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Caveolin-1-dependent occludin endocytosis is required for TNF-induced tight junction regulation in vivo

The Journal of cell biology, 2010-04, Vol.189 (1), p.111-126

2010

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The Epithelial Barrier Is Maintained by In Vivo Tight Junction Expansion During Pathologic Intestinal Epithelial Shedding

Gastroenterology (New York, N.Y. 1943), 2011-04, Vol.140 (4), p.1208-1218.e2

2011

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Calcium Elevation in Mitochondria Is the Main Ca2+ Requirement for Mitochondrial Permeability Transition Pore (mPTP) Opening

The Journal of biological chemistry, 2009-07, Vol.284 (31), p.20796-20803

2009

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Rho-A prenylation and signaling link epithelial homeostasis to intestinal inflammation

The Journal of clinical investigation, 2016-02, Vol.126 (2), p.611-626

2016

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Colon Cancer: A Civilization Disorder

Digestive diseases (Basel), 2011-01, Vol.29 (2), p.222-228

2011

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γδ Intraepithelial Lymphocytes Facilitate Pathological Epithelial Cell Shedding Via CD103-Mediated Granzyme Release

Gastroenterology (New York, N.Y. 1943), 2022-03, Vol.162 (3), p.877-889.e7

2022

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The Relative Contributions of the Gut Microbiome, Host Genetics, and Environment to Cytokine Responses to Microbial Stimulation

Gastroenterology (New York, N.Y. 1943), 2017-06, Vol.152 (8), p.2068-2070

2017

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The Mucosal Microbiome and Recurrence After Surgery for Crohn’s Disease

Gastroenterology (New York, N.Y. 1943), 2016-06, Vol.150 (7), p.1682-1684

2016

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Identification of Epithelial Gaps in Human Small and Large Intestine by Confocal Endomicroscopy

Gastroenterology (New York, N.Y. 1943), 2007-12, Vol.133 (6), p.1769-1778

2007

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The Enteric Virome in Inflammatory Bowel Disease

Gastroenterology (New York, N.Y. 1943), 2015-10, Vol.149 (4), p.1120-1121

2015

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Gut Microbiome in New-Onset Crohn's Disease

Gastroenterology (New York, N.Y. 1943), 2014-10, Vol.147 (4), p.932-934

2014

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Mechanisms of Epithelial Cell Shedding in the Mammalian Intestine and Maintenance of Barrier Function

Annals of the New York Academy of Sciences, 2009-05, Vol.1165 (1), p.135-142

2009

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Details Unfold: The Endoplasmic Reticulum Stress Response in Intestinal Inflammation and Cancer

Gastroenterology (New York, N.Y. 1943), 2014-08, Vol.147 (2), p.531-533

2014

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Effect of Neonatal Nutrition on Long-Term Barrier Function and Gut Inflammation

Gastroenterology (New York, N.Y. 1943), 2011-11, Vol.141 (5), p.1939-1941

Watson, Alastair J.M

2011

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Bacterial Translocation Influences the Response to Biological Therapy in Crohn's Disease

Gastroenterology (New York, N.Y. 1943), 2013-10, Vol.145 (4), p.898-901

2013

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Regulation of Host Gene Expression by Gut Microbiota

Gastroenterology (New York, N.Y. 1943), 2013-04, Vol.144 (4), p.841-844

2013

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Mesenteric Fat as a Source of CRP and Target for Bacterial Translocation in Crohn's Disease

Gastroenterology (New York, N.Y. 1943), 2012-08, Vol.143 (2), p.496-498

2012

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Role of Autophagy in NOD2-Induced Inflammation in Crohn's Disease

Gastroenterology (New York, N.Y. 1943), 2012-04, Vol.142 (4), p.1032-1034

2012

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Environmental Risk Factors for Inflammatory Bowel Disease: Microbes, Diet, and the Appendix

Gastroenterology (New York, N.Y. 1943), 2011-08, Vol.141 (2), p.768-770

2011

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Activated Intestinal Macrophages in Patients with Cirrhosis Release NO and IL-6 that May Disrupt Intestinal Barrier Function

Gastroenterology (New York, N.Y. 1943), 2013-12, Vol.145 (6), p.1481-1484

2013

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Mucosal Gene Expression Signatures That Predict Response of Ulcerative Colitis to Infliximab

Gastroenterology (New York, N.Y. 1943), 2011-02, Vol.140 (2), p.729-731

2011

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Cell Shedding: Old Questions Answered

Gastroenterology (New York, N.Y. 1943), 2012-11, Vol.143 (5), p.1389-1391

2012

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Hsp90 inhibitors sensitise human colon cancer cells to topoisomerase I poisons by depletion of key anti-apoptotic and cell cycle checkpoint proteins

Biochemical pharmacology, 2012-02, Vol.83 (3), p.355-367

2012

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Treatment of Inflammatory Bowel Disease With Genetically Engineered Bacteria

Gastroenterology (New York, N.Y. 1943), 2010-08, Vol.139 (2), p.685-687

2010

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Gut Microbiota Control Gut Permeability Through GLP-2

Gastroenterology (New York, N.Y. 1943), 2010-02, Vol.138 (2), p.779-781

2010

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Caveolin-1-dependent occludin endocytosis is required for TNF-induced tight junction regulation in vivo

The Epithelial Barrier Is Maintained by In Vivo Tight Junction Expansion During Pathologic Intestinal Epithelial Shedding

Calcium Elevation in Mitochondria Is the Main Ca2+ Requirement for Mitochondrial Permeability Transition Pore (mPTP) Opening

Rho-A prenylation and signaling link epithelial homeostasis to intestinal inflammation

Colon Cancer: A Civilization Disorder

γδ Intraepithelial Lymphocytes Facilitate Pathological Epithelial Cell Shedding Via CD103-Mediated Granzyme Release

The Relative Contributions of the Gut Microbiome, Host Genetics, and Environment to Cytokine Responses to Microbial Stimulation

The Mucosal Microbiome and Recurrence After Surgery for Crohn’s Disease

Identification of Epithelial Gaps in Human Small and Large Intestine by Confocal Endomicroscopy

The Enteric Virome in Inflammatory Bowel Disease

Gut Microbiome in New-Onset Crohn's Disease

Mechanisms of Epithelial Cell Shedding in the Mammalian Intestine and Maintenance of Barrier Function

Details Unfold: The Endoplasmic Reticulum Stress Response in Intestinal Inflammation and Cancer

Effect of Neonatal Nutrition on Long-Term Barrier Function and Gut Inflammation

Bacterial Translocation Influences the Response to Biological Therapy in Crohn's Disease

Regulation of Host Gene Expression by Gut Microbiota

Mesenteric Fat as a Source of CRP and Target for Bacterial Translocation in Crohn's Disease

Role of Autophagy in NOD2-Induced Inflammation in Crohn's Disease

Environmental Risk Factors for Inflammatory Bowel Disease: Microbes, Diet, and the Appendix

Activated Intestinal Macrophages in Patients with Cirrhosis Release NO and IL-6 that May Disrupt Intestinal Barrier Function

Mucosal Gene Expression Signatures That Predict Response of Ulcerative Colitis to Infliximab

Cell Shedding: Old Questions Answered

Hsp90 inhibitors sensitise human colon cancer cells to topoisomerase I poisons by depletion of key anti-apoptotic and cell cycle checkpoint proteins

Treatment of Inflammatory Bowel Disease With Genetically Engineered Bacteria

Gut Microbiota Control Gut Permeability Through GLP-2

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