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Cellular toxicity of expanded RNA repeats: focus on RNA foci

Human molecular genetics, 2011-10, Vol.20 (19), p.3811-3821

2011

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Differential fates of introns in gene expression due to global alternative splicing

Human genetics, 2022-01, Vol.141 (1), p.31-47

2022

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An Overview of Circular RNAs and Their Implications in Myotonic Dystrophy

International journal of molecular sciences, 2019-09, Vol.20 (18), p.4385

2019

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CAG repeats mimic CUG repeats in the misregulation of alternative splicing

Nucleic acids research, 2011-11, Vol.39 (20), p.8938

2011

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New PCR-specific markers for pollen fertility restoration QRfp-4R in rye (Secale cereale L.) with Pampa sterilizing cytoplasm

Journal of applied genetics, 2021-12, Vol.62 (4), p.545-557

2021

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Splicing in two skeletal muscle transcripts correlates with clinical phenotype in myotonic dystrophy type 1 patients

Journal of neurology, 2022-05, Vol.269 (5), p.2784-2787

2022

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Small Molecules Which Improve Pathogenesis of Myotonic Dystrophy Type 1

Frontiers in neurology, 2018-05, Vol.9, p.349

2018

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RAN translation and frameshifting as translational challenges at simple repeats of human neurodegenerative disorders

Nucleic acids research, 2014-10, Vol.42 (19), p.11849

2014

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Quantitative Methods to Monitor RNA Biomarkers in Myotonic Dystrophy

Scientific reports, 2018-04, Vol.8 (1), p.5885-13, Article 5885

2018

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Cooperation meets competition in microRNA-mediated DMPK transcript regulation

Nucleic acids research, 2015-10, Vol.43 (19), p.9500

2015

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Dense mesh sampling for video-based facial animation

AIP Conference Proceedings, 2016, Vol.1738 (1)

2016

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Global Increase in Circular RNA Levels in Myotonic Dystrophy

Frontiers in genetics, 2019-07, Vol.10, p.649-649

2019

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CAG repeat RNA as an auxiliary toxic agent in polyglutamine disorders

RNA biology, 2011-07, Vol.8 (4), p.565-571

2011

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Small molecule kinase inhibitors alleviate different molecular features of myotonic dystrophy type 1

RNA biology, 2014-06, Vol.11 (6), p.742-754

2014

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(CCUG) n RNA toxicity in a Drosophila model of myotonic dystrophy type 2 (DM2) activates apoptosis

Disease models & mechanisms, 2017-08, Vol.10 (8), p.993-1003

2017

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The involvement of non-B DNA structures in gross chromosomal rearrangements

DNA repair, 2006-09, Vol.5 (9), p.1161-1170

2006

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Non-B DNA Conformations Formed by Long Repeating Tracts of Myotonic Dystrophy Type 1, Myotonic Dystrophy Type 2, and Friedreich's Ataxia Genes, Not the Sequences per se, Promote Mutagenesis in Flanking Regions

The Journal of biological chemistry, 2006-08, Vol.281 (34), p.24531-24543

2006

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The 4R QTLs pollen fertility restoration in triticale with CMS Triticum timopheevii

Crop science, 2023-01, Vol.63 (1), p.43-54

2023

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Mutant CAG repeats of Huntingtin transcript fold into hairpins, form nuclear foci and are targets for RNA interference

Nucleic acids research, 2011-05, Vol.39 (9), p.3852

2011

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The Myotonic Dystrophy Type 1 Triplet Repeat Sequence Induces Gross Deletions and Inversions

The Journal of biological chemistry, 2005-01, Vol.280 (2), p.941-952

2005

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Facial expression reconstruction on the basis of selected vertices of triangle mesh

AIP Conference Proceedings, 2016, Vol.1738 (1)

2016

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Using the Game Engine in the Animation Production Process

Intelligent Information and Database Systems: Recent Developments, p.209-220

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Triplet repeat RNA structure and its role as pathogenic agent and therapeutic target

Nucleic acids research, 2012-01, Vol.40 (1), p.11

2012

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Oncogenomic portals for the visualization and analysis of genome-wide cancer data

Oncotarget, 2016-01, Vol.7 (1), p.176-192

2016

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The 30 kb deletion in the APOBEC3 cluster decreases APOBEC3A and APOBEC3B expression and creates a transcriptionally active hybrid gene but does not associate with breast cancer in the European population

Oncotarget, 2017-09, Vol.8 (44), p.76357-76374

2017

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Cellular toxicity of expanded RNA repeats: focus on RNA foci

Differential fates of introns in gene expression due to global alternative splicing

An Overview of Circular RNAs and Their Implications in Myotonic Dystrophy

CAG repeats mimic CUG repeats in the misregulation of alternative splicing

New PCR-specific markers for pollen fertility restoration QRfp-4R in rye (Secale cereale L.) with Pampa sterilizing cytoplasm

Splicing in two skeletal muscle transcripts correlates with clinical phenotype in myotonic dystrophy type 1 patients

Small Molecules Which Improve Pathogenesis of Myotonic Dystrophy Type 1

RAN translation and frameshifting as translational challenges at simple repeats of human neurodegenerative disorders

Quantitative Methods to Monitor RNA Biomarkers in Myotonic Dystrophy

Cooperation meets competition in microRNA-mediated DMPK transcript regulation

Dense mesh sampling for video-based facial animation

Global Increase in Circular RNA Levels in Myotonic Dystrophy

CAG repeat RNA as an auxiliary toxic agent in polyglutamine disorders

Small molecule kinase inhibitors alleviate different molecular features of myotonic dystrophy type 1

(CCUG) n RNA toxicity in a Drosophila model of myotonic dystrophy type 2 (DM2) activates apoptosis

The involvement of non-B DNA structures in gross chromosomal rearrangements

Non-B DNA Conformations Formed by Long Repeating Tracts of Myotonic Dystrophy Type 1, Myotonic Dystrophy Type 2, and Friedreich's Ataxia Genes, Not the Sequences per se, Promote Mutagenesis in Flanking Regions

The 4R QTLs pollen fertility restoration in triticale with CMS Triticum timopheevii

Mutant CAG repeats of Huntingtin transcript fold into hairpins, form nuclear foci and are targets for RNA interference

The Myotonic Dystrophy Type 1 Triplet Repeat Sequence Induces Gross Deletions and Inversions

Facial expression reconstruction on the basis of selected vertices of triangle mesh

Using the Game Engine in the Animation Production Process

Triplet repeat RNA structure and its role as pathogenic agent and therapeutic target

Oncogenomic portals for the visualization and analysis of genome-wide cancer data

The 30 kb deletion in the APOBEC3 cluster decreases APOBEC3A and APOBEC3B expression and creates a transcriptionally active hybrid gene but does not associate with breast cancer in the European population

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