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Mutant huntingtin impairs post-Golgi trafficking to lysosomes by delocalizing optineurin/Rab8 complex from the Golgi apparatus

Molecular biology of the cell, 2009-03, Vol.20 (5), p.1478-1492

2009

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Protective Effects of Pituitary Adenylate Cyclase-Activating Polypeptide and Vasoactive Intestinal Peptide Against Cognitive Decline in Neurodegenerative Diseases

Frontiers in cellular neuroscience, 2020-07, Vol.14, p.221-221

2020

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Reduced calcineurin protein levels and activity in exon-1 mouse models of Huntington's disease: Role in excitotoxicity

Neurobiology of disease, 2009-12, Vol.36 (3), p.461-469

2009

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Increased PKA signaling disrupts recognition memory and spatial memory: role in Huntington's disease

Human molecular genetics, 2011-11, Vol.20 (21), p.4232-4247

2011

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Brain–gut photobiomodulation restores cognitive alterations in chronically stressed mice through the regulation of Sirt1 and neuroinflammation

Journal of affective disorders, 2024-06, Vol.354, p.574-588

2024

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Novel epigallocatechin-3-gallate (EGCG) derivative as a new therapeutic strategy for reducing neuropathic pain after chronic constriction nerve injury in mice

PloS one, 2015-04, Vol.10 (4), p.e0123122-e0123122

2015

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Decreased Myocyte Enhancer Factor 2 Levels in the Hippocampus of Huntington’s Disease Mice Are Related to Cognitive Dysfunction

Molecular neurobiology, 2020-11, Vol.57 (11), p.4549-4562

2020

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Human alpha 1-antitrypsin protects neurons and glial cells against oxygen and glucose deprivation through inhibition of interleukins expression

Biochimica et biophysica acta. General subjects, 2018-09, Vol.1862 (9), p.1852-1861

2018

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Pituitary Adenylate Cyclase-Activating Polypeptide (PACAP) Protects Striatal Cells and Improves Motor Function in Huntington's Disease Models: Role of PAC1 Receptor

Frontiers in pharmacology, 2022-01, Vol.12, p.797541-797541

2022

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Altered cholesterol homeostasis contributes to enhanced excitotoxicity in Huntington's disease

Journal of neurochemistry, 2010-10, Vol.115 (1), p.153-167

2010

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Loss of striatal 90-kDa ribosomal S6 kinase (Rsk) is a key factor for motor, synaptic and transcription dysfunction in Huntington's disease

Biochimica et biophysica acta, 2016-07, Vol.1862 (7), p.1255-1266

2016

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Prostaglandin E2 EP1 Receptor Antagonist Improves Motor Deficits and Rescues Memory Decline in R6/1 Mouse Model of Huntington's Disease

Molecular neurobiology, 2014-04, Vol.49 (2), p.784-795

2014

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Striatal-enriched protein tyrosine phosphatase expression and activity in Huntington's disease: a STEP in the resistance to excitotoxicity

The Journal of neuroscience, 2011-06, Vol.31 (22), p.8150-8162

2011

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Delineating the Factors and Cellular Mechanisms Involved in the Survival of Cerebellar Granule Neurons

Cerebellum (London, England), 2015-06, Vol.14 (3), p.354-359

2015

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Striatal-enriched protein tyrosine phosphatase modulates nociception: evidence from genetic deletion and pharmacological inhibition

Pain (Amsterdam), 2016-02, Vol.157 (2), p.377-386

2016

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Ras Protein Activation Is a Key Event in Activity-dependent Survival of Cerebellar Granule Neurons

The Journal of biological chemistry, 2014-03, Vol.289 (12), p.8462-8472

2014

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Activation of Elk-1 participates as a neuroprotective compensatory mechanism in models of Huntington's disease

Journal of neurochemistry, 2012-05, Vol.121 (4), p.639-648

2012

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Increased 90-kDa ribosomal S6 kinase (Rsk) activity is protective against mutant huntingtin toxicity

Molecular neurodegeneration, 2011-10, Vol.6 (1), p.74-74, Article 74

2011

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Bone morphogenetic protein-6 promotes cerebellar granule neurons survival by activation of the MEK/ERK/CREB pathway

Molecular biology of the cell, 2009-12, Vol.20 (24), p.5051-5063

2009

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Diagnosis and pharmacological treatment of neuropathic pain in spinal cord injury subjects: from laboratory to clinical trial

Medicina clinica, 2013-02, Vol.140 (4), p.175

2013

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Contribution of caspase-mediated apoptosis to the cell death caused by oxygen–glucose deprivation in cortical cell cultures

Neurobiology of disease, 2005-10, Vol.20 (1), p.27-37

2005

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Calcineurin is involved in the early activation of NMDA‐mediated cell death in mutant huntingtin knock‐in striatal cells

Journal of neurochemistry, 2008-06, Vol.105 (5), p.1596-1612

2008

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Estradiol facilitates neurite maintenance by a Src/Ras/ERK signalling pathway

Molecular and cellular neuroscience, 2008-10, Vol.39 (2), p.143-151

2008

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N-methyl-D-aspartate blocks activation of JNK and mitochondrial apoptotic pathway induced by potassium deprivation in cerebellar granule cells

The Journal of biological chemistry, 2006-03, Vol.281 (10), p.6801-6812

2006

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BH3-only proteins Bid and Bim(EL) are differentially involved in neuronal dysfunction in mouse models of Huntington's disease

Journal of neuroscience research, 2007-09, Vol.85 (12), p.2756-2769

2007

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Mutant huntingtin impairs post-Golgi trafficking to lysosomes by delocalizing optineurin/Rab8 complex from the Golgi apparatus

Protective Effects of Pituitary Adenylate Cyclase-Activating Polypeptide and Vasoactive Intestinal Peptide Against Cognitive Decline in Neurodegenerative Diseases

Reduced calcineurin protein levels and activity in exon-1 mouse models of Huntington's disease: Role in excitotoxicity

Increased PKA signaling disrupts recognition memory and spatial memory: role in Huntington's disease

Brain–gut photobiomodulation restores cognitive alterations in chronically stressed mice through the regulation of Sirt1 and neuroinflammation

Novel epigallocatechin-3-gallate (EGCG) derivative as a new therapeutic strategy for reducing neuropathic pain after chronic constriction nerve injury in mice

Decreased Myocyte Enhancer Factor 2 Levels in the Hippocampus of Huntington’s Disease Mice Are Related to Cognitive Dysfunction

Human alpha 1-antitrypsin protects neurons and glial cells against oxygen and glucose deprivation through inhibition of interleukins expression

Pituitary Adenylate Cyclase-Activating Polypeptide (PACAP) Protects Striatal Cells and Improves Motor Function in Huntington's Disease Models: Role of PAC1 Receptor

Altered cholesterol homeostasis contributes to enhanced excitotoxicity in Huntington's disease

Loss of striatal 90-kDa ribosomal S6 kinase (Rsk) is a key factor for motor, synaptic and transcription dysfunction in Huntington's disease

Prostaglandin E2 EP1 Receptor Antagonist Improves Motor Deficits and Rescues Memory Decline in R6/1 Mouse Model of Huntington's Disease

Striatal-enriched protein tyrosine phosphatase expression and activity in Huntington's disease: a STEP in the resistance to excitotoxicity

Delineating the Factors and Cellular Mechanisms Involved in the Survival of Cerebellar Granule Neurons

Striatal-enriched protein tyrosine phosphatase modulates nociception: evidence from genetic deletion and pharmacological inhibition

Ras Protein Activation Is a Key Event in Activity-dependent Survival of Cerebellar Granule Neurons

Activation of Elk-1 participates as a neuroprotective compensatory mechanism in models of Huntington's disease

Increased 90-kDa ribosomal S6 kinase (Rsk) activity is protective against mutant huntingtin toxicity

Bone morphogenetic protein-6 promotes cerebellar granule neurons survival by activation of the MEK/ERK/CREB pathway

Diagnosis and pharmacological treatment of neuropathic pain in spinal cord injury subjects: from laboratory to clinical trial

Contribution of caspase-mediated apoptosis to the cell death caused by oxygen–glucose deprivation in cortical cell cultures

Calcineurin is involved in the early activation of NMDA‐mediated cell death in mutant huntingtin knock‐in striatal cells

Estradiol facilitates neurite maintenance by a Src/Ras/ERK signalling pathway

N-methyl-D-aspartate blocks activation of JNK and mitochondrial apoptotic pathway induced by potassium deprivation in cerebellar granule cells

BH3-only proteins Bid and Bim(EL) are differentially involved in neuronal dysfunction in mouse models of Huntington's disease

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