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A role for mitochondria in NLRP3 inflammasome activation

Nature (London), 2011-01, Vol.469 (7329), p.221-225

2011

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NLRP1 Is the Key Inflammasome in Primary Human Keratinocytes

Journal of investigative dermatology, 2018-12, Vol.138 (12), p.2507-2510

2018

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Inflammasome Activators Induce Interleukin-1α Secretion via Distinct Pathways with Differential Requirement for the Protease Function of Caspase-1

Immunity (Cambridge, Mass.), 2012-03, Vol.36 (3), p.388-400

2012

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Molecular Mechanisms of Hair Growth and Regeneration: Current Understanding and Novel Paradigms

Dermatology (Basel), 2020, Vol.236 (4), p.271

2020

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Nanoparticles activate the NLR pyrin domain containing 3 (Nlrp3) inflammasome and cause pulmonary inflammation through release of IL-1α and IL-1β

Proceedings of the National Academy of Sciences - PNAS, 2010-11, Vol.107 (45), p.19449-19454

2010

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RIPK1 and TRADD Regulate TNF-Induced Signaling and Ripoptosome Formation

International journal of molecular sciences, 2021-11, Vol.22 (22), p.12459

2021

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The NLRP1 inflammasome in skin diseases

Frontiers in immunology, 2023-02, Vol.14, p.1111611-1111611

2023

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Differential expression of NLRP3 among hematopoietic cells

The Journal of immunology (1950), 2011-02, Vol.186 (4), p.2529-2534

2011

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Transcriptional adaptation of staphylococci during colonization of the authentic human environment: An overview of transcriptomic changes and their relationship to physiological conditions

Frontiers in cellular and infection microbiology, 2022-11, Vol.12, p.1062329-1062329

2022

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Tissue-specific opposing functions of the inflammasome adaptor ASC in the regulation of epithelial skin carcinogenesis

Proceedings of the National Academy of Sciences - PNAS, 2012-11, Vol.109 (45), p.18384-18389

2012

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Silencing of ASC in Cutaneous Squamous Cell Carcinoma

PloS one, 2016-10, Vol.11 (10), p.e0164742-e0164742

2016

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Macrophage migration inhibitory factor (MIF) and its homolog D-dopachrome tautomerase (D-DT) are significant promotors of UVB- but not chemically induced non-melanoma skin cancer

Scientific reports, 2023-07, Vol.13 (1), p.11611-11611, Article 11611

2023

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Innate immune activation as cofactor in pemphigus disease manifestation

Frontiers in immunology, 2022-07, Vol.13, p.898819-898819

2022

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Apremilast prevents blistering in human epidermis and stabilizes keratinocyte adhesion in pemphigus

Nature communications, 2023-01, Vol.14 (1), p.116-116, Article 116

2023

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Activation of hypoxia inducible factor 1 is a general phenomenon in infections with human pathogens

PloS one, 2010-07, Vol.5 (7), p.e11576-e11576

2010

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The Staphylococcus epidermidis Transcriptional Profile During Carriage

Frontiers in microbiology, 2022-04, Vol.13, p.896311-896311

2022

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TNF Is Partially Required for Cell-Death-Triggered Skin Inflammation upon Acute Loss of cFLIP

International journal of molecular sciences, 2020-11, Vol.21 (22), p.8859

2020

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Induction of skin-pathogenic Th22 cells by epicutaneous allergen exposure

Journal of dermatological science, 2017-09, Vol.87 (3), p.268-277

2017

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Author Correction: Apremilast prevents blistering in human epidermis and stabilizes keratinocyte adhesion in pemphigus

Nature communications, 2023-02, Vol.14 (1), p.665-665, Article 665

2023

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Role of ADAM10 and ADAM17 in the Regulation of Keratinocyte Adhesion in Pemphigus Vulgaris

Frontiers in immunology, 2022-06, Vol.13, p.884248-884248

2022

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MALT1 Protease Activity Controls the Expression of Inflammatory Genes in Keratinocytes upon Zymosan Stimulation

Journal of investigative dermatology, 2016-04, Vol.136 (4), p.788-797

2016

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ST18 Enhances PV-IgG-Induced Loss of Keratinocyte Cohesion in Parallel to Increased ERK Activation

Frontiers in immunology, 2019-04, Vol.10, p.770-770

2019

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Pitfalls in the Application of Dispase-Based Keratinocyte Dissociation Assay for In Vitro Analysis of Pemphigus Vulgaris

Vaccines (Basel), 2022-01, Vol.10 (2), p.208

2022

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Differential induction of ATF3 and HO-1 in myeloid cells and keratinocytes via Dimethylfumarate or Cyclosporine A

Journal of dermatological science, 2017-09, Vol.87 (3), p.246-251

2017

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The Interleukin-1 Family

Advances in experimental medicine and biology, 2016, Vol.941, p.21-29

2016

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A role for mitochondria in NLRP3 inflammasome activation

NLRP1 Is the Key Inflammasome in Primary Human Keratinocytes

Inflammasome Activators Induce Interleukin-1α Secretion via Distinct Pathways with Differential Requirement for the Protease Function of Caspase-1

Molecular Mechanisms of Hair Growth and Regeneration: Current Understanding and Novel Paradigms

Nanoparticles activate the NLR pyrin domain containing 3 (Nlrp3) inflammasome and cause pulmonary inflammation through release of IL-1α and IL-1β

RIPK1 and TRADD Regulate TNF-Induced Signaling and Ripoptosome Formation

The NLRP1 inflammasome in skin diseases

Differential expression of NLRP3 among hematopoietic cells

Transcriptional adaptation of staphylococci during colonization of the authentic human environment: An overview of transcriptomic changes and their relationship to physiological conditions

Tissue-specific opposing functions of the inflammasome adaptor ASC in the regulation of epithelial skin carcinogenesis

Silencing of ASC in Cutaneous Squamous Cell Carcinoma

Macrophage migration inhibitory factor (MIF) and its homolog D-dopachrome tautomerase (D-DT) are significant promotors of UVB- but not chemically induced non-melanoma skin cancer

Innate immune activation as cofactor in pemphigus disease manifestation

Apremilast prevents blistering in human epidermis and stabilizes keratinocyte adhesion in pemphigus

Activation of hypoxia inducible factor 1 is a general phenomenon in infections with human pathogens

The Staphylococcus epidermidis Transcriptional Profile During Carriage

TNF Is Partially Required for Cell-Death-Triggered Skin Inflammation upon Acute Loss of cFLIP

Induction of skin-pathogenic Th22 cells by epicutaneous allergen exposure

Author Correction: Apremilast prevents blistering in human epidermis and stabilizes keratinocyte adhesion in pemphigus

Role of ADAM10 and ADAM17 in the Regulation of Keratinocyte Adhesion in Pemphigus Vulgaris

MALT1 Protease Activity Controls the Expression of Inflammatory Genes in Keratinocytes upon Zymosan Stimulation

ST18 Enhances PV-IgG-Induced Loss of Keratinocyte Cohesion in Parallel to Increased ERK Activation

Pitfalls in the Application of Dispase-Based Keratinocyte Dissociation Assay for In Vitro Analysis of Pemphigus Vulgaris

Differential induction of ATF3 and HO-1 in myeloid cells and keratinocytes via Dimethylfumarate or Cyclosporine A

The Interleukin-1 Family

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