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Epigenetic regulation of cancer stem cell marker CD133 by transforming growth factor‐β

Hepatology (Baltimore, Md.), 2010-05, Vol.51 (5), p.1635-1644

2010

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Macrophages directly mediate diabetic renal injury

American journal of physiology. Renal physiology, 2013-12, Vol.305 (12), p.F1719-F1727

2013

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c‐Met represents a potential therapeutic target for personalized treatment in hepatocellular carcinoma

Hepatology (Baltimore, Md.), 2011-09, Vol.54 (3), p.879-889

2011

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Epithelial‐to‐mesenchymal transition of murine liver tumor cells promotes invasion

Hepatology (Baltimore, Md.), 2010-09, Vol.52 (3), p.945-953

2010

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Podocyte-specific chemokine (C-C motif) receptor 2 overexpression mediates diabetic renal injury in mice

Kidney international, 2017-03, Vol.91 (3), p.671-682

2017

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The EGFR/ErbB3 Pathway Acts as a Compensatory Survival Mechanism upon c-Met Inhibition in Human c-Met+ Hepatocellular Carcinoma

PloS one, 2015-05, Vol.10 (5), p.e0128159-e0128159

2015

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Delayed Treatment with a Small Pigment Epithelium Derived Factor (PEDF) Peptide Prevents the Progression of Diabetic Renal Injury

PloS one, 2015-07, Vol.10 (7), p.e0133777-e0133777

2015

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Clinical application for the preservation of phospho-proteins through in-situ tissue stabilization

Proteome science, 2010-11, Vol.8 (1), p.61-61

2010

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Macrophage-derived tumor necrosis factor-α mediates diabetic renal injury

Kidney international, 2015-10, Vol.88 (4), p.722-733

2015

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Arginase inhibition mediates renal tissue protection in diabetic nephropathy by a nitric oxide synthase 3-dependent mechanism

Kidney international, 2013-12, Vol.84 (6), p.1189-1197

2013

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Arginase inhibition: a new treatment for preventing progression of established diabetic nephropathy

American journal of physiology. Renal physiology, 2015-09, Vol.309 (5), p.F447-F455

2015

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The Central Role of Protein Kinase C Epsilon in Cyanide Cardiotoxicity and Its Treatment

Toxicological sciences, 2019-09, Vol.171 (1), p.247-257

2019

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Diabetic nephropathy is resistant to oral L-arginine or L-citrulline supplementation

American journal of physiology. Renal physiology, 2014-12, Vol.307 (11), p.F1292-F1301

2014

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CD133+ liver cancer stem cells from methionine adenosyl transferase 1A–deficient mice demonstrate resistance to transforming growth factor (TGF)‐β–induced apoptosis

Hepatology (Baltimore, Md.), 2009-04, Vol.49 (4), p.1277-1286

2009

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c-Met, a potential personalized and therapeutic biomarker for mesenchymal and stemness characteristics in metastatic hepatocellular carcinoma

Clinical cancer research, 2010-10, Vol.16 (19_Supplement), p.A34-A34

2010

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Distinct roles of arginases 1 and 2 in diabetic nephropathy

American journal of physiology. Renal physiology, 2017-10, Vol.313 (4), p.F899-F905

2017

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Diabetic renal injury is not prevented or reduced by increasing renal arginine availability (689.11)

The FASEB journal, 2014-04, Vol.28 (S1), p.n/a

2014

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Elucidating the Role of Arginases in Diabetic Nephropathy: Arginase 2‐dependent and ‐independent Effects

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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Abstract C5: Epigenetic regulation of cancer stem cell marker CD133 by transforming growth factor-β

Cancer research (Chicago, Ill.), 2009-12, Vol.69 (23_Supplement), p.C5-C5

2009

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The EGFR/ErbB3 Pathway Acts as a Compensatory Survival Mechanism upon c-Met Inhibition in Human c-Met.sup.+ Hepatocellular Carcinoma

PloS one, 2015-05, Vol.10 (5)

2015

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Abstract 3393: Targeting c-Met oncogene and the compensatory EGFR/ErbB3 pathways to eliminate c-Met activated hepatocellular carcinoma

Cancer research (Chicago, Ill.), 2013-04, Vol.73 (8_Supplement), p.3393-3393

2013

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Epigenetic regulation of cancer stem cell marker CD133 by transforming growth factor-[beta]

Hepatology (Baltimore, Md.), 2010-05, Vol.51 (5), p.1635

2010

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Protective role of small pigment epithelium-derived factor (PEDF) peptide in diabetic renal injury

American journal of physiology. Renal physiology, 2013-09, Vol.305 (6), p.F891-F900

2013

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Macrophage-derived tumor necrosis factor-[alpha] mediates diabetic renal injury

Kidney international, 2015-10, Vol.88 (4), p.722

2015

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485 Transforming Growth Factor-β Induces CD133 Expression and Epithelial-Mesenchymal Transition in Liver Cancer Stem Cells

Gastroenterology (New York, N.Y. 1943), 2009, Vol.136 (5), p.A-78-A-78

2009

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Epigenetic regulation of cancer stem cell marker CD133 by transforming growth factor‐β

Macrophages directly mediate diabetic renal injury

c‐Met represents a potential therapeutic target for personalized treatment in hepatocellular carcinoma

Epithelial‐to‐mesenchymal transition of murine liver tumor cells promotes invasion

Podocyte-specific chemokine (C-C motif) receptor 2 overexpression mediates diabetic renal injury in mice

The EGFR/ErbB3 Pathway Acts as a Compensatory Survival Mechanism upon c-Met Inhibition in Human c-Met+ Hepatocellular Carcinoma

Delayed Treatment with a Small Pigment Epithelium Derived Factor (PEDF) Peptide Prevents the Progression of Diabetic Renal Injury

Clinical application for the preservation of phospho-proteins through in-situ tissue stabilization

Macrophage-derived tumor necrosis factor-α mediates diabetic renal injury

Arginase inhibition mediates renal tissue protection in diabetic nephropathy by a nitric oxide synthase 3-dependent mechanism

Arginase inhibition: a new treatment for preventing progression of established diabetic nephropathy

The Central Role of Protein Kinase C Epsilon in Cyanide Cardiotoxicity and Its Treatment

Diabetic nephropathy is resistant to oral L-arginine or L-citrulline supplementation

CD133+ liver cancer stem cells from methionine adenosyl transferase 1A–deficient mice demonstrate resistance to transforming growth factor (TGF)‐β–induced apoptosis

c-Met, a potential personalized and therapeutic biomarker for mesenchymal and stemness characteristics in metastatic hepatocellular carcinoma

Distinct roles of arginases 1 and 2 in diabetic nephropathy

Diabetic renal injury is not prevented or reduced by increasing renal arginine availability (689.11)

Elucidating the Role of Arginases in Diabetic Nephropathy: Arginase 2‐dependent and ‐independent Effects

Abstract C5: Epigenetic regulation of cancer stem cell marker CD133 by transforming growth factor-β

The EGFR/ErbB3 Pathway Acts as a Compensatory Survival Mechanism upon c-Met Inhibition in Human c-Met.sup.+ Hepatocellular Carcinoma

Abstract 3393: Targeting c-Met oncogene and the compensatory EGFR/ErbB3 pathways to eliminate c-Met activated hepatocellular carcinoma

Epigenetic regulation of cancer stem cell marker CD133 by transforming growth factor-[beta]

Protective role of small pigment epithelium-derived factor (PEDF) peptide in diabetic renal injury

Macrophage-derived tumor necrosis factor-[alpha] mediates diabetic renal injury

485 Transforming Growth Factor-β Induces CD133 Expression and Epithelial-Mesenchymal Transition in Liver Cancer Stem Cells

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