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A peptide encoded by a transcript annotated as long noncoding RNA enhances SERCA activity in muscle

Science (American Association for the Advancement of Science), 2016-01, Vol.351 (6270), p.271-275

2016

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Rapid and Inexpensive Method of Loading Fluorescent Dye into Pollen Tubes and Root Hairs

PloS one, 2016-04, Vol.11 (4), p.e0152320-e0152320

2016

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Stac3 enhances expression of human CaV1.1 in Xenopus oocytes and reveals gating pore currents in HypoPP mutant channels

The Journal of general physiology, 2018-03, Vol.150 (3), p.475-489

2018

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Skeletal muscle-specific T-tubule protein STAC3 mediates voltage-induced Ca²⁺ release and contractility

Proceedings of the National Academy of Sciences - PNAS, 2013-07, Vol.110 (29), p.11881-11886

2013

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Gating Pore Currents in DIII Hypopp Mutations of CaV1.1

Biophysical journal, 2018-02, Vol.114 (3), p.639a-639a

2018

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Bumetanide prevents transient decreases in muscle force in murine hypokalemic periodic paralysis

Neurology, 2013-03, Vol.80 (12), p.1110

2013

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Beneficial effects of bumetanide in a CaV1.1-R528H mouse model of hypokalaemic periodic paralysis

Brain (London, England : 1878), 2013-12, Vol.136 (Pt 12), p.3766-3774

2013

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A sodium channel knockin mutant (NaV1.4-R669H) mouse model of hypokalemic periodic paralysis

The Journal of clinical investigation, 2011-10, Vol.121 (10), p.4082-4094

2011

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Gating pore currents occur in CaV1.1 domain III mutants associated with HypoPP

The Journal of general physiology, 2021-11, Vol.153 (11), p.1

2021

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CaV1.1 defects in hypokalemic periodic paralysis and harnessing multiple approaches for therapeutic intervention

The Journal of general physiology, 2022-09, Vol.154 (9)

2022

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CaV1.1 defects in hypokalemic periodic paralysis and harnessing multiple approaches for therapeutic intervention: Calcium Signaling and Excitation–Contraction in Cardiac, Skeletal and Smooth Muscle

The Journal of general physiology, 2022-09, Vol.154 (9), p.1

2022

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Stac3 Facilitated Expression of CaV1.1 in Xenopus Oocytes to Assess Functional Consequences of Hypopp Mutant CaV1.1-R528H

Biophysical journal, 2017-02, Vol.112 (3), p.245a-245a

2017

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Stac3 Facilitated Expression of Ca V 1.1 in Xenopus Oocytes to Assess Functional Consequences of Hypopp Mutant Ca V 1.1-R528H

Biophysical journal, 2017-02, Vol.112 (3), p.245

2017

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Contrast Media Volume to Creatinine Clearance Ratio in Predicting Nephropathy in Patients Undergoing Percutaneous Coronary Intervention: A Systematic Review and Meta-Analysis

Angiology, 2023-07, Vol.74 (6), p.545-552

2023

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Retigabine suppresses loss of force in mouse models of hypokalaemic periodic paralysis

Brain (London, England : 1878), 2023-04, Vol.146 (4), p.1554-1560

2023

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Proposed Mechanism for Post‐Exercise Weakness in Hypokalemic Periodic Paralysis

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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The distinct role of the four voltage sensors of the skeletal CaV1.1 channel in voltage-dependent activation

The Journal of general physiology, 2021-11, Vol.153 (11), p.1

2021

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The role of Ca2+ and Ca2+ channels in the gametophytic self-incompatibility of Pyrus pyrifolia

Cell calcium (Edinburgh), 2016-11, Vol.60 (5), p.299-308

2016

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The application of whole-course nursing in patients undergoing emergency PCI and its impact on cardiac function

American journal of translational research, 2021-01, Vol.13 (7), p.8323-8329

2021

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Stac3 enhances expression of human Ca^sub V^1.1 in Xenopus oocytes and reveals gating pore currents in HypoPP mutant channels

The Journal of general physiology, 2018-03, Vol.150 (3), p.475

2018

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Recovery from acidosis is a robust trigger for loss of force in murine hypokalemic periodic paralysis

The Journal of general physiology, 2019-04, Vol.151 (4), p.555-566

2019

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A Mutation Linked to Malignant Hyperthermia in the Skeletal CaV1.1 Channel Stabilizes the Resting State of Voltage Sensor I and Impairs Channel Activation

Biophysical journal, 2019-02, Vol.116 (3), p.111a-111a

2019

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Recovery from Intracellular Acidosis Triggers Loss of Force in Hypokalemic Periodic Paralysis

Biophysical journal, 2019-02, Vol.116 (3), p.32

2019

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The Contribution of the Individual Voltage Sensors to the Activation of Skeletal CaV1.1 Channels

Biophysical journal, 2020-02, Vol.118 (3), p.105a-105a

2020

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Mice with an NaV1.4 sodium channel null allele have latent myasthenia, without susceptibility to periodic paralysis

Brain (London, England : 1878), 2016-06, Vol.139 (Pt 6), p.1688-1699

2016

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A peptide encoded by a transcript annotated as long noncoding RNA enhances SERCA activity in muscle

Rapid and Inexpensive Method of Loading Fluorescent Dye into Pollen Tubes and Root Hairs

Stac3 enhances expression of human CaV1.1 in Xenopus oocytes and reveals gating pore currents in HypoPP mutant channels

Skeletal muscle-specific T-tubule protein STAC3 mediates voltage-induced Ca²⁺ release and contractility

Gating Pore Currents in DIII Hypopp Mutations of CaV1.1

Bumetanide prevents transient decreases in muscle force in murine hypokalemic periodic paralysis

Beneficial effects of bumetanide in a CaV1.1-R528H mouse model of hypokalaemic periodic paralysis

A sodium channel knockin mutant (NaV1.4-R669H) mouse model of hypokalemic periodic paralysis

Gating pore currents occur in CaV1.1 domain III mutants associated with HypoPP

CaV1.1 defects in hypokalemic periodic paralysis and harnessing multiple approaches for therapeutic intervention

CaV1.1 defects in hypokalemic periodic paralysis and harnessing multiple approaches for therapeutic intervention: Calcium Signaling and Excitation–Contraction in Cardiac, Skeletal and Smooth Muscle

Stac3 Facilitated Expression of CaV1.1 in Xenopus Oocytes to Assess Functional Consequences of Hypopp Mutant CaV1.1-R528H

Stac3 Facilitated Expression of Ca V 1.1 in Xenopus Oocytes to Assess Functional Consequences of Hypopp Mutant Ca V 1.1-R528H

Contrast Media Volume to Creatinine Clearance Ratio in Predicting Nephropathy in Patients Undergoing Percutaneous Coronary Intervention: A Systematic Review and Meta-Analysis

Retigabine suppresses loss of force in mouse models of hypokalaemic periodic paralysis

Proposed Mechanism for Post‐Exercise Weakness in Hypokalemic Periodic Paralysis

The distinct role of the four voltage sensors of the skeletal CaV1.1 channel in voltage-dependent activation

The role of Ca2+ and Ca2+ channels in the gametophytic self-incompatibility of Pyrus pyrifolia

The application of whole-course nursing in patients undergoing emergency PCI and its impact on cardiac function

Stac3 enhances expression of human Ca^sub V^1.1 in Xenopus oocytes and reveals gating pore currents in HypoPP mutant channels

Recovery from acidosis is a robust trigger for loss of force in murine hypokalemic periodic paralysis

A Mutation Linked to Malignant Hyperthermia in the Skeletal CaV1.1 Channel Stabilizes the Resting State of Voltage Sensor I and Impairs Channel Activation

Recovery from Intracellular Acidosis Triggers Loss of Force in Hypokalemic Periodic Paralysis

The Contribution of the Individual Voltage Sensors to the Activation of Skeletal CaV1.1 Channels

Mice with an NaV1.4 sodium channel null allele have latent myasthenia, without susceptibility to periodic paralysis

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