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White matter microstructural abnormalities and default network degeneration are associated with early memory deficit in Alzheimer's disease continuum

Scientific reports, 2019-03, Vol.9 (1), p.4749-4749, Article 4749

2019

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Distinct BOLD variability changes in the default mode and salience networks in Alzheimer's disease spectrum and associations with cognitive decline

Scientific reports, 2020-04, Vol.10 (1), p.6457-6457, Article 6457

2020

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Amyloid burden accelerates white matter degradation in cognitively normal elderly individuals

Human brain mapping, 2019-05, Vol.40 (7), p.2065-2075

2019

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Cerebrovascular disease influences functional and structural network connectivity in patients with amnestic mild cognitive impairment and Alzheimer's disease

Alzheimer's research & therapy, 2018-08, Vol.10 (1), p.82-82, Article 82

2018

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Hepatitis B virus core interacts with the host cell nucleolar protein, nucleophosmin 1

The Journal of Microbiology, 2009, 47(6), , pp.746-752

2009

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Brain structure, amyloid, and behavioral features for predicting clinical progression in subjective cognitive decline

Human brain mapping, 2024-07, Vol.45 (10), p.e26765-n/a

2024

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Heat shock protein 90 facilitates formation of the HBV capsid via interacting with the HBV core protein dimers

Virology (New York, N.Y.), 2011-02, Vol.410 (1), p.161-169

2011

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Stage‐dependent amyloid beta‐ and tau‐associated longitudinal white matter degeneration in early stages of Alzheimer’s disease

Alzheimer's & dementia, 2020-12, Vol.16, p.n/a

2020

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Stage‐dependent amyloid beta‐ and tau‐associated longitudinal white matter degeneration in early stages of Alzheimer’s disease: Neuroimaging / Optimal neuroimaging measures for early detection

Alzheimer's & dementia, 2020-12, Vol.16 (S5)

2020

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Cerebral microinfarcts affect brain structural network topology in cognitively impaired patients

Journal of cerebral blood flow and metabolism, 2021-01, Vol.41 (1), p.105-115

2021

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Mechanisms Linking White Matter Lesions, Tract Integrity, and Depression in Alzheimer Disease

The American journal of geriatric psychiatry, 2019-09, Vol.27 (9), p.948-959

2019

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P3‐376: CEREBRAL MICROINFARCT INFLUENCES STRUCTURAL NETWORK TOPOLOGY IN ALZHEIMER'S DISEASE AND COGNITIVE IMPAIRMENT NO DEMENTIA

Alzheimer's & dementia, 2018-07, Vol.14 (7S_Part_23), p.P1235-P1236

2018

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CEREBRAL MICROINFARCT INFLUENCES STRUCTURAL NETWORK TOPOLOGY IN ALZHEIMER’S DISEASE AND COGNITIVE IMPAIRMENT NO DEMENTIA

Alzheimer's & dementia, 2018-07, Vol.14 (7), p.P1235-P1236

2018

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IC‐P‐102: β‐AMYLOID RELATED FOCAL TO WIDESPREAD PROGRESSION OF HIPPOCAMPAL SUBFIELDS ATROPHY IN NON‐DEMENTED ELDERLY

Alzheimer's & dementia, 2019-07, Vol.15 (7S_Part_2), p.P88-P89

2019

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P4‐289: BOLD SIGNAL VARIABILITY CHANGES IN THE DEFAULT MODE AND SALIENCE NETWORKS IN AMNESTIC MILD COGNITIVE IMPAIRMENT AND ALZHEIMER'S DISEASE AND THEIR ASSOCIATIONS WITH COGNITIVE DECLINE

Alzheimer's & dementia, 2019-07, Vol.15 (7S_Part_27), p.P1398-P1399

2019

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P2‐074: β‐AMYLOID RELATED FOCAL TO WIDESPREAD PROGRESSION OF HIPPOCAMPAL SUBFIELDS ATROPHY IN NONDEMENTED ELDERLY

Alzheimer's & dementia, 2019-07, Vol.15 (7S_Part_11), p.P599-P600

2019

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IC‐P‐035: BOLD SIGNAL VARIABILITY CHANGES IN THE DEFAULT MODE AND SALIENCE NETWORKS IN AMNESTIC MILD COGNITIVE IMPAIRMENT AND ALZHEIMER'S DISEASE AND THEIR ASSOCIATIONS WITH COGNITIVE DECLINE

Alzheimer's & dementia, 2019-07, Vol.15 (7S_Part_1), p.P41-P42

2019

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BOLD SIGNAL VARIABILITY CHANGES IN THE DEFAULT MODE AND SALIENCE NETWORKS IN AMNESTIC MILD COGNITIVE IMPAIRMENT AND ALZHEIMER’S DISEASE AND THEIR ASSOCIATIONS WITH COGNITIVE DECLINE

Alzheimer's & dementia, 2019-07, Vol.15 (7), p.P41-P42

2019

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β-AMYLOID RELATED FOCAL TO WIDESPREAD PROGRESSION OF HIPPOCAMPAL SUBFIELDS ATROPHY IN NONDEMENTED ELDERLY

Alzheimer's & dementia, 2019-07, Vol.15 (7), p.P599-P600

2019

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β-AMYLOID RELATED FOCAL TO WIDESPREAD PROGRESSION OF HIPPOCAMPAL SUBFIELDS ATROPHY IN NON-DEMENTED ELDERLY

Alzheimer's & dementia, 2019-07, Vol.15 (7), p.P88-P89

2019

Volltextzugriff (PDF)

BOLD SIGNAL VARIABILITY CHANGES IN THE DEFAULT MODE AND SALIENCE NETWORKS IN AMNESTIC MILD COGNITIVE IMPAIRMENT AND ALZHEIMER’S DISEASE AND THEIR ASSOCIATIONS WITH COGNITIVE DECLINE

Alzheimer's & dementia, 2019-07, Vol.15 (7), p.P1398-P1399

2019

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Longitudinal associations between β-amyloid and cortical thickness in mild cognitive impairment

Brain communications, 2023, Vol.5 (4), p.fcad192-fcad192

2023

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Ag or Au Nanoparticle-Embedded One-Dimensional Composite TiO2 Nanofibers Prepared via Electrospinning for Use in Lithium-Ion Batteries

ACS applied materials & interfaces, 2010-07, Vol.2 (7), p.2046-2052

2010

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Large Accumulation of Micro-sized Synthetic Polymer Particles in the Sea Surface Microlayer

Environmental science & technology, 2014-08, Vol.48 (16), p.9014-9021

2014

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Spatial distribution of microplastic in the surface waters along the coast of Korea

Marine pollution bulletin, 2020-06, Vol.155, p.110729-110729, Article 110729

2020

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White matter microstructural abnormalities and default network degeneration are associated with early memory deficit in Alzheimer's disease continuum

Distinct BOLD variability changes in the default mode and salience networks in Alzheimer's disease spectrum and associations with cognitive decline

Amyloid burden accelerates white matter degradation in cognitively normal elderly individuals

Cerebrovascular disease influences functional and structural network connectivity in patients with amnestic mild cognitive impairment and Alzheimer's disease

Hepatitis B virus core interacts with the host cell nucleolar protein, nucleophosmin 1

Brain structure, amyloid, and behavioral features for predicting clinical progression in subjective cognitive decline

Heat shock protein 90 facilitates formation of the HBV capsid via interacting with the HBV core protein dimers

Stage‐dependent amyloid beta‐ and tau‐associated longitudinal white matter degeneration in early stages of Alzheimer’s disease

Stage‐dependent amyloid beta‐ and tau‐associated longitudinal white matter degeneration in early stages of Alzheimer’s disease: Neuroimaging / Optimal neuroimaging measures for early detection

Cerebral microinfarcts affect brain structural network topology in cognitively impaired patients

Mechanisms Linking White Matter Lesions, Tract Integrity, and Depression in Alzheimer Disease

P3‐376: CEREBRAL MICROINFARCT INFLUENCES STRUCTURAL NETWORK TOPOLOGY IN ALZHEIMER'S DISEASE AND COGNITIVE IMPAIRMENT NO DEMENTIA

CEREBRAL MICROINFARCT INFLUENCES STRUCTURAL NETWORK TOPOLOGY IN ALZHEIMER’S DISEASE AND COGNITIVE IMPAIRMENT NO DEMENTIA

IC‐P‐102: β‐AMYLOID RELATED FOCAL TO WIDESPREAD PROGRESSION OF HIPPOCAMPAL SUBFIELDS ATROPHY IN NON‐DEMENTED ELDERLY

P4‐289: BOLD SIGNAL VARIABILITY CHANGES IN THE DEFAULT MODE AND SALIENCE NETWORKS IN AMNESTIC MILD COGNITIVE IMPAIRMENT AND ALZHEIMER'S DISEASE AND THEIR ASSOCIATIONS WITH COGNITIVE DECLINE

P2‐074: β‐AMYLOID RELATED FOCAL TO WIDESPREAD PROGRESSION OF HIPPOCAMPAL SUBFIELDS ATROPHY IN NONDEMENTED ELDERLY

IC‐P‐035: BOLD SIGNAL VARIABILITY CHANGES IN THE DEFAULT MODE AND SALIENCE NETWORKS IN AMNESTIC MILD COGNITIVE IMPAIRMENT AND ALZHEIMER'S DISEASE AND THEIR ASSOCIATIONS WITH COGNITIVE DECLINE

BOLD SIGNAL VARIABILITY CHANGES IN THE DEFAULT MODE AND SALIENCE NETWORKS IN AMNESTIC MILD COGNITIVE IMPAIRMENT AND ALZHEIMER’S DISEASE AND THEIR ASSOCIATIONS WITH COGNITIVE DECLINE

β-AMYLOID RELATED FOCAL TO WIDESPREAD PROGRESSION OF HIPPOCAMPAL SUBFIELDS ATROPHY IN NONDEMENTED ELDERLY

β-AMYLOID RELATED FOCAL TO WIDESPREAD PROGRESSION OF HIPPOCAMPAL SUBFIELDS ATROPHY IN NON-DEMENTED ELDERLY

BOLD SIGNAL VARIABILITY CHANGES IN THE DEFAULT MODE AND SALIENCE NETWORKS IN AMNESTIC MILD COGNITIVE IMPAIRMENT AND ALZHEIMER’S DISEASE AND THEIR ASSOCIATIONS WITH COGNITIVE DECLINE

Longitudinal associations between β-amyloid and cortical thickness in mild cognitive impairment

Ag or Au Nanoparticle-Embedded One-Dimensional Composite TiO2 Nanofibers Prepared via Electrospinning for Use in Lithium-Ion Batteries

Large Accumulation of Micro-sized Synthetic Polymer Particles in the Sea Surface Microlayer

Spatial distribution of microplastic in the surface waters along the coast of Korea

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