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NFKB1: a suppressor of inflammation, ageing and cancer

The FEBS journal, 2016-05, Vol.283 (10), p.1812-1822

2016

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Cellular senescence drives age-dependent hepatic steatosis

Nature communications, 2017-06, Vol.8 (1), p.15691-15691, Article 15691

2017

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NFKB1 and Cancer: Friend or Foe?

Cells (Basel, Switzerland), 2018-09, Vol.7 (9), p.133

2018

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Multigenerational epigenetic adaptation of the hepatic wound-healing response

Nature medicine, 2012-09, Vol.18 (9), p.1369-1377

2012

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A TLR2/S100A9/CXCL-2 signaling network is necessary for neutrophil recruitment in acute and chronic liver injury in the mouse

Journal of hepatology, 2014-04, Vol.60 (4), p.782-791

2014

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Histone methyltransferase ASH1 orchestrates fibrogenic gene transcription during myofibroblast transdifferentiation

Hepatology (Baltimore, Md.), 2012-09, Vol.56 (3), p.1129-1139

2012

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UCHL1-dependent control of Hypoxia-Inducible Factor Transcriptional Activity during Liver Fibrosis

Bioscience reports, 2024-06, Vol.44 (6), p.1

2024

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Quiescent hepatic stellate cells functionally contribute to the hepatic innate immune response via TLR3

PloS one, 2014-01, Vol.9 (1), p.e83391-e83391

2014

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Tumor progression locus 2/Cot is required for activation of extracellular regulated kinase in liver injury and toll‐like receptor–induced TIMP‐1 gene transcription in hepatic stellate cells in mice

Hepatology (Baltimore, Md.), 2013-03, Vol.57 (3), p.1238-1249

2013

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Epigenetic reprogramming in liver fibrosis and cancer

Advanced drug delivery reviews, 2017-11, Vol.121, p.124-132

2017

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Centrosome dysfunction associated with somatic expression of the synaptonemal complex protein TEX12

Communications biology, 2021-12, Vol.4 (1), p.1371-1371, Article 1371

2021

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Moderate Exercise Inhibits Age-Related Inflammation, Liver Steatosis, Senescence, and Tumorigenesis

The Journal of immunology (1950), 2021-02, Vol.206 (4), p.904-916

2021

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Neutrophils: driving progression and poor prognosis in hepatocellular carcinoma?

British journal of cancer, 2018-01, Vol.118 (2), p.248-257

2018

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Multigenerational epigenetic adaptation of the hepatic wound-healing response

Nature medicine, 2012-09, Vol.18 (9), p.1369-1377

2012

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Roles of c-Rel signalling in inflammation and disease

The international journal of biochemistry & cell biology, 2012-06, Vol.44 (6), p.851-860

2012

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Ubiquitin C-terminal hydrolase 1: A novel functional marker for liver myofibroblasts and a therapeutic target in chronic liver disease

Journal of hepatology, 2015-12, Vol.63 (6), p.1421-1428

2015

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The NF-κB1 is a key regulator of acute but not chronic renal injury

Cell death & disease, 2017-06, Vol.8 (6), p.e2883-e2883

2017

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Antigen‐specific B lymphocytes acquire proteoglycan aggrecan from cartilage extracellular matrix resulting in antigen presentation and CD4+ T‐cell activation

Immunology, 2014-01, Vol.141 (1), p.70-78

2014

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Presentation of the candidate rheumatoid arthritis autoantigen aggrecan by antigen‐specific B cells induces enhanced CD4+ T helper type 1 subset differentiation

Immunology, 2012-04, Vol.135 (4), p.344-354

2012

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Multigenerational epigenetic adaptation of the hepatic wound-healing response

Nature medicine, 2012-10, Vol.18 (10), p.1592

2012

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Platelet GPIbα is a mediator and potential interventional target for NASH and subsequent liver cancer

Nature medicine, 2019-04, Vol.25 (4), p.641-655

2019

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HDAC1 interacts with the p50 NF-κB subunit via its nuclear localization sequence to constrain inflammatory gene expression

Biochimica et biophysica acta. Gene regulatory mechanisms, 2018-10, Vol.1861 (10), p.962-970

2018

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Author Correction: Platelet GPIbα is a mediator and potential interventional target for NASH and subsequent liver cancer

Nature medicine, 2022-03, Vol.28 (3), p.600-600

2022

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Antigen‐specific B lymphocytes acquire proteoglycan aggrecan from cartilage extracellular matrix resulting in antigen presentation and CD 4 + T ‐cell activation

Immunology, 2014-01, Vol.141 (1), p.70-78

2014

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NFKB1: a suppressor of inflammation, ageing and cancer

Cellular senescence drives age-dependent hepatic steatosis

NFKB1 and Cancer: Friend or Foe?

Multigenerational epigenetic adaptation of the hepatic wound-healing response

A TLR2/S100A9/CXCL-2 signaling network is necessary for neutrophil recruitment in acute and chronic liver injury in the mouse

Histone methyltransferase ASH1 orchestrates fibrogenic gene transcription during myofibroblast transdifferentiation

UCHL1-dependent control of Hypoxia-Inducible Factor Transcriptional Activity during Liver Fibrosis

Quiescent hepatic stellate cells functionally contribute to the hepatic innate immune response via TLR3

Tumor progression locus 2/Cot is required for activation of extracellular regulated kinase in liver injury and toll‐like receptor–induced TIMP‐1 gene transcription in hepatic stellate cells in mice

Epigenetic reprogramming in liver fibrosis and cancer

Centrosome dysfunction associated with somatic expression of the synaptonemal complex protein TEX12

Neutrophils induce paracrine telomere dysfunction and senescence in ROS‐dependent manner

Moderate Exercise Inhibits Age-Related Inflammation, Liver Steatosis, Senescence, and Tumorigenesis

Neutrophils: driving progression and poor prognosis in hepatocellular carcinoma?

Multigenerational epigenetic adaptation of the hepatic wound-healing response

Roles of c-Rel signalling in inflammation and disease

Ubiquitin C-terminal hydrolase 1: A novel functional marker for liver myofibroblasts and a therapeutic target in chronic liver disease

The NF-κB1 is a key regulator of acute but not chronic renal injury

Antigen‐specific B lymphocytes acquire proteoglycan aggrecan from cartilage extracellular matrix resulting in antigen presentation and CD4+ T‐cell activation

Presentation of the candidate rheumatoid arthritis autoantigen aggrecan by antigen‐specific B cells induces enhanced CD4+ T helper type 1 subset differentiation

Multigenerational epigenetic adaptation of the hepatic wound-healing response

Platelet GPIbα is a mediator and potential interventional target for NASH and subsequent liver cancer

HDAC1 interacts with the p50 NF-κB subunit via its nuclear localization sequence to constrain inflammatory gene expression

Author Correction: Platelet GPIbα is a mediator and potential interventional target for NASH and subsequent liver cancer

Antigen‐specific B lymphocytes acquire proteoglycan aggrecan from cartilage extracellular matrix resulting in antigen presentation and CD 4 + T ‐cell activation

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