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High-density lipoprotein inhibits serum amyloid A–mediated reactive oxygen species generation and NLRP3 inflammasome activation

The Journal of biological chemistry, 2018-08, Vol.293 (34), p.13257-13269

2018

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Dysfunctional HDL and atherosclerotic cardiovascular disease

Nature reviews cardiology, 2016-01, Vol.13 (1), p.48-60

2016

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Hepatocytes direct the formation of a pro-metastatic niche in the liver

Nature (London), 2019-03, Vol.567 (7747), p.249-252

2019

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Secreted Phospholipases A2 Are Intestinal Stem Cell Niche Factors with Distinct Roles in Homeostasis, Inflammation, and Cancer

Cell stem cell, 2016-07, Vol.19 (1), p.38-51

2016

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Serum Amyloid A is not obligatory for high-fat, high-sucrose, cholesterol-fed diet-induced obesity and its metabolic and inflammatory complications

PloS one, 2022-04, Vol.17 (4), p.e0266688-e0266688

2022

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High-Density Lipoproteins and Serum Amyloid A (SAA)

Current atherosclerosis reports, 2021-02, Vol.23 (2), p.7-7, Article 7

Webb, Nancy R.

2021

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Serum amyloid A augments the atherogenic effects of cholesteryl ester transfer protein

Journal of lipid research, 2023-05, Vol.64 (5), p.100365-100365, Article 100365

2023

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Serum amyloid A3 is a high density lipoprotein-associated acute-phase protein

Journal of lipid research, 2018-02, Vol.59 (2), p.339-347

2018

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Serum amyloid A is not incorporated into HDL during HDL biogenesis[S]

Journal of lipid research, 2020-03, Vol.61 (3), p.328-337

2020

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Deficiency of Acute-Phase Serum Amyloid A Exacerbates Sepsis-Induced Mortality and Lung Injury in Mice

International journal of molecular sciences, 2023-12, Vol.24 (24), p.17501

2023

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Group X Secretory Phospholipase A2 Regulates Insulin Secretion through a Cyclooxygenase-2-dependent Mechanism

The Journal of biological chemistry, 2014-10, Vol.289 (40), p.27410-27417

2014

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Nascent HDL formation in hepatocytes and role of ABCA1, ABCG1, and SR-BI

Journal of lipid research, 2012-03, Vol.53 (3), p.446-455

2012

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HDL remodeling during the acute phase response

Arteriosclerosis, thrombosis, and vascular biology, 2009-02, Vol.29 (2), p.261-267

2009

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Impact of individual acute phase serum amyloid A isoforms on HDL metabolism in mice[S]

Journal of lipid research, 2016-06, Vol.57 (6), p.969-979

2016

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Correction to: High-Density Lipoproteins and Serum Amyloid A (SAA)

Current atherosclerosis reports, 2022, Vol.24 (1), p.73-73

Webb, Nancy R.

2022

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Impact of serum amyloid A on high density lipoprotein composition and levels

Journal of lipid research, 2010-11, Vol.51 (11), p.3117-3125

2010

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Aortic Aneurysms and Dissections Series

Arteriosclerosis, thrombosis, and vascular biology, 2020-03, Vol.40 (3), p.e37

2020

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Syndecan-4 mediates macrophage uptake of group V secretory phospholipase A2-modified LDL

Journal of lipid research, 2009-04, Vol.50 (4), p.641-650

2009

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Group X secretory phospholipase A2 enhances TLR4 signaling in macrophages

The Journal of immunology (1950), 2011-07, Vol.187 (1), p.482-489

2011

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Group X secretory phospholipase A2 augments angiotensin II-induced inflammatory responses and abdominal aortic aneurysm formation in apoE-deficient mice

Atherosclerosis, 2011-01, Vol.214 (1), p.58-64

2011

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Distinct mechanisms for OxLDL uptake and cellular trafficking by class B scavenger receptors CD36 and SR-BI

Journal of lipid research, 2007-12, Vol.48 (12), p.2560-2570

2007

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Nascent HDL formation by hepatocytes is reduced by the concerted action of serum amyloid A and endothelial lipase

Journal of lipid research, 2011-12, Vol.52 (12), p.2255-2261

2011

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Group V Secretory Phospholipase A2-modified Low Density Lipoprotein Promotes Foam Cell Formation by a SR-A- and CD36-independent Process That Involves Cellular Proteoglycans

The Journal of biological chemistry, 2005-09, Vol.280 (38), p.32746-32752

2005

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Translation of High-Density Lipoprotein Function Into Clinical Practice: Current Prospects and Future Challenges

Circulation (New York, N.Y.), 2013-09, Vol.128 (11), p.1256-1267

2013

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High Density Lipoprotein Uptake by Scavenger Receptor SR-BII

The Journal of biological chemistry, 2004-04, Vol.279 (14), p.14372-14381

2004

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High-density lipoprotein inhibits serum amyloid A–mediated reactive oxygen species generation and NLRP3 inflammasome activation

Dysfunctional HDL and atherosclerotic cardiovascular disease

Hepatocytes direct the formation of a pro-metastatic niche in the liver

Secreted Phospholipases A2 Are Intestinal Stem Cell Niche Factors with Distinct Roles in Homeostasis, Inflammation, and Cancer

Serum Amyloid A is not obligatory for high-fat, high-sucrose, cholesterol-fed diet-induced obesity and its metabolic and inflammatory complications

High-Density Lipoproteins and Serum Amyloid A (SAA)

Serum amyloid A augments the atherogenic effects of cholesteryl ester transfer protein

Serum amyloid A3 is a high density lipoprotein-associated acute-phase protein

Serum amyloid A is not incorporated into HDL during HDL biogenesis[S]

Deficiency of Acute-Phase Serum Amyloid A Exacerbates Sepsis-Induced Mortality and Lung Injury in Mice

Group X Secretory Phospholipase A2 Regulates Insulin Secretion through a Cyclooxygenase-2-dependent Mechanism

Nascent HDL formation in hepatocytes and role of ABCA1, ABCG1, and SR-BI

HDL remodeling during the acute phase response

Impact of individual acute phase serum amyloid A isoforms on HDL metabolism in mice[S]

Correction to: High-Density Lipoproteins and Serum Amyloid A (SAA)

Impact of serum amyloid A on high density lipoprotein composition and levels

Aortic Aneurysms and Dissections Series

Syndecan-4 mediates macrophage uptake of group V secretory phospholipase A2-modified LDL

Group X secretory phospholipase A2 enhances TLR4 signaling in macrophages

Group X secretory phospholipase A2 augments angiotensin II-induced inflammatory responses and abdominal aortic aneurysm formation in apoE-deficient mice

Distinct mechanisms for OxLDL uptake and cellular trafficking by class B scavenger receptors CD36 and SR-BI

Nascent HDL formation by hepatocytes is reduced by the concerted action of serum amyloid A and endothelial lipase

Group V Secretory Phospholipase A2-modified Low Density Lipoprotein Promotes Foam Cell Formation by a SR-A- and CD36-independent Process That Involves Cellular Proteoglycans

Translation of High-Density Lipoprotein Function Into Clinical Practice: Current Prospects and Future Challenges

High Density Lipoprotein Uptake by Scavenger Receptor SR-BII

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