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Different pathways of molecular pathophysiology underlie cognitive and motor tauopathy phenotypes in transgenic models for Alzheimer’s disease and frontotemporal lobar degeneration

Cellular and molecular life sciences : CMLS, 2015-06, Vol.72 (11), p.2199-2222

2015

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Selective Inhibition of Alzheimer Disease-Like Tau Aggregation by Phenothiazines

Proceedings of the National Academy of Sciences - PNAS, 1996-10, Vol.93 (20), p.11213-11218

1996

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Oral Tau Aggregation Inhibitor for Alzheimer’s Disease: Design, Progress and Basis for Selection of the 16 mg/day Dose in a Phase 3, Randomized, Placebo-Controlled Trial of Hydromethylthionine Mesylate

The Journal of Prevention of Alzheimer's Disease, 2022, Vol.9 (4), p.780-790

2022

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Loss of synaptic but not cytoskeletal proteins in the cerebellum of chronic schizophrenics

Neuroscience letters, 2002-01, Vol.317 (3), p.161-165

2002

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Molecular characterization of the minimal protease resistant tau unit of the Alzheimer's disease paired helical filament

The EMBO journal, 1993-01, Vol.12 (1), p.365-370

1993

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Staging of Cytoskeletal and β-Amyloid Changes in Human Isocortex Reveals Biphasic Synaptic Protein Response during Progression of Alzheimer’s Disease

The American journal of pathology, 2000-08, Vol.157 (2), p.623-636

2000

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Cloning and Sequencing of the cDNA Encoding a Core Protein of the Paired Helical Filament of Alzheimer Disease: Identification as the Microtubule-Associated Protein Tau

Proceedings of the National Academy of Sciences - PNAS, 1988-06, Vol.85 (11), p.4051-4055

1988

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Structural Characterization of the Core of the Paired Helical Filament of Alzheimer Disease

Proceedings of the National Academy of Sciences - PNAS, 1988-07, Vol.85 (13), p.4884-4888

1988

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Isolation of a Fragment of Tau Derived from the Core of the Paired Helical Filament of Alzheimer Disease

Proceedings of the National Academy of Sciences - PNAS, 1988-06, Vol.85 (12), p.4506-4510

1988

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Influence of apolipoprotein E genotype on senile dementia of the Alzheimer and Lewy body types. Significance for etiological theories of Alzheimer's disease

The American journal of pathology, 1994-12, Vol.145 (6), p.1472-1484

1994

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The extent of neurofibrillary pathology in perforant pathway neurons is the key determinant of dementia in the very old

Acta neuropathologica, 2000-07, Vol.100 (1), p.29-35

2000

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Examination of the validity of the hierarchical model of neuropathological staging in normal aging and Alzheimer's disease

Acta neuropathologica, 1998-02, Vol.95 (2), p.154-158

1998

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α-Synuclein Inclusions in Alzheimer and Lewy Body Diseases

Journal of neuropathology and experimental neurology, 2000-05, Vol.59 (5), p.408-417

2000

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Alzheimer's-disease-like changes in tau protein processing: association with aluminium accumulation in brains of renal dialysis patients

The Lancet (British edition), 1994-04, Vol.343 (8904), p.993-997

1994

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Difference between the Tau Protein of Alzheimer Paired Helical Filament Core and Normal Tau Revealed by Epitope Analysis of Monoclonal Antibodies 423 and 7.51

Proceedings of the National Academy of Sciences - PNAS, 1991-07, Vol.88 (13), p.5837-5841

1991

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Staging the pathological assembly of truncated tau protein into paired helical filaments in Alzheimer's disease

Acta neuropathologica, 1996, Vol.91 (6), p.633-641

1996

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Biochemical and anatomical redistribution of tau protein in Alzheimer's disease

The American journal of pathology, 1993-08, Vol.143 (2), p.565-578

1993

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Molecular analysis of neurofibrillary degeneration in Alzheimer's disease. An immunohistochemical study

The American journal of pathology, 1990-09, Vol.137 (3), p.711-723

1990

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Monitoring pathological assembly of tau and β-amyloid proteins in Alzheimer's disease

Acta neuropathologica, 1995, Vol.89 (1), p.50-56

1995

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Image reconstruction of the Alzheimer paired helical filament

The EMBO journal, 1985-12, Vol.4 (13B), p.3661-3665

1985

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Sequestration of tau by granulovacuolar degeneration in Alzheimer's disease

The American journal of pathology, 1991-09, Vol.139 (3), p.641-647

1991

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Subunit Structure of Paired Helical Filaments in Alzheimer's Disease

The Journal of cell biology, 1985-06, Vol.100 (6), p.1905-1912

1985

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Apolipoprotein E genotype in the prediction of cognitive decline and dementia in a prospectively studied elderly population

Dementia (Basel, Switzerland), 1996-05, Vol.7 (3), p.169-174

1996

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Immunohistochemical Staging of Neurofibrillary Degeneration in Alzheimerʼs Disease

Journal of neuropathology and experimental neurology, 1994-03, Vol.53 (2), p.158-164

1994

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Angiotensin converting enzyme density is increased in temporal cortex from patients with Alzheimer's disease

European journal of pharmacology, 1991-08, Vol.200 (2-3), p.289-292

1991

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Different pathways of molecular pathophysiology underlie cognitive and motor tauopathy phenotypes in transgenic models for Alzheimer’s disease and frontotemporal lobar degeneration

Selective Inhibition of Alzheimer Disease-Like Tau Aggregation by Phenothiazines

Oral Tau Aggregation Inhibitor for Alzheimer’s Disease: Design, Progress and Basis for Selection of the 16 mg/day Dose in a Phase 3, Randomized, Placebo-Controlled Trial of Hydromethylthionine Mesylate

Loss of synaptic but not cytoskeletal proteins in the cerebellum of chronic schizophrenics

Molecular characterization of the minimal protease resistant tau unit of the Alzheimer's disease paired helical filament

Staging of Cytoskeletal and β-Amyloid Changes in Human Isocortex Reveals Biphasic Synaptic Protein Response during Progression of Alzheimer’s Disease

Cloning and Sequencing of the cDNA Encoding a Core Protein of the Paired Helical Filament of Alzheimer Disease: Identification as the Microtubule-Associated Protein Tau

Structural Characterization of the Core of the Paired Helical Filament of Alzheimer Disease

Isolation of a Fragment of Tau Derived from the Core of the Paired Helical Filament of Alzheimer Disease

Influence of apolipoprotein E genotype on senile dementia of the Alzheimer and Lewy body types. Significance for etiological theories of Alzheimer's disease

The extent of neurofibrillary pathology in perforant pathway neurons is the key determinant of dementia in the very old

Examination of the validity of the hierarchical model of neuropathological staging in normal aging and Alzheimer's disease

α-Synuclein Inclusions in Alzheimer and Lewy Body Diseases

Alzheimer's-disease-like changes in tau protein processing: association with aluminium accumulation in brains of renal dialysis patients

Difference between the Tau Protein of Alzheimer Paired Helical Filament Core and Normal Tau Revealed by Epitope Analysis of Monoclonal Antibodies 423 and 7.51

Staging the pathological assembly of truncated tau protein into paired helical filaments in Alzheimer's disease

Biochemical and anatomical redistribution of tau protein in Alzheimer's disease

Molecular analysis of neurofibrillary degeneration in Alzheimer's disease. An immunohistochemical study

Monitoring pathological assembly of tau and β-amyloid proteins in Alzheimer's disease

Image reconstruction of the Alzheimer paired helical filament

Sequestration of tau by granulovacuolar degeneration in Alzheimer's disease

Subunit Structure of Paired Helical Filaments in Alzheimer's Disease

Apolipoprotein E genotype in the prediction of cognitive decline and dementia in a prospectively studied elderly population

Immunohistochemical Staging of Neurofibrillary Degeneration in Alzheimerʼs Disease

Angiotensin converting enzyme density is increased in temporal cortex from patients with Alzheimer's disease

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