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Structural basis for the gating mechanism of the type 2 ryanodine receptor RyR2

Science (American Association for the Advancement of Science), 2016-10, Vol.354 (6310), p.301-301

2016

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Modulation of cardiac ryanodine receptor 2 by calmodulin

Nature (London), 2019-08, Vol.572 (7769), p.347-351

2019

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Popularity enhances the interdependent network reciprocity

New journal of physics, 2018-12, Vol.20 (12), p.123012

2018

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Elevated expression of immune and DNA repair genes in mated queens and kings of the Reticulitermes chinensis termites

Experimental gerontology, 2023-07, Vol.178, p.112228-112228, Article 112228

2023

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Differential microbial assembly processes and co‐occurrence networks in the soil‐root continuum along an environmental gradient

iMeta, 2022-06, Vol.1 (2), p.e18-n/a

2022

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Cardiac ryanodine receptor calcium release deficiency syndrome

Science translational medicine, 2021-02, Vol.13 (579)

2021

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The ryanodine receptor store-sensing gate controls Ca2+ waves and Ca2+-triggered arrhythmias

Nature medicine, 2014-02, Vol.20 (2), p.184-192

2014

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Abnormal Termination of Ca2+ Release Is a Common Defect of RyR2 Mutations Associated With Cardiomyopathies

Circulation research, 2012-03, Vol.110 (7), p.968-977

2012

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Limiting RyR2 Open Time Prevents Alzheimer’s Disease-Related Neuronal Hyperactivity and Memory Loss but Not β-Amyloid Accumulation

Cell reports (Cambridge), 2020-09, Vol.32 (12), p.108169-108169, Article 108169

2020

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Arrhythmogenic Calmodulin Mutations Affect the Activation and Termination of Cardiac Ryanodine Receptor-mediated Ca2+ Release

The Journal of biological chemistry, 2015-10, Vol.290 (43), p.26151-26162

2015

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Carvedilol and its new analogs suppress arrhythmogenic store overload-induced Ca2+ release

Nature medicine, 2011-07, Vol.17 (8), p.1003-1009

2011

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Disease-Induced Cooperation Mitigates Populations Against Decline: The Cascade Effect of Cooperation Evolution

Frontiers in ecology and evolution, 2022-03, Vol.10

2022

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Infanticide vs. inherited cardiac arrhythmias

Europace (London, England), 2021-03, Vol.23 (3), p.441-450

2021

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Enhanced Cytosolic Ca2+ Activation Underlies a Common Defect of Central Domain Cardiac Ryanodine Receptor Mutations Linked to Arrhythmias

The Journal of biological chemistry, 2016-11, Vol.291 (47), p.24528-24537

2016

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De novo ITPR1 variants are a recurrent cause of early-onset ataxia, acting via loss of channel function

European journal of human genetics : EJHG, 2018-06, Vol.26 (11), p.1623-1634

2018

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Enhanced Store Overload–Induced Ca2+ Release and Channel Sensitivity to Luminal Ca2+ Activation Are Common Defects of RyR2 Mutations Linked to Ventricular Tachycardia and Sudden Death

Circulation research, 2005-11, Vol.97 (11), p.1173-1181

2005

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Pathogenic mechanism of a catecholaminergic polymorphic ventricular tachycardia causing-mutation in cardiac calcium release channel RyR2

Journal of molecular and cellular cardiology, 2018-04, Vol.117, p.26-35

2018

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RyR2 Mutations Linked to Ventricular Tachycardia and Sudden Death Reduce the Threshold for Store-Overload-Induced Ca2+Release (SOICR)

Proceedings of the National Academy of Sciences - PNAS, 2004-08, Vol.101 (35), p.13062-13067

2004

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Phospholamban Knockout Breaks Arrhythmogenic Ca2+ Waves and Suppresses Catecholaminergic Polymorphic Ventricular Tachycardia in Mice

Circulation research, 2013-08, Vol.113 (5), p.517-526

2013

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Loss of luminal Ca²⁺ activation in the cardiac ryanodine receptor is associated with ventricular fibrillation and sudden death

Proceedings of the National Academy of Sciences - PNAS, 2007-11, Vol.104 (46), p.18309-18314

2007

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CPVT-associated cardiac ryanodine receptor mutation G357S with reduced penetrance impairs Ca2+ release termination and diminishes protein expression

PloS one, 2017-09, Vol.12 (9), p.e0184177-e0184177

2017

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Decreased occurrence of carbon cycle functions in microbial communities along with long-term secondary succession

Soil biology & biochemistry, 2018-08, Vol.123, p.207-217

2018

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Novel RyR2 Mutation (G3118R) Is Associated With Autosomal Recessive Ventricular Fibrillation and Sudden Death: Clinical, Functional, and Computational Analysis

Journal of the American Heart Association, 2021-03, Vol.10 (6), p.e017128-e017128

2021

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A Novel RYR2 Loss-of-Function Mutation (I4855M) is Associated with Left Ventricular Non-compaction and Atypical Catecholaminergic Polymorphic Ventricular Tachycardia

Journal of electrocardiology, 2017-03, Vol.50 (2), p.227-233

2017

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On the methodology of Darwinism and Non-Darwinism

Sheng wu duo yang xing, 2022, Vol.30 (9), p.22482

Wang, Ruiwu

2022

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Structural basis for the gating mechanism of the type 2 ryanodine receptor RyR2

Modulation of cardiac ryanodine receptor 2 by calmodulin

Popularity enhances the interdependent network reciprocity

Elevated expression of immune and DNA repair genes in mated queens and kings of the Reticulitermes chinensis termites

Differential microbial assembly processes and co‐occurrence networks in the soil‐root continuum along an environmental gradient

Cardiac ryanodine receptor calcium release deficiency syndrome

The ryanodine receptor store-sensing gate controls Ca2+ waves and Ca2+-triggered arrhythmias

Abnormal Termination of Ca2+ Release Is a Common Defect of RyR2 Mutations Associated With Cardiomyopathies

Limiting RyR2 Open Time Prevents Alzheimer’s Disease-Related Neuronal Hyperactivity and Memory Loss but Not β-Amyloid Accumulation

Arrhythmogenic Calmodulin Mutations Affect the Activation and Termination of Cardiac Ryanodine Receptor-mediated Ca2+ Release

Carvedilol and its new analogs suppress arrhythmogenic store overload-induced Ca2+ release

Disease-Induced Cooperation Mitigates Populations Against Decline: The Cascade Effect of Cooperation Evolution

Infanticide vs. inherited cardiac arrhythmias

Enhanced Cytosolic Ca2+ Activation Underlies a Common Defect of Central Domain Cardiac Ryanodine Receptor Mutations Linked to Arrhythmias

De novo ITPR1 variants are a recurrent cause of early-onset ataxia, acting via loss of channel function

Enhanced Store Overload–Induced Ca2+ Release and Channel Sensitivity to Luminal Ca2+ Activation Are Common Defects of RyR2 Mutations Linked to Ventricular Tachycardia and Sudden Death

Pathogenic mechanism of a catecholaminergic polymorphic ventricular tachycardia causing-mutation in cardiac calcium release channel RyR2

RyR2 Mutations Linked to Ventricular Tachycardia and Sudden Death Reduce the Threshold for Store-Overload-Induced Ca2+Release (SOICR)

Phospholamban Knockout Breaks Arrhythmogenic Ca2+ Waves and Suppresses Catecholaminergic Polymorphic Ventricular Tachycardia in Mice

Loss of luminal Ca²⁺ activation in the cardiac ryanodine receptor is associated with ventricular fibrillation and sudden death

CPVT-associated cardiac ryanodine receptor mutation G357S with reduced penetrance impairs Ca2+ release termination and diminishes protein expression

Decreased occurrence of carbon cycle functions in microbial communities along with long-term secondary succession

Novel RyR2 Mutation (G3118R) Is Associated With Autosomal Recessive Ventricular Fibrillation and Sudden Death: Clinical, Functional, and Computational Analysis

A Novel RYR2 Loss-of-Function Mutation (I4855M) is Associated with Left Ventricular Non-compaction and Atypical Catecholaminergic Polymorphic Ventricular Tachycardia

On the methodology of Darwinism and Non-Darwinism

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