.

2008

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Chronic administration of nano-sized PAMAM dendrimers in vivo inhibits EGFR-ERK1/2-ROCK signaling pathway and attenuates diabetes-induced vascular remodeling and dysfunction

Nanomedicine, 2019-06, Vol.18, p.78-89

2019

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Role of angiotensin II and angiotensin-(1–7) in diabetes-induced oxidative DNA damage in the corpus cavernosum

Fertility and sterility, 2013-07, Vol.100 (1), p.226-233

2013

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The dual targeting of EGFR and ErbB2 with the inhibitor Lapatinib corrects high glucose-induced apoptosis and vascular dysfunction by opposing multiple diabetes-induced signaling changes

Journal of drug targeting, 2015-07, Vol.23 (6), p.506-518

2015

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Angiotensin-(1-7) Blockade Attenuates Captopril- or Hydralazine-induced Cardiovascular Protection in Spontaneously Hypertensive Rats Treated With NG-nitro-l-Arginine Methyl Ester

Journal of cardiovascular pharmacology, 2011-05, Vol.57 (5), p.559-567

2011

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Role of protein kinases in mediating diabetes-induced augmented vasoconstriction to endothelin-1 in the renal arteries of STZ-diabetic rats

Cell biochemistry and function, 2006-09, Vol.24 (5), p.397-405

Yousif, Mariam H. M.

2006

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Early inhibition of EGFR signaling prevents diabetes-induced up-regulation of multiple gene pathways in the mesenteric vasculature

Vascular pharmacology, 2009-10, Vol.51 (4), p.236-245

2009

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Global upregulation of gene expression associated with renal dysfunction in DOCA–salt-induced hypertensive rats occurs via signaling cascades involving epidermal growth factor receptor: A microarray analysis

Vascular pharmacology, 2009-08, Vol.51 (2), p.101-109

2009

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Transactivation of ErbB Family of Receptor Tyrosine Kinases Is Inhibited by Angiotensin-(1-7) via Its Mas Receptor

Activation of EGFR/ERBB2 via pathways involving ERK1/2, P38 MAPK, AKT and FOXO enhances recovery of diabetic hearts from ischemia-reperfusion injury

Cationic Polyamidoamine Dendrimers as Modulators of EGFR Signaling In Vitro and In Vivo

Angiotensin-(1-7) prevents diabetes-induced cardiovascular dysfunction

Angiotensin‐(1‐7) inhibits epidermal growth factor receptor transactivation via a Mas receptor‐dependent pathway

Vasodilator Effect of Trace Amines, 3-Iodothyronamine, and RO5263397 in the Rat Perfused Kidney: Comparison with Tryptamine

Angiotensin-(1-7) prevents development of severe hypertension and end-organ damage in spontaneously hypertensive rats treated with L-NAME

Activation of ErbB2 and Downstream Signalling via Rho Kinases and ERK1/2 Contributes to Diabetes-Induced Vascular Dysfunction

Angiotensin-(1–7) prevents diabetes-induced attenuation in PPAR-γ and catalase activities

Hydrogen sulfide donor GYY4137 attenuates vascular complications in mesenteric bed of streptozotocin-induced diabetic rats

Chronic treatment with LY294002, an inhibitor of phosphatidylinositol 3-kinase, attenuates ischemia/reperfusion-induced cardiac dysfunction in normotensive and hypertensive diabetic animals

Chronic Treatment with Ang-(1-7) Reverses Abnormal Reactivity in the Corpus Cavernosum and Normalizes Diabetes-Induced Changes in the Protein Levels of ACE, ACE2, ROCK1, ROCK2 and Omega-Hydroxylase in a Rat Model of Type 1 Diabetes

Chronic Treatment With Hydrogen Sulfide Donor GYY4137 Mitigates Microglial and Astrocyte Activation in the Spinal Cord of Streptozotocin-Induced Diabetic Rats

Alleviation of impaired reactivity in the corpus cavernosum of STZ-diabetic rats by slow-release H2S donor GYY4137

On the nanotoxicity of PAMAM dendrimers: Superfect® stimulates the EGFR–ERK1/2 signal transduction pathway via an oxidative stress-dependent mechanism in HEK 293 cells

Characterization of Angiotensin-(1–7) effects on the cardiovascular system in an experimental model of Type-1 diabetes

Angiotensin-(1–7) Prevents Activation of NADPH Oxidase and Renal Vascular Dysfunction in Diabetic Hypertensive Rats

Phosphoinositide 3-kinase contributes to diabetes-induced abnormal vascular reactivity in rat perfused mesenteric bed

Chronic administration of nano-sized PAMAM dendrimers in vivo inhibits EGFR-ERK1/2-ROCK signaling pathway and attenuates diabetes-induced vascular remodeling and dysfunction

Role of angiotensin II and angiotensin-(1–7) in diabetes-induced oxidative DNA damage in the corpus cavernosum

The dual targeting of EGFR and ErbB2 with the inhibitor Lapatinib corrects high glucose-induced apoptosis and vascular dysfunction by opposing multiple diabetes-induced signaling changes

Angiotensin-(1-7) Blockade Attenuates Captopril- or Hydralazine-induced Cardiovascular Protection in Spontaneously Hypertensive Rats Treated With NG-nitro-l-Arginine Methyl Ester

Role of protein kinases in mediating diabetes-induced augmented vasoconstriction to endothelin-1 in the renal arteries of STZ-diabetic rats

Early inhibition of EGFR signaling prevents diabetes-induced up-regulation of multiple gene pathways in the mesenteric vasculature

Global upregulation of gene expression associated with renal dysfunction in DOCA–salt-induced hypertensive rats occurs via signaling cascades involving epidermal growth factor receptor: A microarray analysis

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