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Genome-Wide Association Study of Late-Onset Myasthenia Gravis: Confirmation of TNFRSF11A and Identification of ZBTB10 and Three Distinct HLA Associations

Molecular medicine (Cambridge, Mass.), 2015-01, Vol.21 (1), p.769-781

2015

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Spontaneous production of anti‐IFN‐α and anti‐IL‐12 autoantibodies by thymoma cells from myasthenia gravis patients suggests autoimmunization in the tumor

International immunology, 2003-08, Vol.15 (8), p.903-913

2003

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Mature, long-lived CD4+ and CD8+ T cells are generated by the thymoma in myasthenia gravis

Annals of neurology, 2001-07, Vol.50 (1), p.64-72

2001

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Acetylcholine receptors in human thymic myoid cells in situ: An immunohistological study

Annals of neurology, 1987-08, Vol.22 (2), p.212-222

1987

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Human vascular endothelial cells process and present autoantigen to human T cell lines

International immunology, 1995-03, Vol.7 (3), p.471-479

1995

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Clonal expansions of CD4+ B helper T cells in autoimmune myasthenia gravis

European journal of immunology, 2007-03, Vol.37 (3), p.849-863

2007

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Preferential expression of AChR ε-subunit in thymomas from patients with myasthenia gravis

Journal of neuroimmunology, 2008-09, Vol.201, p.28-32

2008

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Distant interactions between dimorphisms in HLA-DR4 radically affect recognition of defined peptides by a specific T cell clone

International immunology, 1999-05, Vol.11 (5), p.835-843

1999

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VAV1 and BAFF, via NFκB pathway, are genetic risk factors for myasthenia gravis

Annals of clinical and translational neurology, 2014-05, Vol.1 (5), p.329-339

2014

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Clonal expansions of CD4 super(+) B helper T cells in autoimmune myasthenia gravis

European Journal of Immunology, 2007-01, Vol.37 (3), p.849-863

2007

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Eurojunction ’99. The 5th European Conference on Myasthenia gravis and Other Disorders of the Neuromuscular Junction

European neurology, 2000-02, Vol.43 (2), p.123-124

2000

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VAV1 and BAFF, via NFκB pathway, are genetic risk factors for myasthenia gravis

Annals of clinical and translational neurology, 2014-04

2014

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A pathogenetic role for the thymoma in myasthenia gravis. Autosensitization of IL-4- producing T cell clones recognizing extracellular acetylcholine receptor epitopes presented by minority class II isotypes

The Journal of clinical investigation, 1998-05, Vol.101 (10), p.2268-2277

1998

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Obituaries: Philip Henry Almroth Willcox

BMJ. British medical journal (International ed.), 2000-04, Vol.320 (7241), p.1080

International edition, 2000

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VAV 1 and BAFF , via NF κB pathway, are genetic risk factors for myasthenia gravis

Annals of clinical and translational neurology, 2014-05, Vol.1 (5), p.329-339

2014

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Control of autoimmunity by ‘epitope theft’

Trends in molecular medicine, 2005, Vol.11 (1), p.1-4

2005

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MHC class II‐associated invariant chain peptide replacement by T cell epitopes: engineered invariant chain as a vehicle for directed and enhanced MHC class II antigen processing and presentation

European journal of immunology, 1998-05, Vol.28 (5), p.1524-1533

1998

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Major Differences in Antigen-Processing Correlate with a Single Arg71{leftrightarrow}Lys Substitution in HLA-DR Molecules Predisposing to Rheumatoid Arthritis and with Their Selective Interactions with 70-kDa Heat Shock Protein Chaperones

The Journal of immunology (1950), 2002-09, Vol.169 (6), p.3015-3020

2002

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Scanning a DRB30101 (DR52a)-Restricted Epitope Cross-Presented by DR3: Overlapping Natural and Artificial Determinants in the Human Acetylcholine Receptor

The Journal of immunology (1950), 1999-04, Vol.162 (7), p.4079-4087

1999

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Antibodies Affecting Ion Channel Function in Acquired Neuromyotonia, in Seropositive and Seronegative Myasthenia Gravis, and in Antibody-mediated Arthrogryposis Multiplex Congenita

Annals of the New York Academy of Sciences, 1998-05, Vol.841 (1), p.482-496

1998

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Risk for myasthenia gravis maps to a 151Pro→Ala change in TNIP1 and to human leukocyte antigen-B08

Genome-Wide Association Study of Late-Onset Myasthenia Gravis: Confirmation of TNFRSF11A and Identification of ZBTB10 and Three Distinct HLA Associations

Spontaneous production of anti‐IFN‐α and anti‐IL‐12 autoantibodies by thymoma cells from myasthenia gravis patients suggests autoimmunization in the tumor

Risk for myasthenia gravis maps to a (151) Pro→Ala change in TNIP1 and to human leukocyte antigen-B08

Mature, long-lived CD4+ and CD8+ T cells are generated by the thymoma in myasthenia gravis

Acetylcholine receptors in human thymic myoid cells in situ: An immunohistological study

Human vascular endothelial cells process and present autoantigen to human T cell lines

Clonal expansions of CD4+ B helper T cells in autoimmune myasthenia gravis

Preferential expression of AChR ε-subunit in thymomas from patients with myasthenia gravis

Distant interactions between dimorphisms in HLA-DR4 radically affect recognition of defined peptides by a specific T cell clone

Risk for myasthenia gravis maps to a super(151)Pro arrow right Ala change in TNIP1 and to human leukocyte antigen-B08

Risk for myasthenia gravis maps to a 151 Pro→Ala change in TNIP1 and to human leukocyte antigen‐B08

Risk for Myasthenia Gravis maps to 151Pro→ Ala change in TNIP1 and to HLA-B08

VAV1 and BAFF, via NFκB pathway, are genetic risk factors for myasthenia gravis

Clonal expansions of CD4 super(+) B helper T cells in autoimmune myasthenia gravis

Eurojunction ’99. The 5th European Conference on Myasthenia gravis and Other Disorders of the Neuromuscular Junction

VAV1 and BAFF, via NFκB pathway, are genetic risk factors for myasthenia gravis

A pathogenetic role for the thymoma in myasthenia gravis. Autosensitization of IL-4- producing T cell clones recognizing extracellular acetylcholine receptor epitopes presented by minority class II isotypes

Obituaries: Philip Henry Almroth Willcox

VAV 1 and BAFF , via NF κB pathway, are genetic risk factors for myasthenia gravis

Control of autoimmunity by ‘epitope theft’

MHC class II‐associated invariant chain peptide replacement by T cell epitopes: engineered invariant chain as a vehicle for directed and enhanced MHC class II antigen processing and presentation

Major Differences in Antigen-Processing Correlate with a Single Arg71{leftrightarrow}Lys Substitution in HLA-DR Molecules Predisposing to Rheumatoid Arthritis and with Their Selective Interactions with 70-kDa Heat Shock Protein Chaperones

Scanning a DRB30101 (DR52a)-Restricted Epitope Cross-Presented by DR3: Overlapping Natural and Artificial Determinants in the Human Acetylcholine Receptor

Antibodies Affecting Ion Channel Function in Acquired Neuromyotonia, in Seropositive and Seronegative Myasthenia Gravis, and in Antibody-mediated Arthrogryposis Multiplex Congenita

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