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The small-molecule BGP-15 protects against heart failure and atrial fibrillation in mice

Nature communications, 2014-12, Vol.5 (1), p.5705-5705, Article 5705

2014

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Protective effects of exercise and phosphoinositide 3-kinase(p110α) signaling in dilated and hypertrophic cardiomyopathy

Proceedings of the National Academy of Sciences - PNAS, 2007-01, Vol.104 (2), p.612-617

2007

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Chronic Contractile Dysfunction without Hypertrophy Does Not Provoke a Compensatory Transcriptional Response in Mouse Hearts

PloS one, 2016-06, Vol.11 (6), p.e0158317-e0158317

2016

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Distribution and Functional Role of Inositol 1,4,5-trisphosphate Receptors in Mouse Sinoatrial Node

Circulation research, 2011-09, Vol.109 (8), p.848-857

2011

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Inositol 1,4,5-trisphosphate receptors and pacemaker rhythms

Journal of molecular and cellular cardiology, 2012-09, Vol.53 (3), p.375-381

2012

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Relaxin Antagonizes Hypertrophy and Apoptosis in Neonatal Rat Cardiomyocytes

Endocrinology (Philadelphia), 2007-04, Vol.148 (4), p.1582-1589

2007

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Novel Therapeutic Targets in Heart Failure: The Phospholipase Cβ1b-Shank3 Interface

Clinical Medicine Insights: Therapeutics, 2015-05, Vol.2015 (7), p.11

2015

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Regulation of the Proapoptotic Factor FOXO1 (FKHR) in Cardiomyocytes by Growth Factors and α1-Adrenergic Agonists

Endocrinology (Philadelphia), 2005-10, Vol.146 (10), p.4370-4376

2005

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FoxO1 is required for physiological cardiac hypertrophy induced by exercise but not by constitutively active PI3K

American journal of physiology. Heart and circulatory physiology, 2021-04, Vol.320 (4), p.H1470-H1485

2021

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Phospholipase Cβ1b directly binds the SH3 domain of Shank3 for targeting and activation in cardiomyocytes

Biochemical and biophysical research communications, 2015-06, Vol.461 (3), p.519-524

2015

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The phosphatidylinositol(4,5)bisphosphate-binding sequence of transient receptor potential channel canonical 4α is critical for its contribution to cardiomyocyte hypertrophy

Molecular pharmacology, 2014-10, Vol.86 (4), p.399-405

2014

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Cardiomyocytes structure, function and associated pathologies

The international journal of biochemistry & cell biology, 2005-09, Vol.37 (9), p.1746-1751

2005

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Phosphoinositide 3-kinase p110α is a master regulator of exercise-induced cardioprotection and PI3K gene therapy rescues cardiac dysfunction

Circulation. Heart failure, 2012-07, Vol.5 (4), p.523-534

2012

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Gq-initiated cardiomyocyte hypertrophy is mediated by phospholipase Cβ1b

The FASEB journal, 2009-10, Vol.23 (10), p.3564-3570

2009

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Reduced Phosphoinositide 3-Kinase (p110α) Activation Increases the Susceptibility to Atrial Fibrillation

The American journal of pathology, 2009-09, Vol.175 (3), p.998-1009

2009

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PI3K(p110 alpha) protects against myocardial infarction-induced heart failure: identification of PI3K-regulated miRNA and mRNA

Arteriosclerosis, thrombosis, and vascular biology, 2010-04, Vol.30 (4), p.724-732

2010

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ROLES OF α1A- AND α1B-ADRENOCEPTORS IN HEART: INSIGHTS FROM STUDIES OF GENETICALLY MODIFIED MICE

Clinical and experimental pharmacology & physiology, 2007-09, Vol.34 (9), p.884-888

Woodcock, Elizabeth A

2007

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Regulation of autophagy in cardiomyocytes by Ins(1,4,5)P3 and IP3 -receptors

Journal of molecular and cellular cardiology, 2013-01, Vol.54, p.19-24

2013

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Influence of atrial fibrillation on microRNA expression profiles in left and right atria from patients with valvular heart disease

Physiological genomics, 2012-02, Vol.44 (3), p.211-219

2012

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Scaffolding protein Homer 1c mediates hypertrophic responses downstream of Gq in cardiomyocytes

The FASEB journal, 2012-02, Vol.26 (2), p.596-603

2012

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UTP Transactivates Epidermal Growth Factor Receptors and Promotes Cardiomyocyte Hypertrophy Despite Inhibiting Transcription of the Hypertrophic Marker Gene, Atrial Natriuretic Peptide

The Journal of biological chemistry, 2004-03, Vol.279 (10), p.8740-8746

2004

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The atypical ‘b’ splice variant of phospholipase Cβ1 promotes cardiac contractile dysfunction

Journal of molecular and cellular cardiology, 2015-07, Vol.84, p.95-103

2015

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Cardiac α1-adrenergic drive in pathological remodelling

Cardiovascular research, 2008-02, Vol.77 (3), p.452-462

2008

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Expressing an inhibitor of PLCβ1b sustains contractile function following pressure overload

Journal of molecular and cellular cardiology, 2016-04, Vol.93, p.12-17

2016

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The extreme C‐terminal region of phospholipase Cβ1 determines subcellular localization and function; the “b” splice variant mediates α1‐adrenergic receptor responses in cardiomyocytes

The FASEB journal, 2008-08, Vol.22 (8), p.2768-2774

2008

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The small-molecule BGP-15 protects against heart failure and atrial fibrillation in mice

Protective effects of exercise and phosphoinositide 3-kinase(p110α) signaling in dilated and hypertrophic cardiomyopathy

Chronic Contractile Dysfunction without Hypertrophy Does Not Provoke a Compensatory Transcriptional Response in Mouse Hearts

Distribution and Functional Role of Inositol 1,4,5-trisphosphate Receptors in Mouse Sinoatrial Node

Inositol 1,4,5-trisphosphate receptors and pacemaker rhythms

Relaxin Antagonizes Hypertrophy and Apoptosis in Neonatal Rat Cardiomyocytes

Novel Therapeutic Targets in Heart Failure: The Phospholipase Cβ1b-Shank3 Interface

Regulation of the Proapoptotic Factor FOXO1 (FKHR) in Cardiomyocytes by Growth Factors and α1-Adrenergic Agonists

FoxO1 is required for physiological cardiac hypertrophy induced by exercise but not by constitutively active PI3K

Phospholipase Cβ1b directly binds the SH3 domain of Shank3 for targeting and activation in cardiomyocytes

The phosphatidylinositol(4,5)bisphosphate-binding sequence of transient receptor potential channel canonical 4α is critical for its contribution to cardiomyocyte hypertrophy

Cardiomyocytes structure, function and associated pathologies

Phosphoinositide 3-kinase p110α is a master regulator of exercise-induced cardioprotection and PI3K gene therapy rescues cardiac dysfunction

Gq-initiated cardiomyocyte hypertrophy is mediated by phospholipase Cβ1b

Reduced Phosphoinositide 3-Kinase (p110α) Activation Increases the Susceptibility to Atrial Fibrillation

PI3K(p110 alpha) protects against myocardial infarction-induced heart failure: identification of PI3K-regulated miRNA and mRNA

ROLES OF α1A- AND α1B-ADRENOCEPTORS IN HEART: INSIGHTS FROM STUDIES OF GENETICALLY MODIFIED MICE

Regulation of autophagy in cardiomyocytes by Ins(1,4,5)P3 and IP3 -receptors

Influence of atrial fibrillation on microRNA expression profiles in left and right atria from patients with valvular heart disease

Scaffolding protein Homer 1c mediates hypertrophic responses downstream of Gq in cardiomyocytes

UTP Transactivates Epidermal Growth Factor Receptors and Promotes Cardiomyocyte Hypertrophy Despite Inhibiting Transcription of the Hypertrophic Marker Gene, Atrial Natriuretic Peptide

The atypical ‘b’ splice variant of phospholipase Cβ1 promotes cardiac contractile dysfunction

Cardiac α1-adrenergic drive in pathological remodelling

Expressing an inhibitor of PLCβ1b sustains contractile function following pressure overload

The extreme C‐terminal region of phospholipase Cβ1 determines subcellular localization and function; the “b” splice variant mediates α1‐adrenergic receptor responses in cardiomyocytes

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