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Etiology of inflammatory bowel disease：A unified hypothesis

World journal of gastroenterology : WJG, 2012-04, Vol.18 (15), p.1708-1722

Qin, Xiaofa

2012

Volltextzugriff (PDF)

Letter: Non-Nutritive Sweeteners May Have a Bell-Shaped Dose Response for Dysbiosis-Related Adverse Effects That Contributed to Discrepancies between Prospective Cohort and Randomized Controlled Studies

Journal of obesity & metabolic syndrome, 2020-09, Vol.29 (3), p.237-239

Qin, Xiaofa

2020

Volltextzugriff (PDF)

May bacterial or pancreatic proteases play a critical role in inflammatory bowel disease?

World journal of gastroenterology : WJG, 2014-09, Vol.20 (35), p.12709-12710

Qin, Xiaofa

2014

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Food Additives: Possible Cause for Recent Remarkable Increase of Inflammatory Bowel Disease in Children

Journal of pediatric gastroenterology and nutrition, 2012-04, Vol.54 (4), p.564-564

Qin, Xiaofa

2012

Volltextzugriff (PDF)

How to explain recent multiple reports on the decline of inflammatory bowel disease in Canada

Canadian journal of gastroenterology & hepatology, 2014-12, Vol.28 (11), p.620-620

Qin, Xiaofa

2014

Volltextzugriff (PDF)

Antibacterial Agents May Have Shifted Impacts on Inflammatory Bowel Diseases Along with Decrease in Gut Bacteria

Inflammatory bowel diseases, 2024-07, Vol.30 (7), p.1228-1231

Qin, Xiaofa

2024

Volltextzugriff (PDF)

Impaired inactivation of digestive proteases: The possible key factor for the high susceptibility of germ-free and antibiotic-treated animals to gut epithelial injury

World journal of gastrointestinal pathophysiology, 2017-02, Vol.8 (1), p.1-2

Qin, Xiaofa

2017

Volltextzugriff (PDF)

Sucralose Promotes Colitis-Associated Colorectal Cancer Risk in a Murine Model Along With Changes in Microbiota

Frontiers in oncology, 2020-06, Vol.10, p.710-710

2020

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High Fecal Proteolytic Activity That Precedes Ulcerative Colitis Likely Results From Impaired Inactivation of Pancreatic Proteases Rather Than Bacteria

Inflammatory bowel diseases, 2021-09, Vol.27 (9), p.e104-e104

Qin, Xiaofa

2021

Volltextzugriff (PDF)

More Epidemiological Studies Warranted to Determine the Cause of Inflammatory Bowel Disease

Inflammatory bowel diseases, 2021-04, Vol.27 (4), p.e43-e44

Qin, Xiaofa

2021

Volltextzugriff (PDF)

Inhibition of Experimental Colitis by Saccharin in Animals: Should We Dismiss or Raise Concerns Regarding Possible Adverse Effects of Saccharin on Human Gut Microbiota and Health?

Inflammatory bowel diseases, 2020-12, Vol.26 (12), p.e159-e160

Qin, Xiaofa

2020

Volltextzugriff (PDF)

Impaired Inactivation of Digestive Proteases in Lower Gut Due to Inhibition of Gut Bacteria by Food Additives Such as Saccharin and Sucralose as Main Cause of Inflammatory Bowel Disease: A Two-Decades-Long Hypothesis Warrants Testing

Inflammatory bowel diseases, 2019-10, Vol.25 (11), p.e141-e141

Qin, Xiaofa

2019

Volltextzugriff (PDF)

May artificial sweeteners not sugar be the culprit of dramatic increase of inflammatory bowel disease in China

Chinese medical journal, 2014, Vol.127 (17), p.3196-3197

Qin, Xiaofa

2014

Volltextzugriff (PDF)

The Functional Role of Fecal Microbiota Transplantation on Dextran Sulfate Sodium-Induced Colitis in Mice

Frontiers in cellular and infection microbiology, 2019-11, Vol.9, p.393

2019

Volltextzugriff (PDF)

May Dysbiosis Caused by Dietary Chemicals Such as Sucralose and Saccharin Be More Detrimental to the Gut and Health Than Antibiotics? How?

Inflammatory bowel diseases, 2019-03, Vol.25 (3), p.e20-e20

Qin, Xiaofa

2019

Volltextzugriff (PDF)

The Effect of Splenda on Gut Microbiota of Humans Could be Much More Detrimental Than in Animals and Deserves More Extensive Research

Inflammatory bowel diseases, 2019-01, Vol.25 (2), p.e7-e7

Qin, Xiaofa

2019

Volltextzugriff (PDF)

Damage of the Mucus Layer: The Possible Shared Critical Common Cause for Both Inflammatory Bowel Disease (IBD) and Irritable Bowel Syndrome (IBS)

Inflammatory bowel diseases, 2017-02, Vol.23 (2), p.E11-E12

Qin, Xiaofa

2017

Volltextzugriff (PDF)

Ligands can differentially and temporally modulate GPCR interaction with 14-3-3 isoforms

Current research in pharmacology and drug discovery, 2022-01, Vol.3, p.100123-100123, Article 100123

2022

Volltextzugriff (PDF)

Inactivation of digestive proteases: another aspect of gut bacteria that should be taken into more consideration

World journal of gastroenterology : WJG, 2007-04, Vol.13 (16), p.2390

Qin, Xiaofa

2007

Volltextzugriff (PDF)

What caused the increase of autoimmune and allergic diseases: a decreased or an increased exposure to luminal microbial components?

World journal of gastroenterology : WJG, 2007-02, Vol.13 (8), p.1306

Qin, Xiaofa

2007

Volltextzugriff (PDF)

The Possible Link Between Artificial Sweeteners Such as Saccharin and Sucralose and Inflammatory Bowel Disease Deserves Further Study

Inflammatory bowel diseases, 2016-06, Vol.22 (6), p.E17-E17

Qin, Xiaofa

2016

Volltextzugriff (PDF)

Inactivation of Digestive Proteases and Degradation of Mucus: The Possible Key Factors That Determined the Different Effects on the Gut by Antibiotics

Inflammatory bowel diseases, 2017-02, Vol.23 (2), p.E7-E7

Qin, Xiaofa

2017

Volltextzugriff (PDF)

Effect of bile pigments on the compromised gut barrier function in a rat model of bile duct ligation

PloS one, 2014-06, Vol.9 (6), p.e98905-e98905

2014

Volltextzugriff (PDF)

Increased Milk Consumption but Decreased Risk of Crohn's Disease (CD): Critical Evidence Negated Causative Role of Mycobacterium avium Subspecies paratuberculosis (MAP) in CD

Inflammatory bowel diseases, 2016-09, Vol.22 (9), p.E37-E38

Qin, Xiaofa

2016

Volltextzugriff (PDF)

Perinuclear Antineutrophil Cytoplasmic Antibody with a Large Portion Being Anti-β-Glucuronidase, May Have Played a Causative Role in the Pathogenesis of Inflammatory Bowel Disease

Inflammatory bowel diseases, 2016-11, Vol.22 (11), p.E41-E42

Qin, Xiaofa

2016

Volltextzugriff (PDF)

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Etiology of inflammatory bowel disease：A unified hypothesis

Letter: Non-Nutritive Sweeteners May Have a Bell-Shaped Dose Response for Dysbiosis-Related Adverse Effects That Contributed to Discrepancies between Prospective Cohort and Randomized Controlled Studies

May bacterial or pancreatic proteases play a critical role in inflammatory bowel disease?

Food Additives: Possible Cause for Recent Remarkable Increase of Inflammatory Bowel Disease in Children

How to explain recent multiple reports on the decline of inflammatory bowel disease in Canada

Antibacterial Agents May Have Shifted Impacts on Inflammatory Bowel Diseases Along with Decrease in Gut Bacteria

Impaired inactivation of digestive proteases: The possible key factor for the high susceptibility of germ-free and antibiotic-treated animals to gut epithelial injury

Sucralose Promotes Colitis-Associated Colorectal Cancer Risk in a Murine Model Along With Changes in Microbiota

High Fecal Proteolytic Activity That Precedes Ulcerative Colitis Likely Results From Impaired Inactivation of Pancreatic Proteases Rather Than Bacteria

More Epidemiological Studies Warranted to Determine the Cause of Inflammatory Bowel Disease

Inhibition of Experimental Colitis by Saccharin in Animals: Should We Dismiss or Raise Concerns Regarding Possible Adverse Effects of Saccharin on Human Gut Microbiota and Health?

Impaired Inactivation of Digestive Proteases in Lower Gut Due to Inhibition of Gut Bacteria by Food Additives Such as Saccharin and Sucralose as Main Cause of Inflammatory Bowel Disease: A Two-Decades-Long Hypothesis Warrants Testing

May artificial sweeteners not sugar be the culprit of dramatic increase of inflammatory bowel disease in China

The Functional Role of Fecal Microbiota Transplantation on Dextran Sulfate Sodium-Induced Colitis in Mice

May Dysbiosis Caused by Dietary Chemicals Such as Sucralose and Saccharin Be More Detrimental to the Gut and Health Than Antibiotics? How?

The Effect of Splenda on Gut Microbiota of Humans Could be Much More Detrimental Than in Animals and Deserves More Extensive Research

Damage of the Mucus Layer: The Possible Shared Critical Common Cause for Both Inflammatory Bowel Disease (IBD) and Irritable Bowel Syndrome (IBS)

Ligands can differentially and temporally modulate GPCR interaction with 14-3-3 isoforms

Inactivation of digestive proteases: another aspect of gut bacteria that should be taken into more consideration

What caused the increase of autoimmune and allergic diseases: a decreased or an increased exposure to luminal microbial components?

The Possible Link Between Artificial Sweeteners Such as Saccharin and Sucralose and Inflammatory Bowel Disease Deserves Further Study

Inactivation of Digestive Proteases and Degradation of Mucus: The Possible Key Factors That Determined the Different Effects on the Gut by Antibiotics

Effect of bile pigments on the compromised gut barrier function in a rat model of bile duct ligation

Increased Milk Consumption but Decreased Risk of Crohn's Disease (CD): Critical Evidence Negated Causative Role of Mycobacterium avium Subspecies paratuberculosis (MAP) in CD

Perinuclear Antineutrophil Cytoplasmic Antibody with a Large Portion Being Anti-β-Glucuronidase, May Have Played a Causative Role in the Pathogenesis of Inflammatory Bowel Disease

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