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Direct reversal of DNA damage by mutant methyltransferase protein protects mice against dose-intensified chemotherapy and leads to in vivo selection of hematopoietic stem cells

Cancer research (Chicago, Ill.), 2000-09, Vol.60 (18), p.5187-5195

2000

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The Role of Qualitative Analysis of F18-FDG Positron Emission Tomography in Predicting Pathologic Response following Chemoradiotherapy in Patients with Esophageal Carcinoma

International journal of radiation oncology, biology, physics, 2009, Vol.75 (3), p.S271-S272

2009

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Combining Targeted Agents With Modern Radiotherapy in Soft Tissue Sarcomas

JNCI : Journal of the National Cancer Institute, 2014-11, Vol.106 (11), p.dju329-dju329

2014

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Dosimetric consequences of pancreatic tumor motion when predetermined treatment margins are employed during intensity-modulated radiation therapy

Journal of B.U. ON., 2012-07, Vol.17 (3), p.526-532

2012

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Down-regulation of survivin and hypoxia-inducible factor-1 α by β-elemene enhances the radiosensitivity of lung adenocarcinoma xenograft

Cancer biotherapy & radiopharmaceuticals, 2012-02, Vol.27 (1), p.56-64

2012

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The adaptive imbalance in base excision-repair enzymes generates microsatellite instability in chronic inflammation

The Journal of clinical investigation, 2003-12, Vol.112 (12), p.1887-1894

2003

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The role of p53 in base excision repair following genotoxic stress

Carcinogenesis (New York), 2004-01, Vol.25 (1), p.11-19

2004

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PV-0040: Patterns of care for the elderly with non-surgically treated stage III non-small cell lung cancer

Radiotherapy and oncology, 2018-04, Vol.127, p.S16-S16

2018

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Characterization of the P140K, PVP(138-140)MLK, and G156A O 6 -Methylguanine-DNA Methyltransferase Mutants: Implications for Drug Resistance Gene Therapy

Human gene therapy, 1999-11, Vol.10 (17), p.2769-2778

1999

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Degradation of the alkylated form of the DNA repair protein, O6-alkylguanine-DNA alkyltransferase

Carcinogenesis (New York), 2002-05, Vol.23 (5), p.823-830

2002

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The adaptive imbalance in base excision-repair enzymes generates microsatellite instability in chronic inflammation

The Journal of clinical investigation, 2004-02, Vol.113 (3), p.490-490

Hofseth, L. J.

2004

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Effect of O6-benzylguanine on alkylating agent-induced toxicity and mutagenicity in Chinese hamster ovary cells expressing wild-type and mutant O6-alkylguanine-DNA alkyltransferases

Cancer research (Chicago, Ill.), 2000-10, Vol.60 (19), p.5464-5469

2000

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Isolation of human O6-alkylguanine-DNA alkyltransferase mutants highly resistant to inactivation by O6-benzylguanine

Cancer research (Chicago, Ill.), 1998-05, Vol.58 (9), p.1936-1945

1998

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Alteration of the conserved residue tyrosine-158 to histidine renders human O6-alkylguanine-DNA alkyltransferase insensitive to the inhibitor O6-benzylguanine

Cancer research (Chicago, Ill.), 1999-04, Vol.59 (7), p.1514-1519

1999

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Degradation of the alkylated form of the DNA repair protein, O super(6)-alkylguanine-DNA alkyltransferase

Carcinogenesis (New York), 2002-05, Vol.23 (5), p.823-830

2002

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Protection of CHO cells by mutant forms of O6-alkylguanine-DNA alkyltransferase from killing by 1,3-Bis-(2-chloroethyl)-1-nitrosourea (BCNU) plus O6-benzylguanine or O6-benzyl-8-oxoguanine

Biochemical pharmacology, 1999-07, Vol.58 (2), p.237-244

1999

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Point mutations at multiple sites including highly conserved amino acids maintain activity, but render O6-alkylguanine-DNA alkyltransferase insensitive to O6-benzylguanine

Biochemical journal, 2000-04, Vol.347 (Pt 2), p.519-526

2000

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Point mutations at multiple sites including highly conserved amino acids maintain activity, but render O super(6)-alkylguanine-DNA alkyltransferase insensitive to O super(6)-benzylguanine

Biochemical journal, 2000-04, Vol.347 (2), p.519-526

2000

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Conserved residue lysine165 is essential for the ability of O6-alkylguanine-DNA alkyltransferase to react with O6-benzylguanine

Biochemical journal, 2000-04, Vol.347 (Pt 2), p.527-534

2000

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Alteration of the Conserved Residue Tyrosine-158 to Histidine Renders Human O super(6)-Alkylguanine-DNA Alkyltransferase Insensitive to the Inhibitor O super(6)-Benzylguanine

Cancer research (Chicago, Ill.), 1999-04, Vol.59 (7), p.1514-1519

1999

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A PI3K gene expression signature predicts for recurrence in early‐stage non–small cell lung cancer treated with stereotactic body radiation therapy

Cancer, 2023-12, Vol.129 (24), p.3971-3977

2023

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Conserved residue lysine super(165) is essential for the ability of O super(6)-alkylguanine-DNA alkyltransferase to react with O super(6)-benzylguanine

Biochemical journal, 2000-04, Vol.347 (2), p.527-534

2000

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Isolation of Human O super(6)-Alkylguanine-DNA Alkyltransferase Mutants Highly Resistant to Inactivation by O super(6)-Benzylguanine

Cancer research (Chicago, Ill.), 1998-05, Vol.58 (9), p.1936-1945

1998

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The role of human O(6)-alkylguanine-DNA alkyltransferase in promoting 1,2-dibromoethane-induced genotoxicity in Escherichia coli

Mutation research, 2000-07, Vol.452 (1), p.1

2000

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Characterization of the P140K, PVP(138-140)MLK, and G156A O super(6)-Methylguanine-DNA Methyltransferase Mutants: Implications for Drug Resistance Gene Therapy

Human gene therapy, 1999-11, Vol.10 (17), p.2769-2778

1999

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Direct reversal of DNA damage by mutant methyltransferase protein protects mice against dose-intensified chemotherapy and leads to in vivo selection of hematopoietic stem cells

The Role of Qualitative Analysis of F18-FDG Positron Emission Tomography in Predicting Pathologic Response following Chemoradiotherapy in Patients with Esophageal Carcinoma

Combining Targeted Agents With Modern Radiotherapy in Soft Tissue Sarcomas

Dosimetric consequences of pancreatic tumor motion when predetermined treatment margins are employed during intensity-modulated radiation therapy

Down-regulation of survivin and hypoxia-inducible factor-1 α by β-elemene enhances the radiosensitivity of lung adenocarcinoma xenograft

The adaptive imbalance in base excision-repair enzymes generates microsatellite instability in chronic inflammation

The role of p53 in base excision repair following genotoxic stress

PV-0040: Patterns of care for the elderly with non-surgically treated stage III non-small cell lung cancer

Characterization of the P140K, PVP(138-140)MLK, and G156A O 6 -Methylguanine-DNA Methyltransferase Mutants: Implications for Drug Resistance Gene Therapy

Degradation of the alkylated form of the DNA repair protein, O6-alkylguanine-DNA alkyltransferase

The adaptive imbalance in base excision-repair enzymes generates microsatellite instability in chronic inflammation

Effect of O6-benzylguanine on alkylating agent-induced toxicity and mutagenicity in Chinese hamster ovary cells expressing wild-type and mutant O6-alkylguanine-DNA alkyltransferases

Isolation of human O6-alkylguanine-DNA alkyltransferase mutants highly resistant to inactivation by O6-benzylguanine

Alteration of the conserved residue tyrosine-158 to histidine renders human O6-alkylguanine-DNA alkyltransferase insensitive to the inhibitor O6-benzylguanine

Degradation of the alkylated form of the DNA repair protein, O super(6)-alkylguanine-DNA alkyltransferase

Protection of CHO cells by mutant forms of O6-alkylguanine-DNA alkyltransferase from killing by 1,3-Bis-(2-chloroethyl)-1-nitrosourea (BCNU) plus O6-benzylguanine or O6-benzyl-8-oxoguanine

Point mutations at multiple sites including highly conserved amino acids maintain activity, but render O6-alkylguanine-DNA alkyltransferase insensitive to O6-benzylguanine

Point mutations at multiple sites including highly conserved amino acids maintain activity, but render O super(6)-alkylguanine-DNA alkyltransferase insensitive to O super(6)-benzylguanine

Conserved residue lysine165 is essential for the ability of O6-alkylguanine-DNA alkyltransferase to react with O6-benzylguanine

Alteration of the Conserved Residue Tyrosine-158 to Histidine Renders Human O super(6)-Alkylguanine-DNA Alkyltransferase Insensitive to the Inhibitor O super(6)-Benzylguanine

A PI3K gene expression signature predicts for recurrence in early‐stage non–small cell lung cancer treated with stereotactic body radiation therapy

Conserved residue lysine super(165) is essential for the ability of O super(6)-alkylguanine-DNA alkyltransferase to react with O super(6)-benzylguanine

Isolation of Human O super(6)-Alkylguanine-DNA Alkyltransferase Mutants Highly Resistant to Inactivation by O super(6)-Benzylguanine

The role of human O(6)-alkylguanine-DNA alkyltransferase in promoting 1,2-dibromoethane-induced genotoxicity in Escherichia coli

Characterization of the P140K, PVP(138-140)MLK, and G156A O super(6)-Methylguanine-DNA Methyltransferase Mutants: Implications for Drug Resistance Gene Therapy

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