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Induced Pluripotent Stem Cell (iPSC)–Derived Extracellular Vesicles Are Safer and More Effective for Cardiac Repair Than iPSCs

Circulation research, 2018-01, Vol.122 (2), p.296-309

2018

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Deletion of Interleukin-6 Attenuates Pressure Overload-Induced Left Ventricular Hypertrophy and Dysfunction

Circulation research, 2016-06, Vol.118 (12), p.1918-1929

2016

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Epigenetic modifiers reduce inflammation and modulate macrophage phenotype during endotoxemia-induced acute lung injury

Journal of cell science, 2015-08, Vol.128 (16), p.3094-3105

2015

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STAT3 balances myocyte hypertrophy vis-à-vis autophagy in response to Angiotensin II by modulating the AMPKα/mTOR axis

PloS one, 2017-07, Vol.12 (7), p.e0179835

2017

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Hydrothermal Synthesis of Functionalized Carbon Nanodots and Their Clusters as Ionic Probe for High Sensitivity and Selectivity for Sulfate Anions with Excellent Detection Level

Polymers, 2023-06, Vol.15 (12), p.2655

2023

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NAPSB as a predictive marker for prognosis and therapy associated with an immuno-hot tumor microenvironment in hepatocellular carcinoma

BMC gastroenterology, 2022-08, Vol.22 (1), p.1-392, Article 392

2022

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O-linked β-N-acetylglucosamine transferase is indispensable in the failing heart

Proceedings of the National Academy of Sciences - PNAS, 2010-10, Vol.107 (41), p.17797-17802

2010

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A murine model of inducible, cardiac-specific deletion of STAT3: Its use to determine the role of STAT3 in the upregulation of cardioprotective proteins by ischemic preconditioning

Journal of molecular and cellular cardiology, 2011-04, Vol.50 (4), p.589-597

2011

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Carbon monoxide induces a late preconditioning-mimetic cardioprotective and antiapoptotic milieu in the myocardium

Journal of molecular and cellular cardiology, 2012-01, Vol.52 (1), p.228-236

2012

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Effect of Solvent on Fluorescence Emission from Polyethylene Glycol-Coated Graphene Quantum Dots under Blue Light Illumination

Nanomaterials (Basel, Switzerland), 2021-05, Vol.11 (6), p.1383

2021

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Cyclooxygenase-2 Mediates the Cardioprotective Effects of the Late Phase of Ischemic Preconditioning in Conscious Rabbits

Proceedings of the National Academy of Sciences - PNAS, 2000-08, Vol.97 (18), p.10197-10202

2000

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Protein glutathiolation by nitric oxide: an intracellular mechanism regulating redox protein modification

The FASEB journal, 2006-08, Vol.20 (10), p.1715-1717

2006

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Role of the JAK–STAT Pathway in Protection Against Myocardial Ischemia/Reperfusion Injury

Trends in Cardiovascular Medicine, 2003-02, Vol.13 (2), p.72-79

2003

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Cardiac Myocyte-Specific Expression of Inducible Nitric Oxide Synthase Protects Against Ischemia/Reperfusion Injury by Preventing Mitochondrial Permeability Transition

Circulation (New York, N.Y.), 2008-11, Vol.118 (19), p.1970-1978

2008

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An Essential Role of the JAK-STAT Pathway in Ischemic Preconditioning

Proceedings of the National Academy of Sciences - PNAS, 2001-07, Vol.98 (16), p.9050-9055

2001

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Hematopoietic cytokines for cardiac repair: mobilization of bone marrow cells and beyond

Basic research in cardiology, 2011-09, Vol.106 (5), p.709-733

2011

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Inducible Nitric Oxide Synthase Modulates Cyclooxygenase-2 Activity in the Heart of Conscious Rabbits During the Late Phase of Ischemic Preconditioning

Circulation research, 2002-03, Vol.90 (5), p.602-608

2002

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The late phase of preconditioning and its natural clinical application--gene therapy

Heart failure reviews, 2007-12, Vol.12 (3-4), p.189-199

2007

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Exercise-induced late preconditioning in mice is triggered by eNOS-dependent generation of nitric oxide and activation of PKCε and is mediated by increased iNOS activity

International journal of cardiology, 2021-10, Vol.340, p.68-78

2021

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Correction: STAT3 balances myocyte hypertrophy vis-à-vis autophagy in response to Angiotensin II by modulating the AMPKα/mTOR axis

PloS one, 2023-11, Vol.18 (11), p.e0294366-e0294366

2023

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The Late Phase of Ischemic Preconditioning Is Abrogated by Targeted Disruption of the Inducible NO Synthase Gene

Proceedings of the National Academy of Sciences - PNAS, 1999-09, Vol.96 (20), p.11507-11512

1999

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Divergent Tumor Necrosis Factor Receptor-Related Remodeling Responses in Heart Failure : Role of Nuclear Factor-κB and Inflammatory Activation

Circulation (New York, N.Y.), 2009-03, Vol.119 (10), p.1386-1397

2009

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Fluorescent nitrogen-doped carbon nanodots synthesized through a hydrothermal method with different isomers

Journal of the Taiwan Institute of Chemical Engineers, 2021-06, Vol.123, p.302-309

2021

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Aldose Reductase Is an Obligatory Mediator of the Late Phase of Ischemic Preconditioning

Circulation research, 2002-08, Vol.91 (3), p.240-246

2002

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Endoplasmic reticulum stress-dependent activation of ATF3 mediates the late phase of ischemic preconditioning

Journal of molecular and cellular cardiology, 2014-11, Vol.76, p.138-147

2014

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Induced Pluripotent Stem Cell (iPSC)–Derived Extracellular Vesicles Are Safer and More Effective for Cardiac Repair Than iPSCs

Deletion of Interleukin-6 Attenuates Pressure Overload-Induced Left Ventricular Hypertrophy and Dysfunction

Epigenetic modifiers reduce inflammation and modulate macrophage phenotype during endotoxemia-induced acute lung injury

STAT3 balances myocyte hypertrophy vis-à-vis autophagy in response to Angiotensin II by modulating the AMPKα/mTOR axis

Hydrothermal Synthesis of Functionalized Carbon Nanodots and Their Clusters as Ionic Probe for High Sensitivity and Selectivity for Sulfate Anions with Excellent Detection Level

NAPSB as a predictive marker for prognosis and therapy associated with an immuno-hot tumor microenvironment in hepatocellular carcinoma

O-linked β-N-acetylglucosamine transferase is indispensable in the failing heart

A murine model of inducible, cardiac-specific deletion of STAT3: Its use to determine the role of STAT3 in the upregulation of cardioprotective proteins by ischemic preconditioning

Carbon monoxide induces a late preconditioning-mimetic cardioprotective and antiapoptotic milieu in the myocardium

Effect of Solvent on Fluorescence Emission from Polyethylene Glycol-Coated Graphene Quantum Dots under Blue Light Illumination

Cyclooxygenase-2 Mediates the Cardioprotective Effects of the Late Phase of Ischemic Preconditioning in Conscious Rabbits

Protein glutathiolation by nitric oxide: an intracellular mechanism regulating redox protein modification

Role of the JAK–STAT Pathway in Protection Against Myocardial Ischemia/Reperfusion Injury

Cardiac Myocyte-Specific Expression of Inducible Nitric Oxide Synthase Protects Against Ischemia/Reperfusion Injury by Preventing Mitochondrial Permeability Transition

An Essential Role of the JAK-STAT Pathway in Ischemic Preconditioning

Hematopoietic cytokines for cardiac repair: mobilization of bone marrow cells and beyond

Inducible Nitric Oxide Synthase Modulates Cyclooxygenase-2 Activity in the Heart of Conscious Rabbits During the Late Phase of Ischemic Preconditioning

The late phase of preconditioning and its natural clinical application--gene therapy

Exercise-induced late preconditioning in mice is triggered by eNOS-dependent generation of nitric oxide and activation of PKCε and is mediated by increased iNOS activity

Correction: STAT3 balances myocyte hypertrophy vis-à-vis autophagy in response to Angiotensin II by modulating the AMPKα/mTOR axis

The Late Phase of Ischemic Preconditioning Is Abrogated by Targeted Disruption of the Inducible NO Synthase Gene

Divergent Tumor Necrosis Factor Receptor-Related Remodeling Responses in Heart Failure : Role of Nuclear Factor-κB and Inflammatory Activation

Fluorescent nitrogen-doped carbon nanodots synthesized through a hydrothermal method with different isomers

Aldose Reductase Is an Obligatory Mediator of the Late Phase of Ischemic Preconditioning

Endoplasmic reticulum stress-dependent activation of ATF3 mediates the late phase of ischemic preconditioning

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