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iNOS as a Driver of Inflammation and Apoptosis in Mouse Skeletal Muscle after Burn Injury: Possible Involvement of Sirt1 S-Nitrosylation-Mediated Acetylation of p65 NF-κB and p53

PloS one, 2017, Vol.12 (1), p.e0170391-e0170391

2017

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Mito-kaede photoactivation and chase experiment for mitophagy: optimizing flux measurement via fluid exchange system

BioTechniques, 2024-08, Vol.76 (8), p.381-393

2024

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Primary role of functional ischemia, quantitative evidence for the two-hit mechanism, and phosphodiesterase-5 inhibitor therapy in mouse muscular dystrophy

PloS one, 2007-08, Vol.2 (8), p.e806-e806

2007

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Prevention of Burn-Induced Inflammatory Responses and Muscle Wasting By GTS-21, A Specific Agonist for α7 Nicotinic Acetylcholine Receptors

Shock (Augusta, Ga.), 2017-01, Vol.47 (1), p.61-69

2017

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Burn-induced muscle metabolic derangements and mitochondrial dysfunction are associated with activation of HIF-1α and mTORC1: Role of protein farnesylation

Scientific reports, 2017-07, Vol.7 (1), p.6618-14, Article 6618

2017

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Coenzyme Q10 protects against burn‐induced mitochondrial dysfunction and impaired insulin signaling in mouse skeletal muscle

FEBS open bio, 2019-02, Vol.9 (2), p.348-363

2019

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Title efficacy of phosphodiesterase 5 inhibitor on distant burn-induced muscle autophagy, microcirculation, and survival rate

American journal of physiology: endocrinology and metabolism, 2013-05, Vol.304 (9), p.E922-E933

2013

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Inducible Nitric-oxide Synthase and NO Donor Induce Insulin Receptor Substrate-1 Degradation in Skeletal Muscle Cells

The Journal of biological chemistry, 2005-04, Vol.280 (14), p.14203-14211

2005

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Myostatin deficiency not only prevents muscle wasting but also improves survival in septic mice

American journal of physiology: endocrinology and metabolism, 2021-01, Vol.320 (1), p.E150-E159

2021

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Burn injury impairs insulin-stimulated Akt/PKB activation in skeletal muscle

American journal of physiology: endocrinology and metabolism, 2005-03, Vol.288 (3), p.E585-E591

2005

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Mitochondria, endoplasmic reticulum, and alternative pathways of cell death in critical illness

Critical care medicine, 2007-09, Vol.35 (9 Suppl), p.S488-S495

2007

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Recombinant Expression of Caveolin-1 in Oncogenically Transformed Cells Abrogates Anchorage-independent Growth

The Journal of biological chemistry, 1997-06, Vol.272 (26), p.16374-16381

1997

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Inducible nitric oxide synthase plays a role in LPS-induced hyperglycemia and insulin resistance

American journal of physiology: endocrinology and metabolism, 2002-02, Vol.282 (2), p.E386-E394

2002

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After a Heart Attack, Who Should Care?

Critical care medicine, 2022-06, Vol.50 (6), p.1030-1032

2022

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104 Parkin-independent Pathway of Mitophagy as a Potential Target Mitochondrial Dysfunctions in Burns

Journal of burn care & research, 2022-03, Vol.43 (Supplement_1), p.S68-S69

2022

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99 Feasibility of Deep Learning-based Automatic Myofiber Size Measurement for Burn-induced Muscle Wasting and Its Reversal

Journal of burn care & research, 2022-03, Vol.43 (Supplement_1), p.S66-S66

2022

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840 Myostatin Deficiency Inhibits Muscle Wasting and Improves Bacterial Clearance and Survival in Septic Mice

Journal of burn care & research, 2020-03, Vol.41 (Supplement_1), p.S259-S260

2020

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Morphine induces inflammatory responses via both TLR4 and cGAS-STING signaling pathways

Cytokine (Philadelphia, Pa.), 2024-11, Vol.183, p.156737, Article 156737

2024

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Obesity-induced Insulin Resistance and Hyperglycemia

Anesthesiology (Philadelphia), 2008-07, Vol.109 (1), p.137-148

2008

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Obesity-induced Insulin Resistance and Hyperglycemia : Etiologic Factors and Molecular Mechanisms

Anesthesiology (Philadelphia), 2008-07, Vol.109 (1), p.137-148

2008

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100 Burn Causes Aberrant Macrophage Polarity Conversion in Skeletal Muscles, Which Is Mitigated by Auto/mitophagy-targeted Therapy

Journal of burn care & research, 2022-03, Vol.43 (Supplement_1), p.S66-S67

2022

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Mechanism of pulmonary immunosuppression: extrapulmonary burn injury suppresses bacterial endotoxin‐induced pulmonary neutrophil recruitment and neutrophil extracellular trap (NET) formation

The FASEB journal, 2019-12, Vol.33 (12), p.13602-13616

2019

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Immobilization depresses insulin signaling in skeletal muscle

American journal of physiology: endocrinology and metabolism, 2000-12, Vol.279 (6), p.E1235-E1241

2000

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Burn-Induced Microglia Activation is Associated With Motor Neuron Degeneration and Muscle Wasting in Mice

Shock (Augusta, Ga.), 2019-05, Vol.51 (5), p.569-579

2019

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43 Activation of CCL2/CCR2 Signaling Axis Is Responsible for Spinal Cord Inflammation and Loss of Muscle Mass in Mice After Burn Injury

Journal of burn care & research, 2020-03, Vol.41 (Supplement_1), p.S28-S29

2020

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iNOS as a Driver of Inflammation and Apoptosis in Mouse Skeletal Muscle after Burn Injury: Possible Involvement of Sirt1 S-Nitrosylation-Mediated Acetylation of p65 NF-κB and p53

Mito-kaede photoactivation and chase experiment for mitophagy: optimizing flux measurement via fluid exchange system

Primary role of functional ischemia, quantitative evidence for the two-hit mechanism, and phosphodiesterase-5 inhibitor therapy in mouse muscular dystrophy

Prevention of Burn-Induced Inflammatory Responses and Muscle Wasting By GTS-21, A Specific Agonist for α7 Nicotinic Acetylcholine Receptors

Burn-induced muscle metabolic derangements and mitochondrial dysfunction are associated with activation of HIF-1α and mTORC1: Role of protein farnesylation

Coenzyme Q10 protects against burn‐induced mitochondrial dysfunction and impaired insulin signaling in mouse skeletal muscle

Title efficacy of phosphodiesterase 5 inhibitor on distant burn-induced muscle autophagy, microcirculation, and survival rate

Inducible Nitric-oxide Synthase and NO Donor Induce Insulin Receptor Substrate-1 Degradation in Skeletal Muscle Cells

Myostatin deficiency not only prevents muscle wasting but also improves survival in septic mice

Burn injury impairs insulin-stimulated Akt/PKB activation in skeletal muscle

Mitochondria, endoplasmic reticulum, and alternative pathways of cell death in critical illness

Recombinant Expression of Caveolin-1 in Oncogenically Transformed Cells Abrogates Anchorage-independent Growth

Inducible nitric oxide synthase plays a role in LPS-induced hyperglycemia and insulin resistance

After a Heart Attack, Who Should Care?

104 Parkin-independent Pathway of Mitophagy as a Potential Target Mitochondrial Dysfunctions in Burns

99 Feasibility of Deep Learning-based Automatic Myofiber Size Measurement for Burn-induced Muscle Wasting and Its Reversal

840 Myostatin Deficiency Inhibits Muscle Wasting and Improves Bacterial Clearance and Survival in Septic Mice

Morphine induces inflammatory responses via both TLR4 and cGAS-STING signaling pathways

Obesity-induced Insulin Resistance and Hyperglycemia

Obesity-induced Insulin Resistance and Hyperglycemia : Etiologic Factors and Molecular Mechanisms

100 Burn Causes Aberrant Macrophage Polarity Conversion in Skeletal Muscles, Which Is Mitigated by Auto/mitophagy-targeted Therapy

Mechanism of pulmonary immunosuppression: extrapulmonary burn injury suppresses bacterial endotoxin‐induced pulmonary neutrophil recruitment and neutrophil extracellular trap (NET) formation

Immobilization depresses insulin signaling in skeletal muscle

Burn-Induced Microglia Activation is Associated With Motor Neuron Degeneration and Muscle Wasting in Mice

43 Activation of CCL2/CCR2 Signaling Axis Is Responsible for Spinal Cord Inflammation and Loss of Muscle Mass in Mice After Burn Injury

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