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Ubiquitination of serine, threonine, or lysine residues on the cytoplasmic tail can induce ERAD of MHC-I by viral E3 ligase mK3

The Journal of cell biology, 2007-05, Vol.177 (4), p.613-624

2007

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TRC8 E3 ligase ubiquitinates MHC class I molecules before dislocation from the ER

The Journal of cell biology, 2009-09, Vol.186 (5), p.685-692

2009

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Ube2j2 ubiquitinates hydroxylated amino acids on ER-associated degradation substrates

The Journal of cell biology, 2009-11, Vol.187 (5), p.655-668

2009

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The Human Cytomegalovirus US11 Gene Product Dislocates MHC Class I Heavy Chains from the Endoplasmic Reticulum to the Cytosol

Cell, 1996-03, Vol.84 (5), p.769-779

1996

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Viral immune evasion: Lessons in MHC class I antigen presentation

Seminars in immunology, 2015-03, Vol.27 (2), p.125-137

2015

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A high-coverage shRNA screen identifies TMEM129 as an E3 ligase involved in ER-associated protein degradation

Nature communications, 2014-05, Vol.5 (1), p.3832, Article 3832

2014

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Viral inhibition of the transporter associated with antigen processing (TAP): a striking example of functional convergent evolution

PLoS pathogens, 2015-04, Vol.11 (4), p.e1004743-e1004743

2015

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Host shutoff during productive Epstein-Barr virus infection is mediated by BGLF5 and may contribute to immune evasion

Proceedings of the National Academy of Sciences - PNAS, 2007-02, Vol.104 (9), p.3366-3371

2007

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Herpesviruses and immunity: The art of evasion

Veterinary microbiology, 2010-06, Vol.143 (1), p.89-100

2010

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A Flow Cytometry-Based Approach to Unravel Viral Interference with the MHC Class I Antigen Processing and Presentation Pathway

Methods in molecular biology (Clifton, N.J.), 2019-01, Vol.1988, p.187

2019

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The Epstein-Barr virus G-protein-coupled receptor contributes to immune evasion by targeting MHC class I molecules for degradation

PLoS pathogens, 2009-01, Vol.5 (1), p.e1000255-e1000255

2009

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Epstein-Barr virus evasion of CD8+ and CD4+ T cell immunity via concerted actions of multiple gene products

Seminars in cancer biology, 2008-12, Vol.18 (6), p.397-408

2008

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Quality Control of ER Membrane Proteins by the RNF185/Membralin Ubiquitin Ligase Complex

Molecular cell, 2020-10, Vol.80 (2), p.374-375

2020

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Exploiting the exploiter: a viral inhibitor stabilizes TAP for cryo-EM

Nature structural & molecular biology, 2016-02, Vol.23 (2), p.95-97

2016

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The Epstein-Barr Virus Glycoprotein gp150 Forms an Immune-Evasive Glycan Shield at the Surface of Infected Cells

PLoS pathogens, 2016-04, Vol.12 (4), p.e1005550-e1005550

2016

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Stage-specific inhibition of MHC class I presentation by the Epstein-Barr virus BNLF2a protein during virus lytic cycle

PLoS pathogens, 2009-06, Vol.5 (6), p.e1000490-e1000490

2009

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A Broad-Spectrum Antiviral Peptide Blocks Infection of Viruses by Binding to Phosphatidylserine in the Viral Envelope

Cells (Basel, Switzerland), 2020-08, Vol.9 (9), p.1989

2020

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Ectromelia-encoded virulence factor C15 specifically inhibits antigen presentation to CD4+ T cells post peptide loading

PLoS pathogens, 2020-08, Vol.16 (8), p.e1008685-e1008685

2020

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In cis inhibition of antigen processing by the latency-associated nuclear antigen I of Kaposi sarcoma Herpes virus

Molecular Immunology, 2007-02, Vol.44 (6), p.1352-1360

2007

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Varicellovirus UL49.5 proteins differentially affect the function of the transporter associated with antigen processing, TAP

PLoS pathogens, 2008-05, Vol.4 (5), p.e1000080-e1000080

2008

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Signaling of a Varicelloviral Factor across the Endoplasmic Reticulum Membrane Induces Destruction of the Peptide-loading Complex and Immune Evasion

The Journal of biological chemistry, 2008-05, Vol.283 (19), p.13428-13436

2008

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Induction of protective CTL immunity against peptide transporter TAP-deficient tumors through dendritic cell vaccination

Cancer research (Chicago, Ill.), 2007-09, Vol.67 (18), p.8450-8455

2007

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The ER-Luminal Domain of the HCMV Glycoprotein US6 Inhibits Peptide Translocation by TAP

Immunity (Cambridge, Mass.), 1997-05, Vol.6 (5), p.613-621

1997

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CRISPR/Cas9-Mediated Genome Editing of Herpesviruses Limits Productive and Latent Infections

PLoS pathogens, 2016-06, Vol.12 (6), p.e1005701-e1005701

2016

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Manipulation of the immune response by Epstein–Barr virus and Kaposi's sarcoma-associated herpesvirus: Consequences for tumor development

Seminars in cancer biology, 2008-12, Vol.18 (6), p.379-380

2008

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Ubiquitination of serine, threonine, or lysine residues on the cytoplasmic tail can induce ERAD of MHC-I by viral E3 ligase mK3

TRC8 E3 ligase ubiquitinates MHC class I molecules before dislocation from the ER

Ube2j2 ubiquitinates hydroxylated amino acids on ER-associated degradation substrates

The Human Cytomegalovirus US11 Gene Product Dislocates MHC Class I Heavy Chains from the Endoplasmic Reticulum to the Cytosol

Viral immune evasion: Lessons in MHC class I antigen presentation

A high-coverage shRNA screen identifies TMEM129 as an E3 ligase involved in ER-associated protein degradation

Viral inhibition of the transporter associated with antigen processing (TAP): a striking example of functional convergent evolution

Host shutoff during productive Epstein-Barr virus infection is mediated by BGLF5 and may contribute to immune evasion

Herpesviruses and immunity: The art of evasion

A Flow Cytometry-Based Approach to Unravel Viral Interference with the MHC Class I Antigen Processing and Presentation Pathway

The Epstein-Barr virus G-protein-coupled receptor contributes to immune evasion by targeting MHC class I molecules for degradation

Epstein-Barr virus evasion of CD8+ and CD4+ T cell immunity via concerted actions of multiple gene products

Quality Control of ER Membrane Proteins by the RNF185/Membralin Ubiquitin Ligase Complex

Exploiting the exploiter: a viral inhibitor stabilizes TAP for cryo-EM

The Epstein-Barr Virus Glycoprotein gp150 Forms an Immune-Evasive Glycan Shield at the Surface of Infected Cells

Stage-specific inhibition of MHC class I presentation by the Epstein-Barr virus BNLF2a protein during virus lytic cycle

A Broad-Spectrum Antiviral Peptide Blocks Infection of Viruses by Binding to Phosphatidylserine in the Viral Envelope

Ectromelia-encoded virulence factor C15 specifically inhibits antigen presentation to CD4+ T cells post peptide loading

In cis inhibition of antigen processing by the latency-associated nuclear antigen I of Kaposi sarcoma Herpes virus

Varicellovirus UL49.5 proteins differentially affect the function of the transporter associated with antigen processing, TAP

Signaling of a Varicelloviral Factor across the Endoplasmic Reticulum Membrane Induces Destruction of the Peptide-loading Complex and Immune Evasion

Induction of protective CTL immunity against peptide transporter TAP-deficient tumors through dendritic cell vaccination

The ER-Luminal Domain of the HCMV Glycoprotein US6 Inhibits Peptide Translocation by TAP

CRISPR/Cas9-Mediated Genome Editing of Herpesviruses Limits Productive and Latent Infections

Manipulation of the immune response by Epstein–Barr virus and Kaposi's sarcoma-associated herpesvirus: Consequences for tumor development

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