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Function of Mitochondrial Stat3 in Cellular Respiration

Science (American Association for the Advancement of Science), 2009-02, Vol.323 (5915), p.793-797

2009

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Growth Factor-dependent AKT Activation and Cell Migration Requires the Function of c-K(B)-Ras Versus Other Cellular Ras Isoforms

The Journal of biological chemistry, 2006-10, Vol.281 (40), p.29730-29738

2006

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K-Ras Regulates the Steady-state Expression of Matrix Metalloproteinase 2 in Fibroblasts

The Journal of biological chemistry, 2003-08, Vol.278 (34), p.31871-31878

2003

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Cellular N-Ras Promotes Cell Survival by Downregulation of Jun N-Terminal Protein Kinase and p38

Molecular and Cellular Biology, 2002-03, Vol.22 (5), p.1589-1606

2002

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Pulsed electromagnetic field treatments enhance the healing of fibular osteotomies

Journal of orthopaedic research, 2005-09, Vol.23 (5), p.1035-1046

2005

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Inhibition of the EGF-Activated MAP Kinase Signaling Pathway by Adenosine 3′,5′-Monophosphate

Science (American Association for the Advancement of Science), 1993-11, Vol.262 (5136), p.1065-1069

1993

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Endogenous c-N-Ras Provides a Steady-state Anti-apoptotic Signal

The Journal of biological chemistry, 2000-06, Vol.275 (25), p.19315-19323

2000

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Tyk2 Tyrosine Kinase Expression Is Required for the Maintenance of Mitochondrial Respiration in Primary Pro-B Lymphocytes

Molecular and Cellular Biology, 2006-11, Vol.26 (22), p.8562-8571

2006

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Mitogen-Activated Protein Kinase Kinase (MEK) Activity Is Required for Inhibition of Skeletal Muscle Differentiation by Insulin-Like Growth Factor 1 or Fibroblast Growth Factor 2

Endocrinology (Philadelphia), 1998-04, Vol.139 (4), p.1794-1800

1998

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Bone mass is preserved in a critical-sized osteotomy by low energy pulsed electromagnetic fields as quantitated by in vivo micro-computed tomography

Journal of orthopaedic research, 2004-09, Vol.22 (5), p.1086-1093

2004

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Cloning of TACC1, an embryonically expressed, potentially transforming coiled coil containing gene, from the 8p11 breast cancer amplicon

Oncogene, 1999-07, Vol.18 (27), p.4032-4038

1999

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Oncogenic Ha-Ras-dependent Mitogen-activated Protein Kinase Activity Requires Signaling Through the Epidermal Growth Factor Receptor

The Journal of biological chemistry, 1998-10, Vol.273 (43), p.28155-28162

1998

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A cytosolic protein catalyzes the release of GDP from p21ras

Science (American Association for the Advancement of Science), 1990-04, Vol.248 (4951), p.67-69

1990

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A Direct Interaction between Oncogenic Ha-Ras and Phosphatidylinositol 3-Kinase Is Not Required for Ha-Ras-dependent Transformation of Epithelial Cells

The Journal of biological chemistry, 2001-10, Vol.276 (43), p.39755-39764

2001

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Ras Mediates the Activation of Phospholipase D by v-Src (∗)

The Journal of biological chemistry, 1995-03, Vol.270 (11), p.6006-6009

1995

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Structural and functional consequences of c-N-Ras constitutively associated with intact mitochondria

Biochimica et biophysica acta, 2006-10, Vol.1763 (10), p.1108-1124

2006

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Complexes of Ras·GTP with Raf-1 and Mitogen-Activated Protein Kinase Kinase

Science (American Association for the Advancement of Science), 1993-06, Vol.260 (5114), p.1658-1661

1993

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Constitutive association of c-N-Ras with c-Raf-1 and protein kinase C epsilon in latent signaling modules

The Journal of biological chemistry, 2001-08, Vol.276 (31), p.29079-29090

2001

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Ha-ras and N-ras regulate MAPK activity by distinct mechanisms in vivo

Oncogene, 1998-03, Vol.16 (11), p.1417-1428

1998

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Mechanism of inhibition of Raf-1 by protein kinase A

Molecular and Cellular Biology, 1994-10, Vol.14 (10), p.6696-6703

1994

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Exposure of murine cells to pulsed electromagnetic fields rapidly activates the mTOR signaling pathway

Bioelectromagnetics, 2006-10, Vol.27 (7), p.535-544

2006

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The 3Rs of life: Ras, Raf and growth regulation

Trends in Genetics, 1994-02, Vol.10 (2), p.44-48

1994

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PKCα regulates phosphorylation and enzymatic activity of cPLA2 in vitro and in activated human monocytes

Cellular signalling, 2007-02, Vol.19 (2), p.359-366

2007

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A Cytosolic Protein Catalyzes the Release of GDP from p21$^{ras}

Science (American Association for the Advancement of Science), 1990-04, Vol.248 (4951), p.67-69

1990

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Exposure of mouse preosteoblasts to pulsed electromagnetic fields reduces the amount of mature, type I collagen in the extracellular matrix

Journal of orthopaedic research, 2006-02, Vol.24 (2), p.242-253

2006

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Function of Mitochondrial Stat3 in Cellular Respiration

Growth Factor-dependent AKT Activation and Cell Migration Requires the Function of c-K(B)-Ras Versus Other Cellular Ras Isoforms

K-Ras Regulates the Steady-state Expression of Matrix Metalloproteinase 2 in Fibroblasts

Cellular N-Ras Promotes Cell Survival by Downregulation of Jun N-Terminal Protein Kinase and p38

Pulsed electromagnetic field treatments enhance the healing of fibular osteotomies

Inhibition of the EGF-Activated MAP Kinase Signaling Pathway by Adenosine 3′,5′-Monophosphate

Endogenous c-N-Ras Provides a Steady-state Anti-apoptotic Signal

Tyk2 Tyrosine Kinase Expression Is Required for the Maintenance of Mitochondrial Respiration in Primary Pro-B Lymphocytes

Mitogen-Activated Protein Kinase Kinase (MEK) Activity Is Required for Inhibition of Skeletal Muscle Differentiation by Insulin-Like Growth Factor 1 or Fibroblast Growth Factor 2

Bone mass is preserved in a critical-sized osteotomy by low energy pulsed electromagnetic fields as quantitated by in vivo micro-computed tomography

Cloning of TACC1, an embryonically expressed, potentially transforming coiled coil containing gene, from the 8p11 breast cancer amplicon

Oncogenic Ha-Ras-dependent Mitogen-activated Protein Kinase Activity Requires Signaling Through the Epidermal Growth Factor Receptor

A cytosolic protein catalyzes the release of GDP from p21ras

A Direct Interaction between Oncogenic Ha-Ras and Phosphatidylinositol 3-Kinase Is Not Required for Ha-Ras-dependent Transformation of Epithelial Cells

Ras Mediates the Activation of Phospholipase D by v-Src (∗)

Structural and functional consequences of c-N-Ras constitutively associated with intact mitochondria

Complexes of Ras·GTP with Raf-1 and Mitogen-Activated Protein Kinase Kinase

Constitutive association of c-N-Ras with c-Raf-1 and protein kinase C epsilon in latent signaling modules

Ha-ras and N-ras regulate MAPK activity by distinct mechanisms in vivo

Mechanism of inhibition of Raf-1 by protein kinase A

Exposure of murine cells to pulsed electromagnetic fields rapidly activates the mTOR signaling pathway

The 3Rs of life: Ras, Raf and growth regulation

PKCα regulates phosphorylation and enzymatic activity of cPLA2 in vitro and in activated human monocytes

A Cytosolic Protein Catalyzes the Release of GDP from p21$^{ras}

Exposure of mouse preosteoblasts to pulsed electromagnetic fields reduces the amount of mature, type I collagen in the extracellular matrix

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