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γδ T Cells but Not NK Cells Are Essential for Cell-Mediated Immunity against Plasmodium chabaudi Malaria

Infection and Immunity, 2010-10, Vol.78 (10), p.4331-4340

2010

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Suppression of Plasmodium chabaudi Parasitemia Is Independent of the Action of Reactive Oxygen Intermediates and/or Nitric Oxide

Infection and Immunity, 2004-11, Vol.72 (11), p.6359-6366

2004

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Splenic γδ T Cells Regulated by CD4+ T Cells Are Required To Control Chronic Plasmodium chabaudi Malaria in the B-Cell-Deficient Mouse

Infection and Immunity, 2006-05, Vol.74 (5), p.2717-2725

2006

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Protection against Plasmodium chabaudi Malaria Induced by Immunization with Apical Membrane Antigen 1 and Merozoite Surface Protein 1 in the Absence of Gamma Interferon or Interleukin-4

Infection and Immunity, 2004-10, Vol.72 (10), p.5605-5612

2004

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Prediction of Merozoite Surface Protein 1 and Apical Membrane Antigen 1 Vaccine Efficacies against Plasmodium chabaudi Malaria Based on Prechallenge Antibody Responses

Clinical and Vaccine Immunology, 2009-03, Vol.16 (3), p.293-302

2009

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CD28 Costimulation Is Required for the Expression of T-Cell-Dependent Cell-Mediated Immunity against Blood-Stage Plasmodium chabaudi Malaria Parasites

Infection and Immunity, 2004-10, Vol.72 (10), p.5768-5774

2004

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Immunity to blood-stage murine malarial parasites is MHC class II dependent

Immunology letters, 2003-10, Vol.89 (2), p.243-249

2003

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Immunization against Plasmodium chabaudi malaria using combined formulations of apical membrane antigen-1 and merozoite surface protein-1

Vaccine, 2003-05, Vol.21 (17), p.1843-1852

2003

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In vitro antibody response to the tetrapeptide repeats of Plasmodium falciparum circumsporozoite protein by splenocytes from mice infected with P. yoelii yoelii

Journal of Veterinary Medical Science, 2001, Vol.63(7), pp.743-749

2001

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Analysis of antigen‐specific antibodies and their isotypes in experimental malaria

Cytometry. Part A, 2007-04, Vol.71A (4), p.242-250

2007

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γδ T cells function in cell-mediated immunity to malaria

Nature reviews. Immunology, 2004-08, Vol.4 (8), p.656-656

2004

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Plasmodium chabaudi adami: Use of the B-cell-deficient mouse to define possible mechanisms modulating parasitemia of chronic malaria

Experimental parasitology, 2005-10, Vol.111 (2), p.97-104

2005

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Immunity to Plasmodium chabaudi adami in the B-cell-deficient mouse

Nature (London), 1981-03, Vol.290 (5802), p.143-145

1981

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Plasticity of Immune Responses Suppressing Parasitemia During Acute Plasmodium chabaudi Malaria

The Journal of immunology (1950), 1999-06, Vol.162 (12), p.7383-7388

1999

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Plasmodium chabaudi adami: interferon-γ but not IL-2 is essential for the expression of cell-mediated immunity against blood-stage parasites in mice

Experimental parasitology, 2003-10, Vol.105 (2), p.159-166

2003

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The effects of interleukin-15 on human γδ T cell responses to Plasmodium falciparum in vitro

Immunology letters, 1998-12, Vol.64 (2), p.125-132

1998

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Participation of lymphocyte subpopulations in the pathogenesis of experimental murine cerebral malaria

The Journal of immunology (1950), 1996-08, Vol.157 (4), p.1620-1624

1996

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Production of interleukin 10 during malaria caused by lethal and nonlethal variants of Plasmodium yoelii yoelii

Parasitology research (1987), 1996-05, Vol.82 (5), p.385-391

1996

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T Cells but Not NK Cells Are Essential for Cell-Mediated Immunity against Plasmodium chabaudi Malaria

Infection and immunity, 2010-10, Vol.78 (10), p.4331-4340

2010

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gd T Cells but Not NK Cells Are Essential for Cell-Mediated Immunity against Plasmodium chabaudi Malaria

Infection and immunity, 2010-01, Vol.78 (10), p.4331-4340

2010

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Signalling through the IL‐2 receptor γc peptide (CD132) is essential for the expression of immunity to Plasmodium chabaudi adami blood‐stage malaria

Parasite immunology, 2011-09, Vol.33 (9), p.512-516

2011

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Expansion of the γδ T cell subset in vivo during bloodstage malaria in B cell‐deficient mice

Journal of leukocyte biology, 1996-08, Vol.60 (2), p.221-229

1996

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Splenic gammadelta T cells regulated by CD4+ T cells are required to control chronic Plasmodium chabaudi malaria in the B-cell-deficient mouse

Infection and immunity, 2006-05, Vol.74 (5), p.2717-2725

2006

Volltextzugriff (PDF)

Splenic gamma delta T Cells Regulated by CD4 super(+) T Cells Are Required To Control Chronic Plasmodium chabaudi Malaria in the B-Cell-Deficient Mouse

Infection and immunity, 2006-05, Vol.74 (5), p.2717-2725

2006

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Cell-mediated immunity to the asexual blood stages of malarial parasites: Animal models

Immunology letters, 1990-08, Vol.25 (1), p.87-95

1990

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γδ T Cells but Not NK Cells Are Essential for Cell-Mediated Immunity against Plasmodium chabaudi Malaria

Suppression of Plasmodium chabaudi Parasitemia Is Independent of the Action of Reactive Oxygen Intermediates and/or Nitric Oxide

Splenic γδ T Cells Regulated by CD4+ T Cells Are Required To Control Chronic Plasmodium chabaudi Malaria in the B-Cell-Deficient Mouse

Protection against Plasmodium chabaudi Malaria Induced by Immunization with Apical Membrane Antigen 1 and Merozoite Surface Protein 1 in the Absence of Gamma Interferon or Interleukin-4

Prediction of Merozoite Surface Protein 1 and Apical Membrane Antigen 1 Vaccine Efficacies against Plasmodium chabaudi Malaria Based on Prechallenge Antibody Responses

CD28 Costimulation Is Required for the Expression of T-Cell-Dependent Cell-Mediated Immunity against Blood-Stage Plasmodium chabaudi Malaria Parasites

Immunity to blood-stage murine malarial parasites is MHC class II dependent

Immunization against Plasmodium chabaudi malaria using combined formulations of apical membrane antigen-1 and merozoite surface protein-1

In vitro antibody response to the tetrapeptide repeats of Plasmodium falciparum circumsporozoite protein by splenocytes from mice infected with P. yoelii yoelii

Analysis of antigen‐specific antibodies and their isotypes in experimental malaria

γδ T cells function in cell-mediated immunity to malaria

Plasmodium chabaudi adami: Use of the B-cell-deficient mouse to define possible mechanisms modulating parasitemia of chronic malaria

Immunity to Plasmodium chabaudi adami in the B-cell-deficient mouse

Plasticity of Immune Responses Suppressing Parasitemia During Acute Plasmodium chabaudi Malaria

Plasmodium chabaudi adami: interferon-γ but not IL-2 is essential for the expression of cell-mediated immunity against blood-stage parasites in mice

The effects of interleukin-15 on human γδ T cell responses to Plasmodium falciparum in vitro

Participation of lymphocyte subpopulations in the pathogenesis of experimental murine cerebral malaria

Production of interleukin 10 during malaria caused by lethal and nonlethal variants of Plasmodium yoelii yoelii

T Cells but Not NK Cells Are Essential for Cell-Mediated Immunity against Plasmodium chabaudi Malaria

gd T Cells but Not NK Cells Are Essential for Cell-Mediated Immunity against Plasmodium chabaudi Malaria

Signalling through the IL‐2 receptor γc peptide (CD132) is essential for the expression of immunity to Plasmodium chabaudi adami blood‐stage malaria

Expansion of the γδ T cell subset in vivo during bloodstage malaria in B cell‐deficient mice

Splenic gammadelta T cells regulated by CD4+ T cells are required to control chronic Plasmodium chabaudi malaria in the B-cell-deficient mouse

Splenic gamma delta T Cells Regulated by CD4 super(+) T Cells Are Required To Control Chronic Plasmodium chabaudi Malaria in the B-Cell-Deficient Mouse

Cell-mediated immunity to the asexual blood stages of malarial parasites: Animal models

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