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Paclitaxel-Associated Mechanical Sensitivity and Neuroinflammation Are Sex-, Time-, and Site-Specific and Prevented through Cannabigerol Administration in C57Bl/6 Mice

International journal of molecular sciences, 2024-04, Vol.25 (8), p.4277

2024

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Cannabidiol inhibits paclitaxel‐induced neuropathic pain through 5‐HT1A receptors without diminishing nervous system function or chemotherapy efficacy

British journal of pharmacology, 2014-02, Vol.171 (3), p.636-645

2014

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Cocaine-mediated activation of microglia and microglial MeCP2 and BDNF production

Neurobiology of disease, 2018-09, Vol.117, p.28-41

2018

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Effects of Cannabidiol and Beta-Caryophyllene Alone or in Combination in a Mouse Model of Permanent Ischemia

International journal of molecular sciences, 2021-03, Vol.22 (6), p.2866

2021

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Non-Psychoactive Cannabinoid Modulation of Nociception and Inflammation Associated with a Rat Model of Pulpitis

Biomolecules (Basel, Switzerland), 2023-05, Vol.13 (5), p.846

2023

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Single and combined effects of plant‐derived and synthetic cannabinoids on cognition and cannabinoid‐associated withdrawal signs in mice

British journal of pharmacology, 2019-05, Vol.176 (10), p.1552-1567

2019

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Effects of Paclitaxel on Mechanical Sensitivity and Morphine Reward in Male and Female C57Bl6 Mice

Experimental and clinical psychopharmacology, 2016-12, Vol.24 (6), p.485-495

2016

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Kratom alkaloid mitragynine: Inhibition of chemotherapy-induced peripheral neuropathy in mice is dependent on sex and active adrenergic and opioid receptors

IBRO neuroscience reports, 2022-12, Vol.13, p.198-206

2022

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Modulation of Morphine Analgesia, Antinociceptive Tolerance, and Mu-Opioid Receptor Binding by the Cannabinoid CB2 Receptor Agonist O-1966

Frontiers in pharmacology, 2022-04, Vol.13, p.803331-803331

2022

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Single and combined effects of Δ9‐tetrahydrocannabinol and cannabidiol in a mouse model of chemotherapy‐induced neuropathic pain

British journal of pharmacology, 2017-09, Vol.174 (17), p.2832-2841

2017

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Effect of Cannabinoid CB1 Receptor Antagonist SR141714A and CB1 Receptor Knockout on Cue-Induced Reinstatement of Ensure and Corn-Oil Seeking in Mice

Neuropsychopharmacology (New York, N.Y.), 2007-12, Vol.32 (12), p.2592-2600

2007

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Cannabidiol inhibits paclitaxel‐induced neuropathic pain through 5‐ HT 1A receptors without diminishing nervous system function or chemotherapy efficacy

British journal of pharmacology, 2014-02, Vol.171 (3), p.636-645

2014

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Cancer, Chemotherapy and Cognition

Life sciences (1973), 2013-10, Vol.93 (17), p.579-580

Ward, Sara Jane

2013

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Effects of a Cannabinoid1 receptor antagonist and Serotonin2C receptor agonist alone and in combination on motivation for palatable food: a dose-addition analysis study in mice

The Journal of pharmacology and experimental therapeutics, 2008-05, Vol.325 (2), p.567

2008

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Cannabidiol prevents the development of cold and mechanical allodynia in paclitaxel-treated female C57Bl6 mice

Anesthesia and analgesia, 2011-10, Vol.113 (4), p.947-950

2011

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Single and combined effects of Δ 9 -tetrahydrocannabinol and cannabidiol in a mouse model of chemotherapy-induced neuropathic pain

British journal of pharmacology, 2017-09, Vol.174 (17), p.2832-2841

2017

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The non-psychoactive phytocannabinoid cannabidiol (CBD) attenuates pro-inflammatory mediators, T cell infiltration, and thermal sensitivity following spinal cord injury in mice

Cellular immunology, 2018-07, Vol.329, p.1-9

2018

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Knockdown siRNA Targeting the Mitochondrial Sodium-Calcium Exchanger-1 Inhibits the Protective Effects of Two Cannabinoids Against Acute Paclitaxel Toxicity

Journal of molecular neuroscience, 2019-08, Vol.68 (4), p.603-619

2019

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Cannabinoids and Cancer Chemotherapy-Associated Adverse Effects

Journal of the National Cancer Institute. Monographs, 2021-11, Vol.2021 (58), p.78-85

2021

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Neurotrophic and Adrenergic Mechanisms Contribute to the Anti‐Allodynic Efficacy of the Kratom Alkaloid Mitragynine Against Chemotherapy‐Induced Peripheral Neuropathy

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a

2022

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Knockdown siRNA Targeting GPR55 Reveals Significant Differences Between the Anti-inflammatory Actions of KLS-13019 and Cannabidiol

Journal of molecular neuroscience, 2024-04, Vol.74 (2), p.41-41

2024

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Behavioural and pharmacological effects of cannabidiol (CBD) and the cannabidiol analogue KLS‐13019 in mouse models of pain and reinforcement

British journal of pharmacology, 2021-08, Vol.178 (15), p.3067-3078

2021

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Botulinum Toxin A Improves Symptoms of Gastroparesis

Digestive diseases and sciences, 2020-05, Vol.65 (5), p.1396-1404

2020

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Cannabinoid mechanisms contribute to the therapeutic efficacy of the kratom alkaloid mitragynine against neuropathic, but not inflammatory pain

Life sciences (1973), 2023-09, Vol.328, p.121878-121878, Article 121878

2023

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Rimonabant Redux and Strategies to Improve the Future Outlook of CB1 Receptor Neutral-Antagonist/Inverse-Agonist Therapies

Obesity (Silver Spring, Md.), 2011-07, Vol.19 (7), p.1325-1334

2011

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Paclitaxel-Associated Mechanical Sensitivity and Neuroinflammation Are Sex-, Time-, and Site-Specific and Prevented through Cannabigerol Administration in C57Bl/6 Mice

Cannabidiol inhibits paclitaxel‐induced neuropathic pain through 5‐HT1A receptors without diminishing nervous system function or chemotherapy efficacy

Cocaine-mediated activation of microglia and microglial MeCP2 and BDNF production

Effects of Cannabidiol and Beta-Caryophyllene Alone or in Combination in a Mouse Model of Permanent Ischemia

Non-Psychoactive Cannabinoid Modulation of Nociception and Inflammation Associated with a Rat Model of Pulpitis

Single and combined effects of plant‐derived and synthetic cannabinoids on cognition and cannabinoid‐associated withdrawal signs in mice

Effects of Paclitaxel on Mechanical Sensitivity and Morphine Reward in Male and Female C57Bl6 Mice

Kratom alkaloid mitragynine: Inhibition of chemotherapy-induced peripheral neuropathy in mice is dependent on sex and active adrenergic and opioid receptors

Modulation of Morphine Analgesia, Antinociceptive Tolerance, and Mu-Opioid Receptor Binding by the Cannabinoid CB2 Receptor Agonist O-1966

Single and combined effects of Δ9‐tetrahydrocannabinol and cannabidiol in a mouse model of chemotherapy‐induced neuropathic pain

Effect of Cannabinoid CB1 Receptor Antagonist SR141714A and CB1 Receptor Knockout on Cue-Induced Reinstatement of Ensure and Corn-Oil Seeking in Mice

Cannabidiol inhibits paclitaxel‐induced neuropathic pain through 5‐ HT 1A receptors without diminishing nervous system function or chemotherapy efficacy

Cancer, Chemotherapy and Cognition

Effects of a Cannabinoid1 receptor antagonist and Serotonin2C receptor agonist alone and in combination on motivation for palatable food: a dose-addition analysis study in mice

Cannabidiol prevents the development of cold and mechanical allodynia in paclitaxel-treated female C57Bl6 mice

Single and combined effects of Δ 9 -tetrahydrocannabinol and cannabidiol in a mouse model of chemotherapy-induced neuropathic pain

The non-psychoactive phytocannabinoid cannabidiol (CBD) attenuates pro-inflammatory mediators, T cell infiltration, and thermal sensitivity following spinal cord injury in mice

Knockdown siRNA Targeting the Mitochondrial Sodium-Calcium Exchanger-1 Inhibits the Protective Effects of Two Cannabinoids Against Acute Paclitaxel Toxicity

Cannabinoids and Cancer Chemotherapy-Associated Adverse Effects

Neurotrophic and Adrenergic Mechanisms Contribute to the Anti‐Allodynic Efficacy of the Kratom Alkaloid Mitragynine Against Chemotherapy‐Induced Peripheral Neuropathy

Knockdown siRNA Targeting GPR55 Reveals Significant Differences Between the Anti-inflammatory Actions of KLS-13019 and Cannabidiol

Behavioural and pharmacological effects of cannabidiol (CBD) and the cannabidiol analogue KLS‐13019 in mouse models of pain and reinforcement

Botulinum Toxin A Improves Symptoms of Gastroparesis

Cannabinoid mechanisms contribute to the therapeutic efficacy of the kratom alkaloid mitragynine against neuropathic, but not inflammatory pain

Rimonabant Redux and Strategies to Improve the Future Outlook of CB1 Receptor Neutral-Antagonist/Inverse-Agonist Therapies

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