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IL‐37 requires IL‐18Rα and SIGIRR/IL‐1R8 to diminish allergic airway inflammation in mice

Allergy (Copenhagen), 2015-04, Vol.70 (4), p.366-373

2015

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NanoCOLT-Modulatory effects of modified carbon black nanoparticles in a mouse model of allergic asthma

Pneumologie (Stuttgart, Germany), 2016, Vol.70 (7)

2016

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D33 NEW BASIC SCIENCE IN ASTHMA: ALLERGIC INFLAMMATION II: Specific Sirna Targeting Ror[gamma]t Inhibits Airway Inflammation And Airway Hyperresponsiveness In A Mouse Model Of Neutrophilic Asthma

American journal of respiratory and critical care medicine, 2015-01, Vol.191, p.1

2015

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D33 NEW BASIC SCIENCE IN ASTHMA: ALLERGIC INFLAMMATION II: The [alpha]-Melanocyte Stimulating Hormone Is Produced In Allergic Asthma And Counterbalances Allergic Airway Inflammation In Mice

American journal of respiratory and critical care medicine, 2015-01, Vol.191, p.1

2015

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Low level Carbon Black nanoparticle (CBNP) exposure of the lung does not aggravate experimental asthma in mice

Pneumologie (Stuttgart, Germany), 2018, Vol.72 (7), p.536-536

2018

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C31 INFLAMMATORY MEDIATORS AND MECHANISMS IN ASTHMA: The [alpha]-Melanocyte Stimulating Hormone Regulates Allergic Airway Inflammation Through Melanocortin Receptor 5

American journal of respiratory and critical care medicine, 2014-01, Vol.189, p.1

2014

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C101 ALLERGIC AIRWAY INFLAMMATION AND HYPERRESPONSIVENESS: NOVEL MECHANISMS AND THERAPY: Il-37 Requires Il18r[alpha] And Sigirr To Ameliorate Experimental Allergic Asthma In Mice

American journal of respiratory and critical care medicine, 2015-01, Vol.191, p.1

2015

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IL-37 requires IL-18R[alpha] and SIGIRR/IL-1R8 to diminish allergic airway inflammation in mice

Allergy (Copenhagen), 2015-04, Vol.70 (4), p.366

2015

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The α-melanocyte stimulating hormone diminishes allergic airway inflammation through melanocortin receptor 5

Pneumologie (Stuttgart, Germany), 2016, Vol.70 (S 01)

2016

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A101 LYMPHOID CELLS AND LUNG INFLAMMATION: Tlr-3 Triggered Aggravation Of Experimental Asthma Depends On Il-17 And Is Associated With Il-17 Producing Nk Cells

American journal of respiratory and critical care medicine, 2014-01, Vol.189, p.1

2014

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IL-1R1 but not IL-18BP contributes to the beneficial effects of IL-37 on allergic asthma

Pneumologie (Stuttgart, Germany), 2017, Vol.71 (S 01), p.S1-S125

2017

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Treatment of airway inflammation and airway hyperresponsiveness in a mouse model of neutrophilic asthma using a RORγt-specific siRNA

Pneumologie (Stuttgart, Germany), 2015, Vol.69 (4)

2015

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Specific siRNA targeting RORγt inhibits airway inflammation and airway hyperresponsiveness in a mouse model of neutrophilic asthma

Pneumologie (Stuttgart, Germany), 2015, Vol.69 (S 01)

2015

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RORγt-specific RNAi decreases allergic airway inflammation and airway hyperresponsiveness in a mouse model of neutrophilic asthma

Pneumologie (Stuttgart, Germany), 2014, Vol.68 (6)

2014

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Treatment of allergic airway inflammation and airway hyperresponsiveness using RORγt specific RNAi

Pneumologie (Stuttgart, Germany), 2014, Vol.68 (S 01)

2014

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Kinetics of an asthma exacerbation in the mouse model

Pneumologie (Stuttgart, Germany), 2018-03, Vol.72 (S 01), p.S115-S116

2018

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IL-17 producing NK cells are suspected to be critical for the IL-17 dependent TLR-3 triggered aggravation of experimental asthma

Pneumologie (Stuttgart, Germany), 2014, Vol.68 (S 01)

2014

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Immunomodulatory role of IL-37 in asthma pathogenesis

Pneumologie (Stuttgart, Germany), 2016-07, Vol.70 (7)

2016

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IL-37 ameliorates experimental asthma via a mechanism that is independent from IL-18 signaling

Pneumologie (Stuttgart, Germany), 2016-02, Vol.70 (S 01)

2016

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α-melanocyte acts as an anti-inflammatory regulator in allergic bronchial asthma

Pneumologie (Stuttgart, Germany), 2014-02, Vol.68 (S 01)

2014

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IL-37 requires IL18Rα and SIGIRR/IL-1R8 to ameliorate experimental allergic asthma in mice

Pneumologie (Stuttgart, Germany), 2015-04, Vol.69 (4)

2015

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IL-37 requires IL18Rα and SIGIRR to ameliorate experimental allergic asthma in mice

Pneumologie (Stuttgart, Germany), 2015-02, Vol.69 (S 01)

2015

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Cooperative Cooperation: Being, Doing, and Becoming

Revista de administração contemporânea, 2020-11, Vol.24 (6), p.567-581A

Webering, Susana Iglesias

2020

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Cooperative Cooperation: Being, Doing, and Becoming/Cooperacao Cooperativa: o Ser, o Fazer e o Devir

RAC - Revista de Administracao Contemporanea, 2020-11, Vol.24 (6), p.567

Webering, Susana Iglesias

2020

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Poly(inosinic-cytidylic) acid-triggered exacerbation of experimental asthma depends on IL-17A produced by NK cells

The Journal of immunology (1950), 2015-06, Vol.194 (12), p.5615-5625

2015

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IL‐37 requires IL‐18Rα and SIGIRR/IL‐1R8 to diminish allergic airway inflammation in mice

NanoCOLT-Modulatory effects of modified carbon black nanoparticles in a mouse model of allergic asthma

D33 NEW BASIC SCIENCE IN ASTHMA: ALLERGIC INFLAMMATION II: Specific Sirna Targeting Ror[gamma]t Inhibits Airway Inflammation And Airway Hyperresponsiveness In A Mouse Model Of Neutrophilic Asthma

D33 NEW BASIC SCIENCE IN ASTHMA: ALLERGIC INFLAMMATION II: The [alpha]-Melanocyte Stimulating Hormone Is Produced In Allergic Asthma And Counterbalances Allergic Airway Inflammation In Mice

Low level Carbon Black nanoparticle (CBNP) exposure of the lung does not aggravate experimental asthma in mice

C31 INFLAMMATORY MEDIATORS AND MECHANISMS IN ASTHMA: The [alpha]-Melanocyte Stimulating Hormone Regulates Allergic Airway Inflammation Through Melanocortin Receptor 5

C101 ALLERGIC AIRWAY INFLAMMATION AND HYPERRESPONSIVENESS: NOVEL MECHANISMS AND THERAPY: Il-37 Requires Il18r[alpha] And Sigirr To Ameliorate Experimental Allergic Asthma In Mice

IL-37 requires IL-18R[alpha] and SIGIRR/IL-1R8 to diminish allergic airway inflammation in mice

The α-melanocyte stimulating hormone diminishes allergic airway inflammation through melanocortin receptor 5

A101 LYMPHOID CELLS AND LUNG INFLAMMATION: Tlr-3 Triggered Aggravation Of Experimental Asthma Depends On Il-17 And Is Associated With Il-17 Producing Nk Cells

IL-1R1 but not IL-18BP contributes to the beneficial effects of IL-37 on allergic asthma

Treatment of airway inflammation and airway hyperresponsiveness in a mouse model of neutrophilic asthma using a RORγt-specific siRNA

Specific siRNA targeting RORγt inhibits airway inflammation and airway hyperresponsiveness in a mouse model of neutrophilic asthma

RORγt-specific RNAi decreases allergic airway inflammation and airway hyperresponsiveness in a mouse model of neutrophilic asthma

Treatment of allergic airway inflammation and airway hyperresponsiveness using RORγt specific RNAi

Kinetics of an asthma exacerbation in the mouse model

IL-17 producing NK cells are suspected to be critical for the IL-17 dependent TLR-3 triggered aggravation of experimental asthma

Immunomodulatory role of IL-37 in asthma pathogenesis

IL-37 ameliorates experimental asthma via a mechanism that is independent from IL-18 signaling

α-melanocyte acts as an anti-inflammatory regulator in allergic bronchial asthma

IL-37 requires IL18Rα and SIGIRR/IL-1R8 to ameliorate experimental allergic asthma in mice

IL-37 requires IL18Rα and SIGIRR to ameliorate experimental allergic asthma in mice

Cooperative Cooperation: Being, Doing, and Becoming

Cooperative Cooperation: Being, Doing, and Becoming/Cooperacao Cooperativa: o Ser, o Fazer e o Devir

Poly(inosinic-cytidylic) acid-triggered exacerbation of experimental asthma depends on IL-17A produced by NK cells

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