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The application of BH3 mimetics in myeloid leukemias

Cell death & disease, 2021-02, Vol.12 (2), p.222-13, Article 222

2021

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Using topic modeling to detect cellular crosstalk in scRNA-seq

PLoS computational biology, 2022-04, Vol.18 (4), p.e1009975-e1009975

2022

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CD93 is expressed on chronic myeloid leukemia stem cells and identifies a quiescent population which persists after tyrosine kinase inhibitor therapy

Leukemia, 2020-06, Vol.34 (6), p.1613-1625

2020

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Overcoming BCR::ABL1 dependent and independent survival mechanisms in chronic myeloid leukaemia using a multi-kinase targeting approach

Cell communication and signaling, 2023-11, Vol.21 (1), p.342-342, Article 342

2023

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PB2475: SECRETED FACTORS FROM BONE MARROW‐DERIVED FIBROBLASTS PROTECT AML CELLS AGAINST APOPTOSIS MEDIATED BY A NOVEL CDK/MCL‐1 INHIBITOR VIA STAT3/MCL‐1 SIGNALLING

HemaSphere, 2023-08, Vol.7 (S3), p.e57461cc-n/a

2023

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Micro‐environment alterations through time leading to myeloid malignancies

British journal of pharmacology, 2024-01, Vol.181 (2), p.283-294

2024

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Regulation of Embryonic Stem Cell Self-renewal by Phosphoinositide 3-Kinase-dependent Signaling

The Journal of biological chemistry, 2004-11, Vol.279 (46), p.48063-48070

2004

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Bioengineered niches that recreate physiological extracellular matrix organisation to support long-term haematopoietic stem cells

Nature communications, 2024-07, Vol.15 (1), p.5791-18, Article 5791

2024

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Macrophages in Acute Myeloid Leukaemia: Significant Players in Therapy Resistance and Patient Outcomes

Frontiers in cell and developmental biology, 2021-06, Vol.9, p.692800-692800

2021

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Leukaemia exposure alters the transcriptional profile and function of BCR::ABL1 negative macrophages in the bone marrow niche

Nature communications, 2024-02, Vol.15 (1), p.1090-1090, Article 1090

2024

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Canonical Wnt signaling promotes early hematopoietic progenitor formation and erythroid specification during embryonic stem cell differentiation

PloS one, 2013-11, Vol.8 (11), p.e81030

2013

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P433: PHARMACOLOGICAL DUAL INHIBITION OF PARP AND TIP60 AS A PROSPECTIVE THERAPEUTIC REGIMEN INDUCES SYNERGISTIC AML CELL DEATH

HemaSphere, 2023-08, Vol.7 (S3), p.e20033ee-n/a

2023

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Magnetically levitated mesenchymal stem cell spheroids cultured with a collagen gel maintain phenotype and quiescence

Journal of tissue engineering, 2017, Vol.8, p.2041731417704428

2017

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BH3 mimetics in combination with nilotinib or ponatinib represent a promising therapeutic strategy in blast phase chronic myeloid leukemia

Cell death discovery, 2022-11, Vol.8 (1), p.457-457, Article 457

2022

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P1350: M2‐LIKE MONOCYTE‐DERIVED MACROPHAGES PROTECT AML CELL LINES AND PRIMARY AML CELLS AGAINST THERAPY‐INDUCED APOPTOSIS

HemaSphere, 2023-08, Vol.7 (S3), p.e92103ee-n/a

2023

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Interrogation of novel CDK2/9 inhibitor fadraciclib (CYC065) as a potential therapeutic approach for AML

Cell death discovery, 2021-06, Vol.7 (1), p.137-137, Article 137

2021

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Bone marrow niche crosses paths with BMPs: a road to protection and persistence in CML

Biochemical Society transactions, 2019-10, Vol.47 (5), p.1307-1325

2019

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Acute Loss of Cited2 Impairs Nanog Expression and Decreases Self‐Renewal of Mouse Embryonic Stem Cells

Stem cells (Dayton, Ohio), 2015-03, Vol.33 (3), p.699-712

2015

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Targeting self-renewal pathways in myeloid malignancies

Cell communication and signaling, 2013-05, Vol.11 (1), p.33-33, Article 33

2013

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Complexities of modeling the bone marrow microenvironment to facilitate hematopoietic research

Experimental hematology, 2024-07, Vol.135, p.104233, Article 104233

2024

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Correction to: Interrogation of novel CDK2/9 inhibitor fadraciclib (CYC065) as a potential therapeutic approach for AML

Cell death discovery, 2021-07, Vol.7 (1), p.171-171, Article 171

2021

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Role of the bone morphogenic protein pathway in developmental haemopoiesis and leukaemogenesis

Biochemical Society transactions, 2016-10, Vol.44 (5), p.1455-1463

2016

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Patterns of Wnt/Fzd/LRP gene expression during embryonic hematopoiesis

Stem cells and development, 2009-06, Vol.18 (5), p.759-772

2009

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Synergistic Leukaemia Cell Death Induced with PARP and TiP60 Inhibitors Is a Prospective Therapeutic Strategy for AML

Blood, 2022-11, Vol.140 (Supplement 1), p.13001-13002

2022

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NFATC2 regulates Targets of MYC Signaling in MLL-AF9 AML

Blood, 2021-11, Vol.138 (Supplement 1), p.3301-3301

2021

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The application of BH3 mimetics in myeloid leukemias

Using topic modeling to detect cellular crosstalk in scRNA-seq

CD93 is expressed on chronic myeloid leukemia stem cells and identifies a quiescent population which persists after tyrosine kinase inhibitor therapy

Overcoming BCR::ABL1 dependent and independent survival mechanisms in chronic myeloid leukaemia using a multi-kinase targeting approach

PB2475: SECRETED FACTORS FROM BONE MARROW‐DERIVED FIBROBLASTS PROTECT AML CELLS AGAINST APOPTOSIS MEDIATED BY A NOVEL CDK/MCL‐1 INHIBITOR VIA STAT3/MCL‐1 SIGNALLING

Micro‐environment alterations through time leading to myeloid malignancies

Regulation of Embryonic Stem Cell Self-renewal by Phosphoinositide 3-Kinase-dependent Signaling

Bioengineered niches that recreate physiological extracellular matrix organisation to support long-term haematopoietic stem cells

Macrophages in Acute Myeloid Leukaemia: Significant Players in Therapy Resistance and Patient Outcomes

Leukaemia exposure alters the transcriptional profile and function of BCR::ABL1 negative macrophages in the bone marrow niche

Canonical Wnt signaling promotes early hematopoietic progenitor formation and erythroid specification during embryonic stem cell differentiation

P433: PHARMACOLOGICAL DUAL INHIBITION OF PARP AND TIP60 AS A PROSPECTIVE THERAPEUTIC REGIMEN INDUCES SYNERGISTIC AML CELL DEATH

Magnetically levitated mesenchymal stem cell spheroids cultured with a collagen gel maintain phenotype and quiescence

BH3 mimetics in combination with nilotinib or ponatinib represent a promising therapeutic strategy in blast phase chronic myeloid leukemia

P1350: M2‐LIKE MONOCYTE‐DERIVED MACROPHAGES PROTECT AML CELL LINES AND PRIMARY AML CELLS AGAINST THERAPY‐INDUCED APOPTOSIS

Interrogation of novel CDK2/9 inhibitor fadraciclib (CYC065) as a potential therapeutic approach for AML

Bone marrow niche crosses paths with BMPs: a road to protection and persistence in CML

Acute Loss of Cited2 Impairs Nanog Expression and Decreases Self‐Renewal of Mouse Embryonic Stem Cells

Targeting self-renewal pathways in myeloid malignancies

Complexities of modeling the bone marrow microenvironment to facilitate hematopoietic research

Correction to: Interrogation of novel CDK2/9 inhibitor fadraciclib (CYC065) as a potential therapeutic approach for AML

Role of the bone morphogenic protein pathway in developmental haemopoiesis and leukaemogenesis

Patterns of Wnt/Fzd/LRP gene expression during embryonic hematopoiesis

Synergistic Leukaemia Cell Death Induced with PARP and TiP60 Inhibitors Is a Prospective Therapeutic Strategy for AML

NFATC2 regulates Targets of MYC Signaling in MLL-AF9 AML

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