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Allergic aspergillus sinusitis: Concepts in diagnosis and treatment of a new clinical entity

The Laryngoscope, 1987-03, Vol.97 (3), p.261-266

1987

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Noncanonical Roles of Voltage-Gated Sodium Channels

Neuron (Cambridge, Mass.), 2013-10, Vol.80 (2), p.280-291

2013

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Sodium Channels in Normal and Pathological Pain

Annual review of neuroscience, 2010-01, Vol.33 (1), p.325-347

2010

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Expression of Nav1.7 in DRG Neurons Extends from Peripheral Terminals in the Skin to Central Preterminal Branches and Terminals in the Dorsal Horn

Molecular pain, 2012-11, Vol.8 (1), p.82-82

2012

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Sodium channels in astroglia and microglia

Glia, 2016-10, Vol.64 (10), p.1628-1645

2016

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The Na(V)1.7 sodium channel: from molecule to man

Nature reviews. Neuroscience, 2013-01, Vol.14 (1), p.49

2013

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VERSE-WRITING CONTEST RESULTS

The Classical outlook, 1965-05, Vol.42 (9), p.100-102

1965

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Screening fluorescent voltage indicators with spontaneously spiking HEK cells

PloS one, 2013-12, Vol.8 (12), p.e85221

2013

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Multiple sodium channel isoforms and mitogen-activated protein kinases are present in painful human neuromas

Annals of neurology, 2008-12, Vol.64 (6), p.644-653

2008

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Sodium-Calcium Exchanger and Multiple Sodium Channel Isoforms in Intra-Epidermal Nerve Terminals

Molecular pain, 2010-11, Vol.6 (1), p.84-84

2010

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Safety Profile of Nivolumab Monotherapy: A Pooled Analysis of Patients With Advanced Melanoma

Journal of clinical oncology, 2017-03, Vol.35 (7), p.785-792

2017

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A single sodium channel mutation produces hyper- or hypoexcitability in different types of neurons

Proceedings of the National Academy of Sciences - PNAS, 2006-05, Vol.103 (21), p.8245-8250

2006

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Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain

Pain (Amsterdam), 2004-04, Vol.108 (3), p.237-247

2004

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Homogenizing Entropy Across Different Environmental Conditions: A Universally Applicable Method for Transforming Continuous Variables

IEEE transactions on information theory, 2023-03, Vol.69 (3), p.1394-1412, Article 1394

2023

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Sodium channel activity modulates multiple functions in microglia

Glia, 2009-08, Vol.57 (10), p.1072-1081

2009

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ERK1/2 Mitogen-Activated Protein Kinase Phosphorylates Sodium Channel Nav1.7 and Alters Its Gating Properties

The Journal of neuroscience, 2010-02, Vol.30 (5), p.1637-1647

2010

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A Novel Persistent Tetrodotoxin-Resistant Sodium Current In SNS-Null And Wild-Type Small Primary Sensory Neurons

The Journal of neuroscience, 1999-12, Vol.19 (24), p.43-RC43

1999

Volltextzugriff (PDF)

Phosphorylation of Sodium Channel Nav1.8 by p38 Mitogen-Activated Protein Kinase Increases Current Density in Dorsal Root Ganglion Neurons

The Journal of neuroscience, 2008-03, Vol.28 (12), p.3190-3201

2008

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Voltage-Gated Sodium Channels: Therapeutic Targets for Pain

Pain medicine (Malden, Mass.), 2009-10, Vol.10 (7), p.1260-1269

2009

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Nav1.7 is the Predominant Sodium Channel in Rodent Olfactory Sensory Neurons

Molecular pain, 2011-05, Vol.7 (1), p.32-32

2011

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Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5

Brain (London, England : 1878), 2010-03, Vol.133 (3), p.835-846

2010

Volltextzugriff (PDF)

What is adaptation and how should it be measured?

Journal of theoretical biology, 2018-06, Vol.447, p.190-198

2018

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Phenytoin Protects Spinal Cord Axons and Preserves Axonal Conduction and Neurological Function in a Model of Neuroinflammation In Vivo

Journal of neurophysiology, 2003-11, Vol.90 (5), p.3566-3571

2003

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Transfection of rat or mouse neurons by biolistics or electroporation

Nature protocols, 2009-08, Vol.4 (8), p.1118-1127

2009

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Sodium channels contribute to degeneration of dorsal root ganglion neurites induced by mitochondrial dysfunction in an in vitro model of axonal injury

The Journal of neuroscience, 2013-12, Vol.33 (49), p.19250-19261

2013

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Allergic aspergillus sinusitis: Concepts in diagnosis and treatment of a new clinical entity

Noncanonical Roles of Voltage-Gated Sodium Channels

Sodium Channels in Normal and Pathological Pain

Expression of Nav1.7 in DRG Neurons Extends from Peripheral Terminals in the Skin to Central Preterminal Branches and Terminals in the Dorsal Horn

Sodium channels in astroglia and microglia

The Na(V)1.7 sodium channel: from molecule to man

VERSE-WRITING CONTEST RESULTS

Screening fluorescent voltage indicators with spontaneously spiking HEK cells

Multiple sodium channel isoforms and mitogen-activated protein kinases are present in painful human neuromas

Sodium-Calcium Exchanger and Multiple Sodium Channel Isoforms in Intra-Epidermal Nerve Terminals

Safety Profile of Nivolumab Monotherapy: A Pooled Analysis of Patients With Advanced Melanoma

A single sodium channel mutation produces hyper- or hypoexcitability in different types of neurons

Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain

Homogenizing Entropy Across Different Environmental Conditions: A Universally Applicable Method for Transforming Continuous Variables

Sodium channel activity modulates multiple functions in microglia

ERK1/2 Mitogen-Activated Protein Kinase Phosphorylates Sodium Channel Nav1.7 and Alters Its Gating Properties

A Novel Persistent Tetrodotoxin-Resistant Sodium Current In SNS-Null And Wild-Type Small Primary Sensory Neurons

Phosphorylation of Sodium Channel Nav1.8 by p38 Mitogen-Activated Protein Kinase Increases Current Density in Dorsal Root Ganglion Neurons

Voltage-Gated Sodium Channels: Therapeutic Targets for Pain

Nav1.7 is the Predominant Sodium Channel in Rodent Olfactory Sensory Neurons

Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5

What is adaptation and how should it be measured?

Phenytoin Protects Spinal Cord Axons and Preserves Axonal Conduction and Neurological Function in a Model of Neuroinflammation In Vivo

Transfection of rat or mouse neurons by biolistics or electroporation

Sodium channels contribute to degeneration of dorsal root ganglion neurites induced by mitochondrial dysfunction in an in vitro model of axonal injury

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