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Caspase-1 inhibition by VX-765 administered at reperfusion in P2Y12 receptor antagonist-treated rats provides long-term reduction in myocardial infarct size and preservation of ventricular function

Basic research in cardiology, 2018-09, Vol.113 (5), p.1-15, Article 32

2018

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Structure and entropy control of polyamorphous transition in high-entropy metallic glasses

Acta materialia, 2024-03, Vol.266, p.119701, Article 119701

2024

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Lethal Caspase-1/4-Dependent Injury Occurs in the First Minutes of Coronary Reperfusion and Requires Calpain Activity

International journal of molecular sciences, 2023-02, Vol.24 (4), p.3801

2023

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Multiple, brief coronary occlusions during early reperfusion protect rabbit hearts by targeting cell signaling pathways

Journal of the American College of Cardiology, 2004-09, Vol.44 (5), p.1103-1110

2004

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Platelet P2Y12 Blockers Confer Direct Postconditioning-Like Protection in Reperfused Rabbit Hearts

Journal of cardiovascular pharmacology and therapeutics, 2013-05, Vol.18 (3), p.251-262

2013

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Nickel Isotope Fractionation During Magmatic Differentiation

Geochemistry, geophysics, geosystems : G3, 2023-06, Vol.24 (6), p.n/a

2023

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Triple Therapy Greatly Increases Myocardial Salvage During Ischemia/Reperfusion in the in situ Rat Heart

Cardiovascular drugs and therapy, 2013-10, Vol.27 (5), p.403-412

2013

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Postconditioning's protection is not dependent on circulating blood factors or cells but involves adenosine receptors and requires PI3-kinase and guanylyl cyclase activation

Basic research in cardiology, 2005-01, Vol.100 (1), p.57-63

2005

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Mitochondrially targeted Endonuclease III has a powerful anti-infarct effect in an in vivo rat model of myocardial ischemia/reperfusion

Basic research in cardiology, 2015-03, Vol.110 (2), p.3-13, Article 3

2015

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Two Classes of Anti-Platelet Drugs Reduce Anatomical Infarct Size in Monkey Hearts

Cardiovascular drugs and therapy, 2013-04, Vol.27 (2), p.109-115

2013

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Nerve trunk healing and neuroma formation after nerve transection injury

Frontiers in neurology, 2023-06, Vol.14, p.1184246-1184246

2023

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Sulfide Dissolution on the Nickel Isotopic Composition of Basaltic Rocks

Journal of geophysical research. Solid earth, 2022-08, Vol.127 (8), p.n/a

2022

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Atrial natriuretic peptide administered just prior to reperfusion limits infarction in rabbit hearts

Basic research in cardiology, 2006-07, Vol.101 (4), p.311-318

2006

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Protein kinase C protects preconditioned rabbit hearts by increasing sensitivity of adenosine A2b -dependent signaling during early reperfusion

Journal of molecular and cellular cardiology, 2007-09, Vol.43 (3), p.262-271

2007

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Cardioprotection by mild hypothermia during ischemia involves preservation of ERK activity

Basic research in cardiology, 2011-05, Vol.106 (3), p.421-430

2011

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Epidemiology and antifungal susceptibilities of yeast isolates causing invasive infections across urban Beijing, China

Future microbiology, 2017-09, Vol.12 (12), p.1075-1086

2017

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The ph hypothesis of postconditioning : Staccato reperfusion reintroduces oxygen and perpetuates myocardial acidosis

Circulation (New York, N.Y.), 2007-04, Vol.115 (14), p.1895-1903

2007

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A measurement system for vertical seawater profiles close to the air–sea interface

Ocean science, 2017-09, Vol.13 (5), p.649-660

2017

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BAY 58-2667, a nitric oxide-independent guanylyl cyclase activator, pharmacologically post-conditions rabbit and rat hearts

European heart journal, 2009-07, Vol.30 (13), p.1607-1613

2009

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Infarct limitation by a protein kinase G activator at reperfusion in rabbit hearts is dependent on sensitizing the heart to A2b agonists by protein kinase C

American journal of physiology. Heart and circulatory physiology, 2008-09, Vol.295 (3), p.H1288-H1295

2008

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Role of Bradykinin in Protection of Ischemic Preconditioning in Rabbit Hearts

Circulation research, 1995-09, Vol.77 (3), p.611-621

1995

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Gamma secretase activating protein promotes end-organ dysfunction after bacterial pneumonia

American journal of physiology. Lung cellular and molecular physiology, 2023-08, Vol.325 (2), p.L174-L189

2023

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Acidosis, oxygen, and interference with mitochondrial permeability transition pore formation in the early minutes of reperfusion are critical to postconditioning’s success

Basic research in cardiology, 2008-09, Vol.103 (5), p.464-471

2008

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A2B adenosine receptors inhibit superoxide production from mitochondrial complex I in rabbit cardiomyocytes via a mechanism sensitive to Pertussis toxin

British journal of pharmacology, 2011-07, Vol.163 (5), p.995-1006

2011

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Postconditioning protects rabbit hearts through a protein kinase C-adenosine A2b receptor cascade

Cardiovascular research, 2006-05, Vol.70 (2), p.308-314

2006

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Caspase-1 inhibition by VX-765 administered at reperfusion in P2Y12 receptor antagonist-treated rats provides long-term reduction in myocardial infarct size and preservation of ventricular function

Structure and entropy control of polyamorphous transition in high-entropy metallic glasses

Lethal Caspase-1/4-Dependent Injury Occurs in the First Minutes of Coronary Reperfusion and Requires Calpain Activity

Multiple, brief coronary occlusions during early reperfusion protect rabbit hearts by targeting cell signaling pathways

Platelet P2Y12 Blockers Confer Direct Postconditioning-Like Protection in Reperfused Rabbit Hearts

Nickel Isotope Fractionation During Magmatic Differentiation

Triple Therapy Greatly Increases Myocardial Salvage During Ischemia/Reperfusion in the in situ Rat Heart

Postconditioning's protection is not dependent on circulating blood factors or cells but involves adenosine receptors and requires PI3-kinase and guanylyl cyclase activation

Mitochondrially targeted Endonuclease III has a powerful anti-infarct effect in an in vivo rat model of myocardial ischemia/reperfusion

Two Classes of Anti-Platelet Drugs Reduce Anatomical Infarct Size in Monkey Hearts

Nerve trunk healing and neuroma formation after nerve transection injury

Sulfide Dissolution on the Nickel Isotopic Composition of Basaltic Rocks

Atrial natriuretic peptide administered just prior to reperfusion limits infarction in rabbit hearts

Protein kinase C protects preconditioned rabbit hearts by increasing sensitivity of adenosine A2b -dependent signaling during early reperfusion

Cardioprotection by mild hypothermia during ischemia involves preservation of ERK activity

Epidemiology and antifungal susceptibilities of yeast isolates causing invasive infections across urban Beijing, China

The ph hypothesis of postconditioning : Staccato reperfusion reintroduces oxygen and perpetuates myocardial acidosis

A measurement system for vertical seawater profiles close to the air–sea interface

BAY 58-2667, a nitric oxide-independent guanylyl cyclase activator, pharmacologically post-conditions rabbit and rat hearts

Infarct limitation by a protein kinase G activator at reperfusion in rabbit hearts is dependent on sensitizing the heart to A2b agonists by protein kinase C

Role of Bradykinin in Protection of Ischemic Preconditioning in Rabbit Hearts

Gamma secretase activating protein promotes end-organ dysfunction after bacterial pneumonia

Acidosis, oxygen, and interference with mitochondrial permeability transition pore formation in the early minutes of reperfusion are critical to postconditioning’s success

A2B adenosine receptors inhibit superoxide production from mitochondrial complex I in rabbit cardiomyocytes via a mechanism sensitive to Pertussis toxin

Postconditioning protects rabbit hearts through a protein kinase C-adenosine A2b receptor cascade

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