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High CO2 Levels Cause Skeletal Muscle Atrophy via AMP-activated Kinase (AMPK), FoxO3a Protein, and Muscle-specific Ring Finger Protein 1 (MuRF1)

The Journal of biological chemistry, 2015-04, Vol.290 (14), p.9183-9194

2015

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Hypercapnia increases airway smooth muscle contractility via caspase-7-mediated miR-133a-RhoA signaling

Science translational medicine, 2018-09, Vol.10 (457)

2018

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Hypercapnia Regulates Gene Expression and Tissue Function

Frontiers in physiology, 2020-11, Vol.11, p.598122-598122

2020

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Hypercapnia impairs lung neutrophil function and increases mortality in murine pseudomonas pneumonia

American journal of respiratory cell and molecular biology, 2013-11, Vol.49 (5), p.821-828

2013

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High CO2 levels impair alveolar epithelial function independently of pH

PloS one, 2007-11, Vol.2 (11), p.e1238

2007

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Elevated CO2 Levels Delay Skeletal Muscle Repair by Increasing Fatty Acid Oxidation

Frontiers in physiology, 2021-01, Vol.11

2021

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Elevated CO2 regulates the Wnt signaling pathway in mammals, Drosophila melanogaster and Caenorhabditis elegans

Scientific reports, 2019-12, Vol.9 (1), p.1-8, Article 18251

2019

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Evolutionary conserved role of c-Jun-N-terminal kinase in CO2-induced epithelial dysfunction

PloS one, 2012-10, Vol.7 (10), p.e46696

2012

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High CO2 Leads to Na,K-ATPase Endocytosis via c-Jun Amino-Terminal Kinase-Induced LMO7b Phosphorylation

Molecular and cellular biology, 2015-12, Vol.35 (23), p.3962-3973

2015

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AMP-activated protein kinase regulates CO2-induced alveolar epithelial dysfunction in rats and human cells by promoting Na,K-ATPase endocytosis

The Journal of clinical investigation, 2008-02, Vol.118 (2), p.752-762

2008

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Cardiac glycosides decrease influenza virus replication by inhibiting cell protein translational machinery

American journal of physiology. Lung cellular and molecular physiology, 2019-06, Vol.316 (6), p.L1094-L1106

2019

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Phosphorylation and ubiquitination are necessary for Na,K-ATPase endocytosis during hypoxia

Cellular signalling, 2007-09, Vol.19 (9), p.1893-1898

2007

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Myosin-Va restrains the trafficking of Na⁺/K⁺-ATPase-containing vesicles in alveolar epithelial cells

Journal of cell science, 2009-11, Vol.122 (21), p.3915-3922

2009

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Carbonic Anhydrase II and Alveolar Fluid Reabsorption during Hypercapnia

American journal of respiratory cell and molecular biology, 2008-01, Vol.38 (1), p.32-37

2008

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High CO2 Levels Impair Lung Wound Healing

American journal of respiratory cell and molecular biology, 2020-08, Vol.63 (2), p.244-254

2020

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Lung Injury Induces Alveolar Type 2 Cell Hypertrophy and Polyploidy with Implications for Repair and Regeneration

American journal of respiratory cell and molecular biology, 2022-05, Vol.66 (5), p.564-576

2022

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Hypercapnia Suppresses the HIF-dependent Adaptive Response to Hypoxia

The Journal of biological chemistry, 2016-05, Vol.291 (22), p.11800-11808

2016

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Hypoxia Leads to Na,K-ATPase Downregulation via Ca2+ Release-Activated Ca2+ Channels and AMPK Activation

Molecular and Cellular Biology, 2011-09, Vol.31 (17), p.3546-3556

2011

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Hypoxia leads to Na,K-ATPase downregulation via Ca(2+) release-activated Ca(2+) channels and AMPK activation

Molecular and cellular biology, 2011-09, Vol.31 (17), p.3546-3556

2011

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Influenza A Virus Infection Induces Muscle Wasting via IL-6 Regulation of the E3 Ubiquitin Ligase Atrogin-1

The Journal of immunology (1950), 2019-01, Vol.202 (2), p.484-493

2019

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HIF and HOIL-1L–mediated PKCζ degradation stabilizes plasma membrane Na,K-ATPase to protect against hypoxia-induced lung injury

Proceedings of the National Academy of Sciences - PNAS, 2017-11, Vol.114 (47), p.E10178-E10186

2017

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High CO 2 Levels Impair Lung Wound Healing

American journal of respiratory cell and molecular biology, 2020-08, Vol.63 (2), p.244-254

2020

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Extracellular signal-regulated kinase (ERK) participates in the hypercapnia-induced Na,K-ATPase downregulation

FEBS letters, 2010-09, Vol.584 (18), p.3985-3989

2010

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High CO2 Levels Cause Skeletal Muscle Atrophy via AMP-activated Kinase (AMPK), FoxO3a Protein, and Muscle-specific Ring Finger Protein 1 (MuRF1)

Hypercapnia increases airway smooth muscle contractility via caspase-7-mediated miR-133a-RhoA signaling

Hypercapnia Regulates Gene Expression and Tissue Function

Hypercapnia impairs lung neutrophil function and increases mortality in murine pseudomonas pneumonia

High CO2 levels impair alveolar epithelial function independently of pH

Elevated CO2 Levels Delay Skeletal Muscle Repair by Increasing Fatty Acid Oxidation

Elevated CO2 regulates the Wnt signaling pathway in mammals, Drosophila melanogaster and Caenorhabditis elegans

Evolutionary conserved role of c-Jun-N-terminal kinase in CO2-induced epithelial dysfunction

High CO2 Leads to Na,K-ATPase Endocytosis via c-Jun Amino-Terminal Kinase-Induced LMO7b Phosphorylation

AMP-activated protein kinase regulates CO2-induced alveolar epithelial dysfunction in rats and human cells by promoting Na,K-ATPase endocytosis

Hypercapnia alters stroma-derived Wnt production to limit β-catenin signaling and proliferation in AT2 cells

Cardiac glycosides decrease influenza virus replication by inhibiting cell protein translational machinery

Phosphorylation and ubiquitination are necessary for Na,K-ATPase endocytosis during hypoxia

Myosin-Va restrains the trafficking of Na⁺/K⁺-ATPase-containing vesicles in alveolar epithelial cells

Carbonic Anhydrase II and Alveolar Fluid Reabsorption during Hypercapnia

High CO2 Levels Impair Lung Wound Healing

Lung Injury Induces Alveolar Type 2 Cell Hypertrophy and Polyploidy with Implications for Repair and Regeneration

Hypercapnia Suppresses the HIF-dependent Adaptive Response to Hypoxia

Hypoxia Leads to Na,K-ATPase Downregulation via Ca2+ Release-Activated Ca2+ Channels and AMPK Activation

Hypoxia leads to Na,K-ATPase downregulation via Ca(2+) release-activated Ca(2+) channels and AMPK activation

Influenza A Virus Infection Induces Muscle Wasting via IL-6 Regulation of the E3 Ubiquitin Ligase Atrogin-1

HIF and HOIL-1L–mediated PKCζ degradation stabilizes plasma membrane Na,K-ATPase to protect against hypoxia-induced lung injury

Impaired phagocytic function in CX3CR1+ tissue‐resident skeletal muscle macrophages prevents muscle recovery after influenza A virus‐induced pneumonia in old mice

High CO 2 Levels Impair Lung Wound Healing

Extracellular signal-regulated kinase (ERK) participates in the hypercapnia-induced Na,K-ATPase downregulation

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