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Neuropeptides Substance P and Calcitonin Gene Related Peptide Accelerate the Development and Fibrogenesis of Endometriosis

Scientific reports, 2019-02, Vol.9 (1), p.2698-2698, Article 2698

2019

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Corroborating evidence for platelet-induced epithelial-mesenchymal transition and fibroblast-to-myofibroblast transdifferentiation in the development of adenomyosis

Human reproduction (Oxford), 2016-04, Vol.31 (4), p.734-749

2016

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Histological and Immunohistochemical Characterization of the Similarity and Difference Between Ovarian Endometriomas and Deep Infiltrating Endometriosis

Reproductive sciences (Thousand Oaks, Calif.), 2018-03, Vol.25 (3), p.329-340

2018

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Platelets impair natural killer cell reactivity and function in endometriosis through multiple mechanisms

Human reproduction (Oxford), 2017-04, Vol.32 (4), p.794-810

2017

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Platelets and Regulatory T Cells May Induce a Type 2 Immunity That Is Conducive to the Progression and Fibrogenesis of Endometriosis

Frontiers in immunology, 2020-12, Vol.11, p.610963-610963

2020

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Platelets are an unindicted culprit in the development of endometriosis: clinical and experimental evidence

Human reproduction (Oxford), 2015-04, Vol.30 (4), p.812-832

2015

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Adenomyosis in mice resulting from mechanically or thermally induced endometrial–myometrial interface disruption and its possible prevention

Reproductive biomedicine online, 2020-11, Vol.41 (5), p.925-942

2020

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Reduced vagal tone in women with endometriosis and auricular vagus nerve stimulation as a potential therapeutic approach

Scientific reports, 2021-01, Vol.11 (1), p.1345-1345, Article 1345

2021

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The establishment of a mouse model of deep endometriosis

Human reproduction (Oxford), 2019-02, Vol.34 (2), p.235-247

2019

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Platelets induce increased estrogen production through NF-κB and TGF-β1 signaling pathways in endometriotic stromal cells

Scientific reports, 2020-01, Vol.10 (1), p.1281-1281, Article 1281

2020

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Higher fibrotic content of endometriotic lesions is associated with diminished prostaglandin E2 signaling

Reproductive medicine and biology, 2022-01, Vol.21 (1), p.e12423-n/a

2022

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Enhancer of Zeste homolog 2 (EZH2) induces epithelial-mesenchymal transition in endometriosis

Scientific reports, 2017-07, Vol.7 (1), p.6804-12, Article 6804

2017

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Evidence in Support for the Progressive Nature of Ovarian Endometriomas

The journal of clinical endocrinology and metabolism, 2020-07, Vol.105 (7), p.2189-2202

2020

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Surgery accelerates the development of endometriosis in mice

American journal of obstetrics and gynecology, 2016-09, Vol.215 (3), p.320.e1-320.e15

2016

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Dysmenorrhea and its severity are associated with increased uterine contractility and overexpression of oxytocin receptor (OTR) in women with symptomatic adenomyosis

Fertility and sterility, 2013, Vol.99 (1), p.231-240

2013

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Activation of α7 nicotinic acetylcholine receptor retards the development of endometriosis

Reproductive biology and endocrinology, 2022-06, Vol.20 (1), p.85-85, Article 85

2022

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Chronic stress accelerates the development of endometriosis in mouse through adrenergic receptor β2

Human reproduction (Oxford), 2016-11, Vol.31 (11), p.2506-2519

2016

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How does the extent of fibrosis in adenomyosis lesions contribute to heavy menstrual bleeding?

Reproductive medicine and biology, 2022-01, Vol.21 (1), p.e12442-n/a

2022

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Changing prostaglandin E2 (PGE2) signaling during lesional progression and exacerbation of endometriosis by inhibition of PGE2 receptor EP2 and EP4

Reproductive medicine and biology, 2022-01, Vol.21 (1), p.e12426-n/a

2022

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Possible involvement of neuropeptide and neurotransmitter receptors in Adenomyosis

Reproductive biology and endocrinology, 2021-02, Vol.19 (1), p.25-25, Article 25

2021

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Early maternal separation accelerates the progression of endometriosis in adult mice

Reproductive biology and endocrinology, 2020-06, Vol.18 (1), p.63-11, Article 63

2020

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Constitutive and tumor necrosis factor-α-induced activation of nuclear factor-κB in adenomyosis and its inhibition by andrographolide

Fertility and sterility, 2013-08, Vol.100 (2), p.568-577

2013

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Anti-platelet therapy holds promises in treating adenomyosis: experimental evidence

Reproductive biology and endocrinology, 2016-10, Vol.14 (1), p.66-66, Article 66

2016

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Anti-platelet therapy is efficacious in treating endometriosis induced in mouse

Reproductive biomedicine online, 2016-10, Vol.33 (4), p.484-499

2016

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P-selectin as a potential therapeutic target for endometriosis

Fertility and sterility, 2015-04, Vol.103 (4), p.990-1000.e8

2015

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Neuropeptides Substance P and Calcitonin Gene Related Peptide Accelerate the Development and Fibrogenesis of Endometriosis

Corroborating evidence for platelet-induced epithelial-mesenchymal transition and fibroblast-to-myofibroblast transdifferentiation in the development of adenomyosis

Histological and Immunohistochemical Characterization of the Similarity and Difference Between Ovarian Endometriomas and Deep Infiltrating Endometriosis

Platelets impair natural killer cell reactivity and function in endometriosis through multiple mechanisms

Platelets and Regulatory T Cells May Induce a Type 2 Immunity That Is Conducive to the Progression and Fibrogenesis of Endometriosis

Platelets are an unindicted culprit in the development of endometriosis: clinical and experimental evidence

Adenomyosis in mice resulting from mechanically or thermally induced endometrial–myometrial interface disruption and its possible prevention

Reduced vagal tone in women with endometriosis and auricular vagus nerve stimulation as a potential therapeutic approach

The establishment of a mouse model of deep endometriosis

Platelets induce increased estrogen production through NF-κB and TGF-β1 signaling pathways in endometriotic stromal cells

Higher fibrotic content of endometriotic lesions is associated with diminished prostaglandin E2 signaling

Enhancer of Zeste homolog 2 (EZH2) induces epithelial-mesenchymal transition in endometriosis

Evidence in Support for the Progressive Nature of Ovarian Endometriomas

Surgery accelerates the development of endometriosis in mice

Dysmenorrhea and its severity are associated with increased uterine contractility and overexpression of oxytocin receptor (OTR) in women with symptomatic adenomyosis

Activation of α7 nicotinic acetylcholine receptor retards the development of endometriosis

Chronic stress accelerates the development of endometriosis in mouse through adrenergic receptor β2

How does the extent of fibrosis in adenomyosis lesions contribute to heavy menstrual bleeding?

Changing prostaglandin E2 (PGE2) signaling during lesional progression and exacerbation of endometriosis by inhibition of PGE2 receptor EP2 and EP4

Possible involvement of neuropeptide and neurotransmitter receptors in Adenomyosis

Early maternal separation accelerates the progression of endometriosis in adult mice

Constitutive and tumor necrosis factor-α-induced activation of nuclear factor-κB in adenomyosis and its inhibition by andrographolide

Anti-platelet therapy holds promises in treating adenomyosis: experimental evidence

Anti-platelet therapy is efficacious in treating endometriosis induced in mouse

P-selectin as a potential therapeutic target for endometriosis

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