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A Warburg-like metabolic program coordinates Wnt, AMPK, and mTOR signaling pathways in epileptogenesis

PloS one, 2021-08, Vol.16 (8), p.e0252282

2021

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Epigallocatechin-3-gallate inhibits stem-like inflammatory breast cancer cells

PloS one, 2013-09, Vol.8 (9), p.e73464

2013

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HBP1-Mediated Transcriptional Regulation of DNA Methyltransferase 1 and Its Impact on Cell Senescence

Molecular and Cellular Biology, 2013-03, Vol.33 (5), p.887-903

2013

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N-acetylcysteine (NAC) inhibits cell growth by mediating the EGFR/Akt/HMG box-containing protein 1 (HBP1) signaling pathway in invasive oral cancer

Oral oncology, 2013-02, Vol.49 (2), p.129-135

2013

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The HBP1 Transcriptional Repressor Participates in RAS-Induced Premature Senescence

Molecular and Cellular Biology, 2006-11, Vol.26 (22), p.8252-8266

2006

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Conflicts of interest: Lack of policy and/or of common sense?

Lab animal, 2019-09, Vol.48 (9), p.247

Yee, Amy S

2019

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The HBP1 transcriptional repressor and the p38 MAP kinase: unlikely partners in G1 regulation and tumor suppression

Gene, 2004-07, Vol.336 (1), p.1-13

2004

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HBP1 Repression of the p47phox Gene: Cell Cycle Regulation via the NADPH Oxidase

Molecular and Cellular Biology, 2004-04, Vol.24 (7), p.3011-3024

2004

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The green tea compound, (−)-epigallocatechin-3-gallate downregulates N-cadherin and suppresses migration of bladder carcinoma cells

Journal of cellular biochemistry, 2007-10, Vol.102 (2), p.377-388

2007

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The Transcriptional Repressor HBP1 Is a Target of the p38 Mitogen-Activated Protein Kinase Pathway in Cell Cycle Regulation

Molecular and Cellular Biology, 2003-12, Vol.23 (23), p.8890-8901

2003

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Suppression of Wnt Signaling by the Green Tea Compound (-)-Epigallocatechin 3-Gallate (EGCG) in Invasive Breast Cancer Cells: REQUIREMENT OF THE TRANSCRIPTIONAL REPRESSOR HBP1

The Journal of biological chemistry, 2006-04, Vol.281 (16), p.10865-10875

2006

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Negative regulation of the Wnt-β-catenin pathway by the transcriptional repressor HBP1

The EMBO journal, 2001-08, Vol.20 (16), p.4500-4511

2001

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Alterations of the HBP1 transcriptional repressor are associated with invasive breast cancer

Cancer research (Chicago, Ill.), 2007-07, Vol.67 (13), p.6136-6145

2007

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Functions of Cyclin A1 in the Cell Cycle and Its Interactions with Transcription Factor E2F-1 and the Rb Family of Proteins

Molecular and Cellular Biology, 1999-03, Vol.19 (3), p.2400-2407

1999

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Regulation of Differentiation by HBP1, a Target of the Retinoblastoma Protein

Molecular and Cellular Biology, 1998-08, Vol.18 (8), p.4732-4743

1998

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HMG Box Transcriptional Repressor HBP1 Maintains a Proliferation Barrier in Differentiated Liver Tissue

Molecular and Cellular Biology, 2001-09, Vol.21 (17), p.5723-5732

2001

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Suppression of Wnt Signaling by the Green Tea Compound (–)-Epigallocatechin 3-Gallate (EGCG) in Invasive Breast Cancer Cells

The Journal of biological chemistry, 2006-04, Vol.281 (16), p.10865-10875

2006

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Multiple Changes in E2F Function and Regulation Occur upon Muscle Differentiation

Molecular and cellular biology, 1995-04, Vol.15 (4), p.2252-2262

1995

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The nitrone spin trap PBN alters the cellular response to H2O2: activation of the EGF receptor/ERK pathway

Free radical biology & medicine, 2002-03, Vol.32 (6), p.551-561

2002

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Abstract A39: A therapeutic strategy to inhibit Wnt signaling also reprograms breast tumor-immune cell interactions: Perspectives for conferring immune checkpoint inhibitor susceptibility

Cancer immunology research, 2020-03, Vol.8 (3_Supplement), p.A39-A39

2020

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The N-terminal Region of E2F-1 Is Required for Transcriptional Activation of a New Class of Target Promoter (∗)

The Journal of biological chemistry, 1996-05, Vol.271 (21), p.12261-12268

1996

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Abstract A94: Epigallocatechin-3-gallate inhibits stem-like inflammatory breast cancer cells

Cancer research (Chicago, Ill.), 2013-02, Vol.73 (3_Supplement), p.A94-A94

2013

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Abstract 5624: HMG box-containing protein 1 (HBP1) participates in the antitumor effect of N-acetylcysteine (NAC) in invasive oral cancer

Cancer research (Chicago, Ill.), 2012-04, Vol.72 (8_Supplement), p.5624-5624

2012

Volltextzugriff (PDF)

Abstract B030: The HBP1 transcriptional repressor: An unexpected window on breast cancer metabolism in triple-negative breast cancer

Molecular cancer research, 2013-10, Vol.11 (10_Supplement), p.B030-B030

2013

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Abstract B260: Wnt signaling, triple-negative breast cancer, and green tea

Molecular cancer therapeutics, 2013-11, Vol.12 (11_Supplement), p.B260-B260

2013

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A Warburg-like metabolic program coordinates Wnt, AMPK, and mTOR signaling pathways in epileptogenesis

Epigallocatechin-3-gallate inhibits stem-like inflammatory breast cancer cells

HBP1-Mediated Transcriptional Regulation of DNA Methyltransferase 1 and Its Impact on Cell Senescence

N-acetylcysteine (NAC) inhibits cell growth by mediating the EGFR/Akt/HMG box-containing protein 1 (HBP1) signaling pathway in invasive oral cancer

The HBP1 Transcriptional Repressor Participates in RAS-Induced Premature Senescence

Conflicts of interest: Lack of policy and/or of common sense?

The HBP1 transcriptional repressor and the p38 MAP kinase: unlikely partners in G1 regulation and tumor suppression

HBP1 Repression of the p47phox Gene: Cell Cycle Regulation via the NADPH Oxidase

The green tea compound, (−)-epigallocatechin-3-gallate downregulates N-cadherin and suppresses migration of bladder carcinoma cells

The Transcriptional Repressor HBP1 Is a Target of the p38 Mitogen-Activated Protein Kinase Pathway in Cell Cycle Regulation

Suppression of Wnt Signaling by the Green Tea Compound (-)-Epigallocatechin 3-Gallate (EGCG) in Invasive Breast Cancer Cells: REQUIREMENT OF THE TRANSCRIPTIONAL REPRESSOR HBP1

Negative regulation of the Wnt-β-catenin pathway by the transcriptional repressor HBP1

Alterations of the HBP1 transcriptional repressor are associated with invasive breast cancer

Functions of Cyclin A1 in the Cell Cycle and Its Interactions with Transcription Factor E2F-1 and the Rb Family of Proteins

Regulation of Differentiation by HBP1, a Target of the Retinoblastoma Protein

HMG Box Transcriptional Repressor HBP1 Maintains a Proliferation Barrier in Differentiated Liver Tissue

Suppression of Wnt Signaling by the Green Tea Compound (–)-Epigallocatechin 3-Gallate (EGCG) in Invasive Breast Cancer Cells

Multiple Changes in E2F Function and Regulation Occur upon Muscle Differentiation

The nitrone spin trap PBN alters the cellular response to H2O2: activation of the EGF receptor/ERK pathway

Abstract A39: A therapeutic strategy to inhibit Wnt signaling also reprograms breast tumor-immune cell interactions: Perspectives for conferring immune checkpoint inhibitor susceptibility

The N-terminal Region of E2F-1 Is Required for Transcriptional Activation of a New Class of Target Promoter (∗)

Abstract A94: Epigallocatechin-3-gallate inhibits stem-like inflammatory breast cancer cells

Abstract 5624: HMG box-containing protein 1 (HBP1) participates in the antitumor effect of N-acetylcysteine (NAC) in invasive oral cancer

Abstract B030: The HBP1 transcriptional repressor: An unexpected window on breast cancer metabolism in triple-negative breast cancer

Abstract B260: Wnt signaling, triple-negative breast cancer, and green tea

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