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Toll-like Receptor 2 Is Required for LPS-induced Toll-like Receptor 4 Signaling and Inhibition of Ion Transport in Renal Thick Ascending Limb

The Journal of biological chemistry, 2012-06, Vol.287 (24), p.20208-20220

2012

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Monophosphoryl lipid A induces protection against LPS in medullary thick ascending limb through a TLR4-TRIF-PI3K signaling pathway

American journal of physiology. Renal physiology, 2017-07, Vol.313 (1), p.F103-F115

2017

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Lipopolysaccharide directly alters renal tubule transport through distinct TLR4-dependent pathways in basolateral and apical membranes

American journal of physiology. Renal physiology, 2009-10, Vol.297 (4), p.F866-F874

2009

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Lumen LPS inhibits HCO3(-) absorption in the medullary thick ascending limb through TLR4-PI3K-Akt-mTOR-dependent inhibition of basolateral Na+/H+ exchange

American journal of physiology. Renal physiology, 2013-08, Vol.305 (4), p.F451-F462

2013

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Monophosphoryl lipid A pretreatment suppresses sepsis- and LPS-induced proinflammatory cytokine production in medullary thick ascending limb

American journal of physiology. Renal physiology, 2020-07, Vol.319 (1), p.F8-F18

2020

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Monophosphoryl lipid A induces protection against LPS in medullary thick ascending limb through induction of Tollip and negative regulation of IRAK-1

American journal of physiology. Renal physiology, 2019-09, Vol.317 (3), p.F705-F719

2019

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High-mobility group box 1 inhibits HCO3- absorption in the medullary thick ascending limb through RAGE-Rho-ROCK-mediated inhibition of basolateral Na+/H+ exchange

American journal of physiology. Renal physiology, 2016-09, Vol.311 (3), p.F600-F613

2016

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Aldosterone inhibits apical NHE3 and HCO3- absorption via a nongenomic ERK-dependent pathway in medullary thick ascending limb

American journal of physiology. Renal physiology, 2006-11, Vol.291 (5), p.F1005-F1013

2006

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A two-hit mechanism for sepsis-induced impairment of renal tubule function

American journal of physiology. Renal physiology, 2013-04, Vol.304 (7), p.F863-F874

2013

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Nongenomic regulation by aldosterone of the epithelial NHE3 Na+/H+ exchanger

American Journal of Physiology: Cell Physiology, 2006-03, Vol.290 (3), p.C757-C763

2006

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Transepithelial HCO3- absorption is defective in renal thick ascending limbs from Na+/H+ exchanger NHE1 null mutant mice

American journal of physiology. Renal physiology, 2004-12, Vol.287 (6), p.F1244-F1249

2004

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Monophosphoryl lipid A prevents impairment of medullary thick ascending limb HCO3− absorption and improves plasma HCO3− concentration in septic mice

American journal of physiology. Renal physiology, 2018-09, Vol.315 (3), p.F711-F725

2018

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The Basolateral NHE1 Na+/H+ Exchanger Regulates Transepithelial HCO3− Absorption through Actin Cytoskeleton Remodeling in Renal Thick Ascending Limb

The Journal of biological chemistry, 2005-03, Vol.280 (12), p.11439-11447

2005

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p38 Kinase Activity Is Essential for Osmotic Induction of mRNAs for HSP70 and Transporter for Organic Solute Betaine in Madin-Darby Canine Kidney Cells

The Journal of biological chemistry, 1998-01, Vol.273 (3), p.1832-1837

1998

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Aldosterone potentiates 1,25-dihydroxyvitamin D3 action in renal thick ascending limb via a nongenomic, ERK-dependent pathway

American Journal of Physiology: Cell Physiology, 2003-11, Vol.285 (5), p.C1122

2003

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Basolateral LPS inhibits NHE3 and HCO3- absorption through TLR4/MyD88-dependent ERK activation in medullary thick ascending limb

American Journal of Physiology: Cell Physiology, 2011-12, Vol.301 (6), p.C1296-C1306

2011

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Nerve Growth Factor Inhibits Na⁺/H⁺ Exchange and {f1} Absorption through Parallel Phosphatidylinositol 3-Kinase-mTOR and ERK Pathways in Thick Ascending Limb

The Journal of biological chemistry, 2008-09, Vol.283 (39), p.26602-26611

2008

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High-mobility group box 1 inhibits HCO(3)(-) absorption in medullary thick ascending limb through a basolateral receptor for advanced glycation end products pathway

American journal of physiology. Renal physiology, 2015-10, Vol.309 (8), p.F720-F730

2015

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Nerve growth factor inhibits Na+/H+ exchange and formula absorption through parallel phosphatidylinositol 3-kinase-mTOR and ERK pathways in thick ascending limb

The Journal of biological chemistry, 2008-09, Vol.283 (39), p.26602

2008

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Hyposmolality stimulates Na+/H+ exchange and HCO3- absorption in thick ascending limb via PI 3-kinase

American Journal of Physiology: Cell Physiology, 2000-11, Vol.279 (5), p.C1443

2000

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High-mobility group box 1 inhibits HCO 3 − absorption in the medullary thick ascending limb through RAGE-Rho-ROCK-mediated inhibition of basolateral Na + /H + exchange

American journal of physiology. Renal physiology, 2016-09, Vol.311 (3), p.F600-F613

2016

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High-mobility group box 1 inhibits HCO^sub 3^- absorption in the medullary thick ascending limb through RAGE-Rho-ROCK-mediated inhibition of basolateral Na+/H+ exchange

American journal of physiology. Renal physiology, 2016-09, Vol.311 (3), p.F600

2016

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High-mobility group box 1 inhibits HCO3− absorption in medullary thick ascending limb through a basolateral receptor for advanced glycation end products pathway

American journal of physiology. Renal physiology, 2015-10, Vol.309 (8), p.F720-F730

2015

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Aldosterone inhibits HCO absorption via a nongenomic pathway in medullary thick ascending limb

American journal of physiology. Renal physiology, 2002-10, Vol.283 (4), p.F699-F706

2002

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Basolateral LPS inhibits NHE3 and HCOFormula absorption through TLR4/MyD88-dependent ERK activation in medullary thick ascending limb

American Journal of Physiology: Cell Physiology, 2011-12, Vol.301 (6), p.C1296

2011

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Toll-like Receptor 2 Is Required for LPS-induced Toll-like Receptor 4 Signaling and Inhibition of Ion Transport in Renal Thick Ascending Limb

Monophosphoryl lipid A induces protection against LPS in medullary thick ascending limb through a TLR4-TRIF-PI3K signaling pathway

Lipopolysaccharide directly alters renal tubule transport through distinct TLR4-dependent pathways in basolateral and apical membranes

Lumen LPS inhibits HCO3(-) absorption in the medullary thick ascending limb through TLR4-PI3K-Akt-mTOR-dependent inhibition of basolateral Na+/H+ exchange

Monophosphoryl lipid A pretreatment suppresses sepsis- and LPS-induced proinflammatory cytokine production in medullary thick ascending limb

Monophosphoryl lipid A induces protection against LPS in medullary thick ascending limb through induction of Tollip and negative regulation of IRAK-1

High-mobility group box 1 inhibits HCO3- absorption in the medullary thick ascending limb through RAGE-Rho-ROCK-mediated inhibition of basolateral Na+/H+ exchange

Aldosterone inhibits apical NHE3 and HCO3- absorption via a nongenomic ERK-dependent pathway in medullary thick ascending limb

A two-hit mechanism for sepsis-induced impairment of renal tubule function

Nongenomic regulation by aldosterone of the epithelial NHE3 Na+/H+ exchanger

Transepithelial HCO3- absorption is defective in renal thick ascending limbs from Na+/H+ exchanger NHE1 null mutant mice

Monophosphoryl lipid A prevents impairment of medullary thick ascending limb HCO3− absorption and improves plasma HCO3− concentration in septic mice

The Basolateral NHE1 Na+/H+ Exchanger Regulates Transepithelial HCO3− Absorption through Actin Cytoskeleton Remodeling in Renal Thick Ascending Limb

p38 Kinase Activity Is Essential for Osmotic Induction of mRNAs for HSP70 and Transporter for Organic Solute Betaine in Madin-Darby Canine Kidney Cells

Aldosterone potentiates 1,25-dihydroxyvitamin D3 action in renal thick ascending limb via a nongenomic, ERK-dependent pathway

Basolateral LPS inhibits NHE3 and HCO3- absorption through TLR4/MyD88-dependent ERK activation in medullary thick ascending limb

Nerve Growth Factor Inhibits Na⁺/H⁺ Exchange and {f1} Absorption through Parallel Phosphatidylinositol 3-Kinase-mTOR and ERK Pathways in Thick Ascending Limb

High-mobility group box 1 inhibits HCO(3)(-) absorption in medullary thick ascending limb through a basolateral receptor for advanced glycation end products pathway

Nerve growth factor inhibits Na+/H+ exchange and formula absorption through parallel phosphatidylinositol 3-kinase-mTOR and ERK pathways in thick ascending limb

Hyposmolality stimulates Na+/H+ exchange and HCO3- absorption in thick ascending limb via PI 3-kinase

High-mobility group box 1 inhibits HCO 3 − absorption in the medullary thick ascending limb through RAGE-Rho-ROCK-mediated inhibition of basolateral Na + /H + exchange

High-mobility group box 1 inhibits HCO^sub 3^- absorption in the medullary thick ascending limb through RAGE-Rho-ROCK-mediated inhibition of basolateral Na+/H+ exchange

High-mobility group box 1 inhibits HCO3− absorption in medullary thick ascending limb through a basolateral receptor for advanced glycation end products pathway

Aldosterone inhibits HCO absorption via a nongenomic pathway in medullary thick ascending limb

Basolateral LPS inhibits NHE3 and HCOFormula absorption through TLR4/MyD88-dependent ERK activation in medullary thick ascending limb

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