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Early deficits in synaptic mitochondria in an Alzheimer’s disease mouse model

Proceedings of the National Academy of Sciences - PNAS, 2010-10, Vol.107 (43), p.18670-18675

2010

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Mitochondrial permeability transition pore in Alzheimer's disease: Cyclophilin D and amyloid beta

Biochimica et biophysica acta, 2010-01, Vol.1802 (1), p.198-204

2010

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Astrocytes Attenuate Mitochondrial Dysfunctions in Human Dopaminergic Neurons Derived from iPSC

Stem cell reports, 2018-02, Vol.10 (2), p.366-374

2018

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Development and Dynamic Regulation of Mitochondrial Network in Human Midbrain Dopaminergic Neurons Differentiated from iPSCs

Stem cell reports, 2016-10, Vol.7 (4), p.678-692

2016

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Mitochondrial permeability transition pore is a potential drug target for neurodegeneration

Biochimica et biophysica acta, 2014-08, Vol.1842 (8), p.1267-1272

2014

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Ginsenoside Rg1 Attenuates Oligomeric Aβ1-42-Induced Mitochondrial Dysfunction

Current Alzheimer research, 2012-03, Vol.9 (3), p.388-395

2012

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Drp1-mediated mitochondrial abnormalities link to synaptic injury in diabetes model

Diabetes (New York, N.Y.), 2015-05, Vol.64 (5), p.1728-1742

2015

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Mitochondrial Dysfunction, Endoplasmic Reticulum Stress, and Apoptosis in Alzheimer’s Disease

Journal of Pharmacological Sciences, 2005, Vol.97(3), pp.312-316

2005

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Inhibition of ERK-DLP1 signaling and mitochondrial division alleviates mitochondrial dysfunction in Alzheimer's disease cybrid cell

Biochimica et biophysica acta, 2014-02, Vol.1842 (2), p.220-231

2014

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Overexpression of endophilin A1 exacerbates synaptic alterations in a mouse model of Alzheimer's disease

Nature communications, 2018-07, Vol.9 (1), p.2968-14, Article 2968

2018

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High Dietary Advanced Glycation End Products Impair Mitochondrial and Cognitive Function

Journal of Alzheimer's disease, 2020-01, Vol.76 (1), p.165-178

2020

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Hypertension Induces Brain β-Amyloid Accumulation, Cognitive Impairment, and Memory Deterioration Through Activation of Receptor for Advanced Glycation End Products in Brain Vasculature

Hypertension (Dallas, Tex. 1979), 2012-07, Vol.60 (1), p.188-197

2012

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RAGE-mediated signaling contributes to intraneuronal transport of amyloid-β and neuronal dysfunction

Proceedings of the National Academy of Sciences - PNAS, 2009-11, Vol.106 (47), p.20021-20026

2009

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Cyclophilin D deficiency rescues axonal mitochondrial transport in Alzheimer's neurons

PloS one, 2013-01, Vol.8 (1), p.e54914

2013

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ABAD Directly Links Aβ to Mitochondrial Toxicity in Alzheimer's Disease

Science (American Association for the Advancement of Science), 2004-04, Vol.304 (5669), p.448-452

2004

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Unlocking the Door to Neuronal Woes in Alzheimer's Disease: Aβ and Mitochondrial Permeability Transition Pore

Pharmaceuticals (Basel, Switzerland), 2010-06, Vol.3 (6), p.1936-1948

2010

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NR2B-dependent cyclophilin D translocation suppresses the recovery of synaptic transmission after oxygen–glucose deprivation

Biochimica et biophysica acta. Molecular basis of disease, 2015-10, Vol.1852 (10), p.2225-2234

2015

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The potential role of damage-associated molecular patterns derived from mitochondria in osteocyte apoptosis and bone remodeling

Bone (New York, N.Y.), 2014-05, Vol.62, p.67-68

2014

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Identification of a Small Molecule Cyclophilin D Inhibitor for Rescuing Aβ-Mediated Mitochondrial Dysfunction

ACS medicinal chemistry letters, 2016-03, Vol.7 (3), p.294-299

2016

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Blockade of Late Stages of Autoimmune Diabetes by Inhibition of the Receptor for Advanced Glycation End Products

The Journal of immunology (1950), 2004-07, Vol.173 (2), p.1399-1405

2004

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Geniposide Attenuates Oligomeric Aβ 1-42 -Induced Inflammatory Response by Targeting RAGE-Dependent Signaling in BV2 Cells

Current Alzheimer research, 2014-06, Vol.11 (5), p.430-440

2014

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High-resolution crystal structures of two crystal forms of human cyclophilin D in complex with PEG 400 molecules

Acta crystallographica. Section F, Structural biology communications, 2014-06, Vol.70 (6), p.717-722

2014

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Synaptic mitochondrial pathology in Alzheimer's disease

Antioxidants & redox signaling, 2012-06, Vol.16 (12), p.1467-1475

2012

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Mitochondrial permeability transition pore: a potential drug target for neurodegeneration

Drug discovery today, 2018-12, Vol.23 (12), p.1983-1989

2018

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Age-dependent accumulation of dicarbonyls and advanced glycation endproducts (AGEs) associates with mitochondrial stress

Free radical biology & medicine, 2021-02, Vol.164, p.429-438

2021

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Early deficits in synaptic mitochondria in an Alzheimer’s disease mouse model

Mitochondrial permeability transition pore in Alzheimer's disease: Cyclophilin D and amyloid beta

Astrocytes Attenuate Mitochondrial Dysfunctions in Human Dopaminergic Neurons Derived from iPSC

Development and Dynamic Regulation of Mitochondrial Network in Human Midbrain Dopaminergic Neurons Differentiated from iPSCs

Mitochondrial permeability transition pore is a potential drug target for neurodegeneration

Ginsenoside Rg1 Attenuates Oligomeric Aβ1-42-Induced Mitochondrial Dysfunction

Drp1-mediated mitochondrial abnormalities link to synaptic injury in diabetes model

Mitochondrial Dysfunction, Endoplasmic Reticulum Stress, and Apoptosis in Alzheimer’s Disease

Inhibition of ERK-DLP1 signaling and mitochondrial division alleviates mitochondrial dysfunction in Alzheimer's disease cybrid cell

Overexpression of endophilin A1 exacerbates synaptic alterations in a mouse model of Alzheimer's disease

High Dietary Advanced Glycation End Products Impair Mitochondrial and Cognitive Function

Hypertension Induces Brain β-Amyloid Accumulation, Cognitive Impairment, and Memory Deterioration Through Activation of Receptor for Advanced Glycation End Products in Brain Vasculature

RAGE-mediated signaling contributes to intraneuronal transport of amyloid-β and neuronal dysfunction

Cyclophilin D deficiency rescues axonal mitochondrial transport in Alzheimer's neurons

ABAD Directly Links Aβ to Mitochondrial Toxicity in Alzheimer's Disease

Unlocking the Door to Neuronal Woes in Alzheimer's Disease: Aβ and Mitochondrial Permeability Transition Pore

NR2B-dependent cyclophilin D translocation suppresses the recovery of synaptic transmission after oxygen–glucose deprivation

The potential role of damage-associated molecular patterns derived from mitochondria in osteocyte apoptosis and bone remodeling

Identification of a Small Molecule Cyclophilin D Inhibitor for Rescuing Aβ-Mediated Mitochondrial Dysfunction

Blockade of Late Stages of Autoimmune Diabetes by Inhibition of the Receptor for Advanced Glycation End Products

Geniposide Attenuates Oligomeric Aβ 1-42 -Induced Inflammatory Response by Targeting RAGE-Dependent Signaling in BV2 Cells

High-resolution crystal structures of two crystal forms of human cyclophilin D in complex with PEG 400 molecules

Synaptic mitochondrial pathology in Alzheimer's disease

Mitochondrial permeability transition pore: a potential drug target for neurodegeneration

Age-dependent accumulation of dicarbonyls and advanced glycation endproducts (AGEs) associates with mitochondrial stress

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