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Inhibiting the transcription factor HSF1 as an anticancer strategy

Expert opinion on therapeutic targets, 2009-04, Vol.13 (4), p.469-478

2009

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Fitness trade-offs restrict the evolution of resistance to amphotericin B

PLoS biology, 2013-10, Vol.11 (10), p.e1001692

2013

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Heat Shock Factor 1 Is a Powerful Multifaceted Modifier of Carcinogenesis

Cell, 2007-09, Vol.130 (6), p.1005-1018

2007

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The Reprogramming of Tumor Stroma by HSF1 Is a Potent Enabler of Malignancy

Cell, 2014-07, Vol.158 (3), p.564-578

2014

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Methods to validate Hsp90 inhibitor specificity, to identify off-target effects, and to rethink approaches for further clinical development

Cell stress & chaperones, 2018-07, Vol.23 (4), p.467-482

2018

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HSF1 phase transition mediates stress adaptation and cell fate decisions

Nature cell biology, 2020-02, Vol.22 (2), p.151-158

2020

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Widespread macromolecular interaction perturbations in human genetic disorders

Cell, 2015-04, Vol.161 (3), p.647-660

2015

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Altered Hsp90 function in cancer: A unique therapeutic opportunity

Molecular cancer therapeutics, 2004-08, Vol.3 (8), p.1021-1030

2004

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HSP90 empowers evolution of resistance to hormonal therapy in human breast cancer models

Proceedings of the National Academy of Sciences - PNAS, 2014-12, Vol.111 (51), p.18297-18302

2014

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A small molecule produced by Lactobacillus species blocks Candida albicans filamentation by inhibiting a DYRK1-family kinase

Nature communications, 2021-10, Vol.12 (1), p.6151-16, Article 6151

2021

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Chaperones as thermodynamic sensors of drug-target interactions reveal kinase inhibitor specificities in living cells

Nature biotechnology, 2013-07, Vol.31 (7), p.630-637

2013

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Tight Coordination of Protein Translation and HSF1 Activation Supports the Anabolic Malignant State

Science (American Association for the Advancement of Science), 2013-07, Vol.341 (6143), p.250-250

2013

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High levels of nuclear heat-shock factor 1 (HSF1) are associated with poor prognosis in breast cancer

Proceedings of the National Academy of Sciences - PNAS, 2011-11, Vol.108 (45), p.18378-18383

2011

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Rieske head domain dynamics and indazole-derivative inhibition of Candida albicans complex III

Structure (London), 2022-01, Vol.30 (1), p.129-138.e4

2022

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Structural basis for species-selective targeting of Hsp90 in a pathogenic fungus

Nature communications, 2019-01, Vol.10 (1), p.402-402, Article 402

2019

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Loss of tumor suppressor NF1 activates HSF1 to promote carcinogenesis

The Journal of clinical investigation, 2012-10, Vol.122 (10), p.3742-3754

2012

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Reply to: Lactobacillus species do not produce 1-acetyl-β-carboline

Nature communications, 2024-08, Vol.15 (1), p.6441-2, Article 6441

2024

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Multifactor transcriptional control of alternative oxidase induction integrates diverse environmental inputs to enable fungal virulence

Nature communications, 2023-07, Vol.14 (1), p.4528-4528, Article 4528

2023

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Overcoming Fungal Echinocandin Resistance through Inhibition of the Non-essential Stress Kinase Yck2

Cell chemical biology, 2020-03, Vol.27 (3), p.269-282.e5

2020

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Harnessing Hsp90 function as a powerful, broadly effective therapeutic strategy for fungal infectious disease

Proceedings of the National Academy of Sciences - PNAS, 2009-02, Vol.106 (8), p.2818-2823

2009

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DYRK-family kinases regulate Candida albicans morphogenesis and virulence through the Ras1/PKA pathway

mBio, 2023-12, Vol.14 (6), p.e0218323

2023

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Targeting fungal membrane homeostasis with imidazopyrazoindoles impairs azole resistance and biofilm formation

Nature communications, 2022-06, Vol.13 (1), p.3634-3634, Article 3634

2022

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An oxindole efflux inhibitor potentiates azoles and impairs virulence in the fungal pathogen Candida auris

Nature communications, 2020-12, Vol.11 (1), p.6429-17, Article 6429

2020

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Calcineurin determines toxic versus beneficial responses to α-synuclein

Proceedings of the National Academy of Sciences - PNAS, 2014-08, Vol.111 (34), p.E3544-E3552

2014

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Translation Inhibition by Rocaglates Activates a Species-Specific Cell Death Program in the Emerging Fungal Pathogen Candida auris

mBio, 2020-03, Vol.11 (2)

2020

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(23)
Hsp90 Heat-Shock Proteins - Antagonists & Inhibitors
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Inhibiting the transcription factor HSF1 as an anticancer strategy

Fitness trade-offs restrict the evolution of resistance to amphotericin B

Heat Shock Factor 1 Is a Powerful Multifaceted Modifier of Carcinogenesis

The Reprogramming of Tumor Stroma by HSF1 Is a Potent Enabler of Malignancy

Methods to validate Hsp90 inhibitor specificity, to identify off-target effects, and to rethink approaches for further clinical development

HSF1 phase transition mediates stress adaptation and cell fate decisions

Widespread macromolecular interaction perturbations in human genetic disorders

Altered Hsp90 function in cancer: A unique therapeutic opportunity

HSP90 empowers evolution of resistance to hormonal therapy in human breast cancer models

A small molecule produced by Lactobacillus species blocks Candida albicans filamentation by inhibiting a DYRK1-family kinase

Chaperones as thermodynamic sensors of drug-target interactions reveal kinase inhibitor specificities in living cells

Tight Coordination of Protein Translation and HSF1 Activation Supports the Anabolic Malignant State

High levels of nuclear heat-shock factor 1 (HSF1) are associated with poor prognosis in breast cancer

Rieske head domain dynamics and indazole-derivative inhibition of Candida albicans complex III

Structural basis for species-selective targeting of Hsp90 in a pathogenic fungus

Loss of tumor suppressor NF1 activates HSF1 to promote carcinogenesis

Reply to: Lactobacillus species do not produce 1-acetyl-β-carboline

Multifactor transcriptional control of alternative oxidase induction integrates diverse environmental inputs to enable fungal virulence

Overcoming Fungal Echinocandin Resistance through Inhibition of the Non-essential Stress Kinase Yck2

Harnessing Hsp90 function as a powerful, broadly effective therapeutic strategy for fungal infectious disease

DYRK-family kinases regulate Candida albicans morphogenesis and virulence through the Ras1/PKA pathway

Targeting fungal membrane homeostasis with imidazopyrazoindoles impairs azole resistance and biofilm formation

An oxindole efflux inhibitor potentiates azoles and impairs virulence in the fungal pathogen Candida auris

Calcineurin determines toxic versus beneficial responses to α-synuclein

Translation Inhibition by Rocaglates Activates a Species-Specific Cell Death Program in the Emerging Fungal Pathogen Candida auris

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