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Protective Effects of Pituitary Adenylate Cyclase-Activating Polypeptide and Vasoactive Intestinal Peptide Against Cognitive Decline in Neurodegenerative Diseases

Frontiers in cellular neuroscience, 2020-07, Vol.14, p.221-221

2020

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Novel epigallocatechin-3-gallate (EGCG) derivative as a new therapeutic strategy for reducing neuropathic pain after chronic constriction nerve injury in mice

PloS one, 2015-04, Vol.10 (4), p.e0123122-e0123122

2015

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Prostaglandin E2 EP2 activation reduces memory decline in R6/1 mouse model of Huntington's disease by the induction of BDNF-dependent synaptic plasticity

Neurobiology of disease, 2016-11, Vol.95, p.22-34

2016

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Increased PKA signaling disrupts recognition memory and spatial memory: role in Huntington's disease

Human molecular genetics, 2011-11, Vol.20 (21), p.4232-4247

2011

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Brain–gut photobiomodulation restores cognitive alterations in chronically stressed mice through the regulation of Sirt1 and neuroinflammation

Journal of affective disorders, 2024-06, Vol.354, p.574-588

2024

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Pituitary Adenylate Cyclase-Activating Polypeptide (PACAP) Protects Striatal Cells and Improves Motor Function in Huntington's Disease Models: Role of PAC1 Receptor

Frontiers in pharmacology, 2022-01, Vol.12, p.797541-797541

2022

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Decreased Myocyte Enhancer Factor 2 Levels in the Hippocampus of Huntington’s Disease Mice Are Related to Cognitive Dysfunction

Molecular neurobiology, 2020-11, Vol.57 (11), p.4549-4562

2020

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Human alpha 1-antitrypsin protects neurons and glial cells against oxygen and glucose deprivation through inhibition of interleukins expression

Biochimica et biophysica acta. General subjects, 2018-09, Vol.1862 (9), p.1852-1861

2018

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Prostaglandin E2 EP1 Receptor Antagonist Improves Motor Deficits and Rescues Memory Decline in R6/1 Mouse Model of Huntington's Disease

Molecular neurobiology, 2014-04, Vol.49 (2), p.784-795

2014

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Striatal-enriched protein tyrosine phosphatase expression and activity in Huntington's disease: a STEP in the resistance to excitotoxicity

The Journal of neuroscience, 2011-06, Vol.31 (22), p.8150-8162

2011

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Altered cholesterol homeostasis contributes to enhanced excitotoxicity in Huntington's disease

Journal of neurochemistry, 2010-10, Vol.115 (1), p.153-167

2010

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Loss of striatal 90-kDa ribosomal S6 kinase (Rsk) is a key factor for motor, synaptic and transcription dysfunction in Huntington's disease

Biochimica et biophysica acta, 2016-07, Vol.1862 (7), p.1255-1266

2016

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Delineating the Factors and Cellular Mechanisms Involved in the Survival of Cerebellar Granule Neurons

Cerebellum (London, England), 2015-06, Vol.14 (3), p.354-359

2015

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Ras Protein Activation Is a Key Event in Activity-dependent Survival of Cerebellar Granule Neurons

The Journal of biological chemistry, 2014-03, Vol.289 (12), p.8462-8472

2014

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Increased 90-kDa ribosomal S6 kinase (Rsk) activity is protective against mutant huntingtin toxicity

Molecular neurodegeneration, 2011-10, Vol.6 (1), p.74-74, Article 74

2011

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Striatal-enriched protein tyrosine phosphatase modulates nociception: evidence from genetic deletion and pharmacological inhibition

Pain (Amsterdam), 2016-02, Vol.157 (2), p.377-386

2016

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Activation of Elk-1 participates as a neuroprotective compensatory mechanism in models of Huntington's disease

Journal of neurochemistry, 2012-05, Vol.121 (4), p.639-648

2012

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Bone morphogenetic protein-6 promotes cerebellar granule neurons survival by activation of the MEK/ERK/CREB pathway

Molecular biology of the cell, 2009-12, Vol.20 (24), p.5051-5063

2009

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Mutant huntingtin impairs post-Golgi trafficking to lysosomes by delocalizing optineurin/Rab8 complex from the Golgi apparatus

Molecular biology of the cell, 2009-03, Vol.20 (5), p.1478-1492

2009

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Calcineurin is involved in the early activation of NMDA‐mediated cell death in mutant huntingtin knock‐in striatal cells

Journal of neurochemistry, 2008-06, Vol.105 (5), p.1596-1612

2008

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Contribution of caspase-mediated apoptosis to the cell death caused by oxygen–glucose deprivation in cortical cell cultures

Neurobiology of disease, 2005-10, Vol.20 (1), p.27-37

2005

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Diagnosis and pharmacological treatment of neuropathic pain in spinal cord injury subjects: from laboratory to clinical trial

Medicina clinica, 2013-02, Vol.140 (4), p.175

2013

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Estradiol facilitates neurite maintenance by a Src/Ras/ERK signalling pathway

Molecular and cellular neuroscience, 2008-10, Vol.39 (2), p.143-151

2008

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Corrigendum to “Brain-gut photobiomodulation restores cognitive alterations in chronically stressed mice through the regulation of Sirt1 and neuroinflammation” [J. Affect. Disord. 354 (2024) 574–588 10.1016/j.jad.2024.03.075 (Jun 1, Epub 2024 Mar 14, PMID: 38490587)]

Journal of affective disorders, 2024-09, Vol.361, p.799-799

2024

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BH3-only proteins Bid and Bim(EL) are differentially involved in neuronal dysfunction in mouse models of Huntington's disease

Journal of neuroscience research, 2007-09, Vol.85 (12), p.2756-2769

2007

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Protective Effects of Pituitary Adenylate Cyclase-Activating Polypeptide and Vasoactive Intestinal Peptide Against Cognitive Decline in Neurodegenerative Diseases

Novel epigallocatechin-3-gallate (EGCG) derivative as a new therapeutic strategy for reducing neuropathic pain after chronic constriction nerve injury in mice

Prostaglandin E2 EP2 activation reduces memory decline in R6/1 mouse model of Huntington's disease by the induction of BDNF-dependent synaptic plasticity

Increased PKA signaling disrupts recognition memory and spatial memory: role in Huntington's disease

Brain–gut photobiomodulation restores cognitive alterations in chronically stressed mice through the regulation of Sirt1 and neuroinflammation

Pituitary Adenylate Cyclase-Activating Polypeptide (PACAP) Protects Striatal Cells and Improves Motor Function in Huntington's Disease Models: Role of PAC1 Receptor

Decreased Myocyte Enhancer Factor 2 Levels in the Hippocampus of Huntington’s Disease Mice Are Related to Cognitive Dysfunction

Human alpha 1-antitrypsin protects neurons and glial cells against oxygen and glucose deprivation through inhibition of interleukins expression

Prostaglandin E2 EP1 Receptor Antagonist Improves Motor Deficits and Rescues Memory Decline in R6/1 Mouse Model of Huntington's Disease

Striatal-enriched protein tyrosine phosphatase expression and activity in Huntington's disease: a STEP in the resistance to excitotoxicity

Altered cholesterol homeostasis contributes to enhanced excitotoxicity in Huntington's disease

Loss of striatal 90-kDa ribosomal S6 kinase (Rsk) is a key factor for motor, synaptic and transcription dysfunction in Huntington's disease

Delineating the Factors and Cellular Mechanisms Involved in the Survival of Cerebellar Granule Neurons

Ras Protein Activation Is a Key Event in Activity-dependent Survival of Cerebellar Granule Neurons

Increased 90-kDa ribosomal S6 kinase (Rsk) activity is protective against mutant huntingtin toxicity

Striatal-enriched protein tyrosine phosphatase modulates nociception: evidence from genetic deletion and pharmacological inhibition

Activation of Elk-1 participates as a neuroprotective compensatory mechanism in models of Huntington's disease

Bone morphogenetic protein-6 promotes cerebellar granule neurons survival by activation of the MEK/ERK/CREB pathway

Mutant huntingtin impairs post-Golgi trafficking to lysosomes by delocalizing optineurin/Rab8 complex from the Golgi apparatus

Calcineurin is involved in the early activation of NMDA‐mediated cell death in mutant huntingtin knock‐in striatal cells

Contribution of caspase-mediated apoptosis to the cell death caused by oxygen–glucose deprivation in cortical cell cultures

Diagnosis and pharmacological treatment of neuropathic pain in spinal cord injury subjects: from laboratory to clinical trial

Estradiol facilitates neurite maintenance by a Src/Ras/ERK signalling pathway

Corrigendum to “Brain-gut photobiomodulation restores cognitive alterations in chronically stressed mice through the regulation of Sirt1 and neuroinflammation” [J. Affect. Disord. 354 (2024) 574–588 10.1016/j.jad.2024.03.075 (Jun 1, Epub 2024 Mar 14, PMID: 38490587)]

BH3-only proteins Bid and Bim(EL) are differentially involved in neuronal dysfunction in mouse models of Huntington's disease

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