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Estimating Podocyte Number and Density Using a Single Histologic Section

Journal of the American Society of Nephrology, 2014-05, Vol.25 (5), p.1118-1129

2014

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Growth-Dependent Podocyte Failure Causes Glomerulosclerosis

Journal of the American Society of Nephrology, 2012-08, Vol.23 (8), p.1351-1363

2012

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Two-Photon Intravital Fluorescence Lifetime Imaging of the Kidney Reveals Cell-Type Specific Metabolic Signatures

Journal of the American Society of Nephrology, 2017-08, Vol.28 (8), p.2420-2430

2017

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mTORC1 activation in podocytes is a critical step in the development of diabetic nephropathy in mice

The Journal of clinical investigation, 2011-06, Vol.121 (6), p.2181-2196

2011

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FSGS as an Adaptive Response to Growth-Induced Podocyte Stress

Journal of the American Society of Nephrology, 2017-10, Vol.28 (10), p.2931-2945

2017

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Podocyte hypertrophic stress and detachment precedes hyperglycemia or albuminuria in a rat model of obesity and type2 diabetes-associated nephropathy

Scientific reports, 2019-12, Vol.9 (1), p.18485-15, Article 18485

2019

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Critical timing of ACEi initiation prevents compensatory glomerular hypertrophy in the remaining single kidney

Scientific reports, 2021-10, Vol.11 (1), p.19605-19605, Article 19605

2021

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Podocyte Depletion in Thin GBM and Alport Syndrome

PloS one, 2016-05, Vol.11 (5), p.e0155255-e0155255

2016

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Accelerated podocyte detachment early after kidney transplantation is related to long-term allograft loss of function

Nephrology, dialysis, transplantation, 2019-07, Vol.34 (7), p.1232-1239

2019

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Urinary podocyte mRNAs precede microalbuminuria as a progression risk marker in human type 2 diabetic nephropathy

Scientific reports, 2020-10, Vol.10 (1), p.18209-18209, Article 18209

2020

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Organoid single cell profiling identifies a transcriptional signature of glomerular disease

JCI insight, 2019-01, Vol.4 (1)

2019

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Podometrics as a Potential Clinical Tool for Glomerular Disease Management

Seminars in nephrology, 2015-05, Vol.35 (3), p.245-255

2015

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Urinary podocyte and TGF-β1 mRNA as markers for disease activity and progression in anti-glomerular basement membrane nephritis

Nephrology, dialysis, transplantation, 2017-11, Vol.32 (11), p.1818-1830

2017

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Podocyte-specific expression of cre recombinase in transgenic mice

Genesis (New York, N.Y. : 2000), 2003-01, Vol.35 (1), p.39-42

2003

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Glomerular Aging and Focal Global Glomerulosclerosis: A Podometric Perspective

Journal of the American Society of Nephrology, 2015-12, Vol.26 (12), p.3162-3178

2015

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Fyn Binds to and Phosphorylates the Kidney Slit Diaphragm Component Nephrin

The Journal of biological chemistry, 2003-06, Vol.278 (23), p.20716-20723

2003

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Accelerated podocyte detachment and progressive podocyte loss from glomeruli with age in Alport Syndrome

Kidney international, 2017-12, Vol.92 (6), p.1515-1525

2017

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Urinary podocyte markers of disease activity, therapeutic efficacy, and long-term outcomes in acute and chronic kidney diseases

Clinical and experimental nephrology, 2024-06, Vol.28 (6), p.496-504

2024

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Growth-dependent podocyte failure causes glomerulosclerosis

Journal of the American Society of Nephrology, 2012-08, Vol.23 (8), p.1351-1363

2012

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Antibodies to protein tyrosine phosphatase receptor type O (PTPro) increase glomerular albumin permeability (P(alb))

American Journal of Physiology - Renal Physiology, 2009-07, Vol.297 (1), p.F138-F144

2009

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Podocyte stress and detachment measured in urine are related to mean arterial pressure in healthy humans

Kidney international, 2020-09, Vol.98 (3), p.699-707

2020

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Podocyte depletion causes glomerulosclerosis : Diphtheria toxin-induced podocyte depletion in rats expressing human diphtheria toxin receptor transgene

Journal of the American Society of Nephrology, 2005-10, Vol.16 (10), p.2941-2952

2005

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Quantitative morphometrics reveals glomerular changes in patients with infrequent segmentally sclerosed glomeruli

Journal of clinical pathology, 2022-02, Vol.75 (2), p.121-127

2022

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Podocyte number and density changes during early human life

Pediatric nephrology (Berlin, West), 2017-05, Vol.32 (5), p.823-834

2017

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Angiotensin II-dependent persistent podocyte loss from destabilized glomeruli causes progression of end stage kidney disease

Kidney international, 2012-01, Vol.81 (1), p.40-55

2012

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Estimating Podocyte Number and Density Using a Single Histologic Section

Growth-Dependent Podocyte Failure Causes Glomerulosclerosis

Two-Photon Intravital Fluorescence Lifetime Imaging of the Kidney Reveals Cell-Type Specific Metabolic Signatures

mTORC1 activation in podocytes is a critical step in the development of diabetic nephropathy in mice

FSGS as an Adaptive Response to Growth-Induced Podocyte Stress

Podocyte hypertrophic stress and detachment precedes hyperglycemia or albuminuria in a rat model of obesity and type2 diabetes-associated nephropathy

Critical timing of ACEi initiation prevents compensatory glomerular hypertrophy in the remaining single kidney

Podocyte Depletion in Thin GBM and Alport Syndrome

Accelerated podocyte detachment early after kidney transplantation is related to long-term allograft loss of function

Urinary podocyte mRNAs precede microalbuminuria as a progression risk marker in human type 2 diabetic nephropathy

Organoid single cell profiling identifies a transcriptional signature of glomerular disease

Podometrics as a Potential Clinical Tool for Glomerular Disease Management

Urinary podocyte and TGF-β1 mRNA as markers for disease activity and progression in anti-glomerular basement membrane nephritis

Podocyte-specific expression of cre recombinase in transgenic mice

Glomerular Aging and Focal Global Glomerulosclerosis: A Podometric Perspective

Fyn Binds to and Phosphorylates the Kidney Slit Diaphragm Component Nephrin

Accelerated podocyte detachment and progressive podocyte loss from glomeruli with age in Alport Syndrome

Urinary podocyte markers of disease activity, therapeutic efficacy, and long-term outcomes in acute and chronic kidney diseases

Growth-dependent podocyte failure causes glomerulosclerosis

Antibodies to protein tyrosine phosphatase receptor type O (PTPro) increase glomerular albumin permeability (P(alb))

Podocyte stress and detachment measured in urine are related to mean arterial pressure in healthy humans

Podocyte depletion causes glomerulosclerosis : Diphtheria toxin-induced podocyte depletion in rats expressing human diphtheria toxin receptor transgene

Quantitative morphometrics reveals glomerular changes in patients with infrequent segmentally sclerosed glomeruli

Podocyte number and density changes during early human life

Angiotensin II-dependent persistent podocyte loss from destabilized glomeruli causes progression of end stage kidney disease

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