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Glutaminase is essential for the growth of triple-negative breast cancer cells with a deregulated glutamine metabolism pathway and its suppression synergizes with mTOR inhibition

PloS one, 2017-09, Vol.12 (9), p.e0185092-e0185092

2017

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Selective VPS34 inhibitor blocks autophagy and uncovers a role for NCOA4 in ferritin degradation and iron homeostasis in vivo

Nature cell biology, 2014-11, Vol.16 (11), p.1069-1079

2014

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Extreme Vulnerability of IDH1 Mutant Cancers to NAD+ Depletion

Cancer cell, 2015-12, Vol.28 (6), p.773-784

2015

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PTEN-deficient cancers depend on PIK3CB

Proceedings of the National Academy of Sciences - PNAS, 2008-09, Vol.105 (35), p.13057-13062

2008

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Isatuximab Acts Through Fc-Dependent, Independent, and Direct Pathways to Kill Multiple Myeloma Cells

Frontiers in immunology, 2020-08, Vol.11, p.1771-1771

2020

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Single-vector inducible lentiviral RNAi system for oncology target validation

Cell cycle (Georgetown, Tex.), 2009-02, Vol.8 (3), p.498-504

2009

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Selective Inhibition of Mutant Isocitrate Dehydrogenase 1 (IDH1) via Disruption of a Metal Binding Network by an Allosteric Small Molecule

The Journal of biological chemistry, 2015-01, Vol.290 (2), p.762-774

2015

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Pan-TGFβ inhibition by SAR439459 relieves immunosuppression and improves antitumor efficacy of PD-1 blockade

Oncoimmunology, 2020-01, Vol.9 (1), p.1811605-1811605

2020

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Tumor-Specific Urinary Matrix Metalloproteinase Fingerprinting: Identification of High Molecular Weight Urinary Matrix Metalloproteinase Species

Clinical cancer research, 2008-10, Vol.14 (20), p.6610-6617

2008

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BND-22, a first-in-class humanized ILT2-blocking antibody, promotes antitumor immunity and tumor regression

Journal for immunotherapy of cancer, 2022-09, Vol.10 (9), p.e004859

2022

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An iron-dependent metabolic vulnerability underlies VPS34-dependence in RKO cancer cells

PloS one, 2020-08, Vol.15 (8), p.e0235551-e0235551

2020

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RING domain-mediated interaction is a requirement for MDM2's E3 ligase activity

Cancer research (Chicago, Ill.), 2007-07, Vol.67 (13), p.6026-6030

2007

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Identification of the endosomal sorting complex required for transport-I (ESCRT-I) as an important modulator of anti-miR uptake by cancer cells

Nucleic acids research, 2015-01, Vol.43 (2), p.1204-1215

2015

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An experimental model of anti-PD-1 resistance exhibits activation of TGFß and Notch pathways and is sensitive to local mRNA immunotherapy

Oncoimmunology, 2021-01, Vol.10 (1), p.1881268-1881268

2021

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The Polycomb Group Protein Bmi-1 Is Essential for the Growth of Multiple Myeloma Cells

Cancer research (Chicago, Ill.), 2010-07, Vol.70 (13), p.5528-5538

2010

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Matrix Metalloproteinase-2 Is Required for the Switch to the Angiogenic Phenotype in a Tumor Model

Proceedings of the National Academy of Sciences - PNAS, 2000-04, Vol.97 (8), p.3884-3889

2000

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Critical Contribution of the MDM2 Acidic Domain to p53 Ubiquitination

Molecular and Cellular Biology, 2003-07, Vol.23 (14), p.4939-4947

2003

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DNA Damage-induced MDMX Degradation Is Mediated by MDM2

The Journal of biological chemistry, 2003-11, Vol.278 (46), p.45946-45953

2003

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Contribution of Polycomb Homologues Bmi-1 and Mel-18 to Medulloblastoma Pathogenesis

Molecular and Cellular Biology, 2007-07, Vol.27 (13), p.4968-4979

2007

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Mutual Dependence of MDM2 and MDMX in Their Functional Inactivation of p53

The Journal of biological chemistry, 2002-05, Vol.277 (22), p.19251-19254

2002

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Combination of PIM and JAK2 inhibitors synergistically suppresses cell proliferation and overcomes drug resistance of myeloproliferative neoplasms

Oncotarget, 2014-05, Vol.5 (10), p.3362-3374

2014

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Glutaminase is essential for the growth of triple-negative breast cancer cells with a deregulated glutamine metabolism pathway and its suppression synergizes with mTOR inhibition

Selective VPS34 inhibitor blocks autophagy and uncovers a role for NCOA4 in ferritin degradation and iron homeostasis in vivo

Extreme Vulnerability of IDH1 Mutant Cancers to NAD+ Depletion

PTEN-deficient cancers depend on PIK3CB

Isatuximab Acts Through Fc-Dependent, Independent, and Direct Pathways to Kill Multiple Myeloma Cells

Single-vector inducible lentiviral RNAi system for oncology target validation

Selective Inhibition of Mutant Isocitrate Dehydrogenase 1 (IDH1) via Disruption of a Metal Binding Network by an Allosteric Small Molecule

Selective blockade of the hydrolysis of the endocannabinoid 2-arachidonoylglycerol impairs learning and memory performance while producing antinociceptive activity in rodents

Pan-TGFβ inhibition by SAR439459 relieves immunosuppression and improves antitumor efficacy of PD-1 blockade

Tumor-Specific Urinary Matrix Metalloproteinase Fingerprinting: Identification of High Molecular Weight Urinary Matrix Metalloproteinase Species

BND-22, a first-in-class humanized ILT2-blocking antibody, promotes antitumor immunity and tumor regression

An iron-dependent metabolic vulnerability underlies VPS34-dependence in RKO cancer cells

RING domain-mediated interaction is a requirement for MDM2's E3 ligase activity

Differential expression of CCR8 in tumors versus normal tissue allows specific depletion of tumor-infiltrating T regulatory cells by GS-1811, a novel Fc-optimized anti-CCR8 antibody

Identification of the endosomal sorting complex required for transport-I (ESCRT-I) as an important modulator of anti-miR uptake by cancer cells

An experimental model of anti-PD-1 resistance exhibits activation of TGFß and Notch pathways and is sensitive to local mRNA immunotherapy

Local delivery of mRNA-encoded cytokines promotes antitumor immunity and tumor eradication across multiple preclinical tumor models

The Polycomb Group Protein Bmi-1 Is Essential for the Growth of Multiple Myeloma Cells

Matrix Metalloproteinase-2 Is Required for the Switch to the Angiogenic Phenotype in a Tumor Model

Critical Contribution of the MDM2 Acidic Domain to p53 Ubiquitination

DNA Damage-induced MDMX Degradation Is Mediated by MDM2

Pre-clinical development of a novel CD3-CD123 bispecific T-cell engager using cross-over dual-variable domain (CODV) format for acute myeloid leukemia (AML) treatment

Contribution of Polycomb Homologues Bmi-1 and Mel-18 to Medulloblastoma Pathogenesis

Mutual Dependence of MDM2 and MDMX in Their Functional Inactivation of p53

Combination of PIM and JAK2 inhibitors synergistically suppresses cell proliferation and overcomes drug resistance of myeloproliferative neoplasms

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