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Targeting toll-like receptor-4 (TLR4)-an emerging therapeutic target for persistent pain states

Pain (Amsterdam), 2018-10, Vol.159 (10), p.1908-1915

2018

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Rapid continuous 3D printing of customizable peripheral nerve guidance conduits

Materials today (Kidlington, England), 2018-11, Vol.21 (9), p.951-959

2018

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Opioid administration following spinal cord injury: Implications for pain and locomotor recovery

Experimental neurology, 2013-09, Vol.247, p.328-341

2013

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Inflammation is increased with anxiety- and depression-like signs in a rat model of spinal cord injury

Brain, behavior, and immunity, 2016-01, Vol.51, p.176-195

2016

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Origins of antidromic activity in sensory afferent fibers and neurogenic inflammation

Seminars in immunopathology, 2018-05, Vol.40 (3), p.237-247

2018

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Inhibition of Neuroinflammation by AIBP: Spinal Effects upon Facilitated Pain States

Cell reports (Cambridge), 2018-05, Vol.23 (9), p.2667-2677

2018

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The novel ROCK2 selective inhibitor NRL-1049 preserves the blood-brain barrier after acute injury

Journal of cerebral blood flow and metabolism, 2024-11, Vol.44 (11), p.1238-1252

2024

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An overview of pathways encoding nociception

Clinical and experimental rheumatology, 2017-01, Vol.35 Suppl 107 (5), p.40

2017

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Neurobiological Effects of Morphine after Spinal Cord Injury

Journal of neurotrauma, 2017-02, Vol.34 (3), p.632-644

2017

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Assessment of depression in a rodent model of spinal cord injury

Journal of neurotrauma, 2014-06, Vol.31 (12), p.1107-1121

2014

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Systemic TAK-242 prevents intrathecal LPS evoked hyperalgesia in male, but not female mice and prevents delayed allodynia following intraplantar formalin in both male and female mice: The role of TLR4 in the evolution of a persistent pain state

Brain, behavior, and immunity, 2016-08, Vol.56, p.271-280

2016

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Neuraxial TNF and IFN-beta co-modulate persistent allodynia in arthritic mice

Brain, behavior, and immunity, 2019-02, Vol.76, p.151-158

2019

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Peripheral noxious stimulation reduces withdrawal threshold to mechanical stimuli after spinal cord injury: Role of tumor necrosis factor alpha and apoptosis

Pain (Amsterdam), 2014-11, Vol.155 (11), p.2344-2359

2014

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The neuropathic phenotype of the K/BxN transgenic mouse with spontaneous arthritis: pain, nerve sprouting and joint remodeling

Scientific reports, 2020-09, Vol.10 (1), p.15596-15596, Article 15596

2020

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INTEGRATE-Pain: a transatlantic consortium to advance development of effective pain management

Pain medicine (Malden, Mass.), 2023-06, Vol.24 (6), p.730-733

2023

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Critical NIH Resources to Advance Therapies for Pain: Preclinical Screening Program and Phase II Human Clinical Trial Network

Neurotherapeutics, 2020-07, Vol.17 (3), p.932-934

2020

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Morphine self-administration following spinal cord injury

Journal of neurotrauma, 2014-09, Vol.31 (18), p.1570-1583

2014

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Long-lasting analgesia via targeted in situ repression of Na V 1.7 in mice

Science translational medicine, 2021-03, Vol.13 (584)

2021

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Preclinical target validation for non-addictive therapeutics development for pain

Expert opinion on therapeutic targets, 2022-09, Vol.26 (9), p.811-822

2022

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NIH HEAL Initiative: National Institute of Neurological Disorders and Stroke Preclinical Program for Non-Addictive Pain Therapeutic Development

The journal of pain, 2022-05, Vol.23 (5), p.28-28

2022

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Analgesia or addiction?: implications for morphine use after spinal cord injury

Journal of neurotrauma, 2012-05, Vol.29 (8), p.1650-1662

2012

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An IL-1 receptor antagonist blocks a morphine-induced attenuation of locomotor recovery after spinal cord injury

Brain, behavior, and immunity, 2011-02, Vol.25 (2), p.349-359

2011

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Self-delivering RNAi compounds as therapeutic agents in the central nervous system to enhance axonal regeneration after injury

iScience, 2022-06, Vol.25 (6), p.104379-104379, Article 104379

2022

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The association between spinal cord trauma-sensitive miRNAs and pain sensitivity, and their regulation by morphine

Neurochemistry international, 2014-11, Vol.77, p.40-49

2014

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ID: 208858 The NIH HEAL Initiative Preclinical Screening Platform for Pain

Neuromodulation (Malden, Mass.), 2023-06, Vol.26 (4), p.S147-S147

2023

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Targeting toll-like receptor-4 (TLR4)-an emerging therapeutic target for persistent pain states

Rapid continuous 3D printing of customizable peripheral nerve guidance conduits

Opioid administration following spinal cord injury: Implications for pain and locomotor recovery

Inflammation is increased with anxiety- and depression-like signs in a rat model of spinal cord injury

Origins of antidromic activity in sensory afferent fibers and neurogenic inflammation

Inhibition of Neuroinflammation by AIBP: Spinal Effects upon Facilitated Pain States

The novel ROCK2 selective inhibitor NRL-1049 preserves the blood-brain barrier after acute injury

An overview of pathways encoding nociception

Neurobiological Effects of Morphine after Spinal Cord Injury

Assessment of depression in a rodent model of spinal cord injury

Systemic TAK-242 prevents intrathecal LPS evoked hyperalgesia in male, but not female mice and prevents delayed allodynia following intraplantar formalin in both male and female mice: The role of TLR4 in the evolution of a persistent pain state

Neuraxial TNF and IFN-beta co-modulate persistent allodynia in arthritic mice

Peripheral noxious stimulation reduces withdrawal threshold to mechanical stimuli after spinal cord injury: Role of tumor necrosis factor alpha and apoptosis

The neuropathic phenotype of the K/BxN transgenic mouse with spontaneous arthritis: pain, nerve sprouting and joint remodeling

INTEGRATE-Pain: a transatlantic consortium to advance development of effective pain management

Critical NIH Resources to Advance Therapies for Pain: Preclinical Screening Program and Phase II Human Clinical Trial Network

Morphine self-administration following spinal cord injury

Long-lasting analgesia via targeted in situ repression of Na V 1.7 in mice

Preclinical target validation for non-addictive therapeutics development for pain

NIH HEAL Initiative: National Institute of Neurological Disorders and Stroke Preclinical Program for Non-Addictive Pain Therapeutic Development

Analgesia or addiction?: implications for morphine use after spinal cord injury

An IL-1 receptor antagonist blocks a morphine-induced attenuation of locomotor recovery after spinal cord injury

Self-delivering RNAi compounds as therapeutic agents in the central nervous system to enhance axonal regeneration after injury

The association between spinal cord trauma-sensitive miRNAs and pain sensitivity, and their regulation by morphine

ID: 208858 The NIH HEAL Initiative Preclinical Screening Platform for Pain

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