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Impact of setanaxib on quality of life outcomes in primary biliary cholangitis in a phase 2 randomized controlled trial

Hepatology communications, 2023-03, Vol.7 (3), p.e0057-e0057

2023

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Transforming Growth Factor-β1 Inhibition of Macrophage Activation Is Mediated via Smad3

The Journal of biological chemistry, 2000-11, Vol.275 (47), p.36653-36658

2000

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Transforming Growth Factor-β1 Inhibits Cytokine-mediated Induction of Human Metalloelastase in Macrophages

The Journal of biological chemistry, 2000-08, Vol.275 (33), p.25766-25773

2000

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Setanaxib, a first‐in‐class selective NADPH oxidase 1/4 inhibitor for primary biliary cholangitis: A randomized, placebo‐controlled, phase 2 trial

Liver international, 2023-07, Vol.43 (7), p.1507-1522

2023

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NOX4 Inhibition Potentiates Immunotherapy by Overcoming Cancer-Associated Fibroblast-Mediated CD8 T-cell Exclusion from Tumors

Cancer research (Chicago, Ill.), 2020-05, Vol.80 (9), p.1846-1860

2020

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Two-Kidney, One Clip and One-Kidney, One Clip Hypertension in Mice

Hypertension (Dallas, Tex. 1979), 1997-04, Vol.29 (4), p.1025-1030

1997

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Quality of life outcomes in patients with primary biliary cholangitis treated with setanaxib: post-hoc results from a phase 2 randomised, placebo-controlled trial

Journal of hepatology, 2022-07, Vol.77, p.S94-S95

2022

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Thioredoxin Facilitates the Induction of Heme Oxygenase-1 in Response to Inflammatory Mediators

The Journal of biological chemistry, 2000-08, Vol.275 (32), p.24840-24846

2000

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Exacerbation of Chronic Renovascular Hypertension and Acute Renal Failure in Heme Oxygenase-1–Deficient Mice

Circulation research, 2001-05, Vol.88 (10), p.1088-1094

2001

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Targeting the NADPH Oxidase-4 and Liver X Receptor Pathway Preserves Schwann Cell Integrity in Diabetic Mice

Diabetes (New York, N.Y.), 2020-03, Vol.69 (3), p.448-464

2020

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Aortic connexin43 is decreased during hypertension induced by inhibition of nitric oxide synthase

Arteriosclerosis, thrombosis, and vascular biology, 1999-07, Vol.19 (7), p.1615

1999

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Hypoxia induces severe right ventricular dilatation and infarction in heme oxygenase-1 null mice

The Journal of clinical investigation, 1999-04, Vol.103 (8), p.R23-R29

1999

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Endotoxin-induced mortality is related to increased oxidative stress and end-organ dysfunction, not refractory hypotension, in heme oxygenase-1-deficient mice

Circulation (New York, N.Y.), 2000-12, Vol.102 (24), p.3015-3022

2000

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Hypoxia induces severe right ventricular dilatation and infarction in heme oxygenase-1 null mice

The Journal of clinical investigation, 1999-04, Vol.103 (8), p.R23-R29

1999

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Down-regulation of High Mobility Group-I(Y) Protein Contributes to the Inhibition of Nitric-oxide Synthase 2 by Transforming Growth Factor-β1

The Journal of biological chemistry, 2001-01, Vol.276 (2), p.1653-1659

2001

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Induction of heme oxygenase-1 during endotoxemia is downregulated by transforming growth factor-β1

Circulation research, 1998-08, Vol.83 (4), p.396-403

1998

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Role of activating protein‐1 and high mobility group‐I(Y) protein in the induction of CD44 gene expression by interleukin‐1β in vascular smooth muscle cells

The FASEB journal, 2000-02, Vol.14 (2), p.368-378

2000

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Induction of Heme Oxygenase-1 During Endotoxemia Is Downregulated by Transforming Growth Factor-beta 1

Circulation research, 1998-08, Vol.83 (4), p.396-403

1998

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High Mobility Group-I(Y) Protein Facilitates Nuclear Factor-κB Binding and Transactivation of the Inducible Nitric-oxide Synthase Promoter/Enhancer

The Journal of biological chemistry, 1999-03, Vol.274 (13), p.9045-9052

1999

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Exacerbation of chronic renovascular hypertension adn acute renal failure in heme oxygenase-1-deficient mice

Circulation research, 2001-05, Vol.88 (10), p.1088

2001

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Induction of heme oxygenase-1 during endotoxemia is downregulated by transforming growth (factor-beta 1)

Circulation research, 1998-08, Vol.83 (4), p.396

1998

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Spliceosomal peptide P140 for immunotherapy of systemic lupus erythematosus: Results of an early phase II clinical trial

Arthritis and rheumatism, 2008-12, Vol.58 (12), p.3873-3883

2008

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Down-regulation of High Mobility Group-I(Y) Protein Contributes to the Inhibition of Nitric-oxide Synthase 2 by Transforming Growth Factor-Î²1

The Journal of biological chemistry, 2001-01, Vol.276 (2), p.1653

2001

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Time Course Changes of the Mechanical Properties of the Carotid Artery in Renal Hypertensive Rats

Hypertension (Dallas, Tex. 1979), 1997-05, Vol.29 (5), p.1199-1203

1997

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Transforming Growth Factor-Î²1 Inhibition of Macrophage Activation Is Mediated via Smad3

The Journal of biological chemistry, 2000-11, Vol.275 (47), p.36653

2000

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Impact of setanaxib on quality of life outcomes in primary biliary cholangitis in a phase 2 randomized controlled trial

Transforming Growth Factor-β1 Inhibition of Macrophage Activation Is Mediated via Smad3

Transforming Growth Factor-β1 Inhibits Cytokine-mediated Induction of Human Metalloelastase in Macrophages

Setanaxib, a first‐in‐class selective NADPH oxidase 1/4 inhibitor for primary biliary cholangitis: A randomized, placebo‐controlled, phase 2 trial

NOX4 Inhibition Potentiates Immunotherapy by Overcoming Cancer-Associated Fibroblast-Mediated CD8 T-cell Exclusion from Tumors

Two-Kidney, One Clip and One-Kidney, One Clip Hypertension in Mice

Quality of life outcomes in patients with primary biliary cholangitis treated with setanaxib: post-hoc results from a phase 2 randomised, placebo-controlled trial

Thioredoxin Facilitates the Induction of Heme Oxygenase-1 in Response to Inflammatory Mediators

Exacerbation of Chronic Renovascular Hypertension and Acute Renal Failure in Heme Oxygenase-1–Deficient Mice

Targeting the NADPH Oxidase-4 and Liver X Receptor Pathway Preserves Schwann Cell Integrity in Diabetic Mice

Aortic connexin43 is decreased during hypertension induced by inhibition of nitric oxide synthase

Hypoxia induces severe right ventricular dilatation and infarction in heme oxygenase-1 null mice

Endotoxin-induced mortality is related to increased oxidative stress and end-organ dysfunction, not refractory hypotension, in heme oxygenase-1-deficient mice

Hypoxia induces severe right ventricular dilatation and infarction in heme oxygenase-1 null mice

Down-regulation of High Mobility Group-I(Y) Protein Contributes to the Inhibition of Nitric-oxide Synthase 2 by Transforming Growth Factor-β1

Induction of heme oxygenase-1 during endotoxemia is downregulated by transforming growth factor-β1

Role of activating protein‐1 and high mobility group‐I(Y) protein in the induction of CD44 gene expression by interleukin‐1β in vascular smooth muscle cells

Induction of Heme Oxygenase-1 During Endotoxemia Is Downregulated by Transforming Growth Factor-beta 1

High Mobility Group-I(Y) Protein Facilitates Nuclear Factor-κB Binding and Transactivation of the Inducible Nitric-oxide Synthase Promoter/Enhancer

Exacerbation of chronic renovascular hypertension adn acute renal failure in heme oxygenase-1-deficient mice

Induction of heme oxygenase-1 during endotoxemia is downregulated by transforming growth (factor-beta 1)

Spliceosomal peptide P140 for immunotherapy of systemic lupus erythematosus: Results of an early phase II clinical trial

Down-regulation of High Mobility Group-I(Y) Protein Contributes to the Inhibition of Nitric-oxide Synthase 2 by Transforming Growth Factor-Î²1

Time Course Changes of the Mechanical Properties of the Carotid Artery in Renal Hypertensive Rats

Transforming Growth Factor-Î²1 Inhibition of Macrophage Activation Is Mediated via Smad3

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