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Protection against Fas-dependent Th1-mediated apoptosis by antigen receptor engagement in B cells

Nature (London), 1995-03, Vol.374 (6518), p.163-165

1995

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Fas ligand-mediated cytotoxicity is directly responsible for apoptosis of normal CD4+ T cells responding to a bacterial superantigen

The Journal of immunology (1950), 1995-05, Vol.154 (9), p.4302-4308

1995

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Protection of T cells from activation-induced cell death by Fas super(+) B cells

European journal of immunology, 2000-03, Vol.30 (3), p.931-937

2000

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Protection of T cells from activation‐induced cell death by Fas+ B cells

European journal of immunology, 2000-03, Vol.30 (3), p.931-937

2000

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Intracellular signaling for inducible antigen receptor-mediated Fas resistance in B cells

The Journal of immunology (1950), 1996-09, Vol.157 (5), p.1878-1885

1996

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Fas (CD95)/Fas ligand interactions regulate antigen‐specific, major histocompatibility complex‐restricted T/B cell proliferative responses

European journal of immunology, 1996-02, Vol.26 (2), p.415-419

1996

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CD4+T Cells Reactivated with Superantigen Are both More Sensitive to FasL-Mediated Killing and Express a Higher Level of FasL

Cellular immunology, 1997-08, Vol.179 (2), p.153-164

1997

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Functional distinctions between MRL-lpr and MRL-gld lymphocytes. Normal cells reverse the gld but not lpr immunoregulatory defect

The Journal of immunology (1950), 1994-02, Vol.152 (4), p.1557-1568

1994

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CD4 super(+) T cells reactivated with superantigen are both more sensitive to FasL-mediated killing and express a higher level of FasL

Cellular immunology, 1997-08, Vol.179 (2), p.153-164

1997

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Mesenchymal-epithelial interactions between normal and transformed human bladder cells

Cancer research (Chicago, Ill.), 1989-05, Vol.49 (10), p.2750-2754

1989

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EEG pre-burst suppression: characterization and inverse association with preoperative cognitive function in older adults

Frontiers in aging neuroscience, 2023-08, Vol.15, p.1229081

2023

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Expression and functional characterization of gfpmut3.1 and its unstable variants in Staphylococcus epidermidis

Journal of microbiological methods, 2007-11, Vol.71 (2), p.123-132

2007

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The structure of bovine rhodopsin

Biophysics of Structure and Mechanism, 1983-01, Vol.9 (4), p.235-244

1983

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Protection against Fas-dependent Th1-mediated apoptosis by antigen receptor engagement in B cells

Fas ligand-mediated cytotoxicity is directly responsible for apoptosis of normal CD4+ T cells responding to a bacterial superantigen

Protection of T cells from activation-induced cell death by Fas super(+) B cells

Protection of T cells from activation‐induced cell death by Fas+ B cells

Intracellular signaling for inducible antigen receptor-mediated Fas resistance in B cells

Fas (CD95)/Fas ligand interactions regulate antigen‐specific, major histocompatibility complex‐restricted T/B cell proliferative responses

CD4+T Cells Reactivated with Superantigen Are both More Sensitive to FasL-Mediated Killing and Express a Higher Level of FasL

Functional distinctions between MRL-lpr and MRL-gld lymphocytes. Normal cells reverse the gld but not lpr immunoregulatory defect

CD4 super(+) T cells reactivated with superantigen are both more sensitive to FasL-mediated killing and express a higher level of FasL

Mesenchymal-epithelial interactions between normal and transformed human bladder cells

EEG pre-burst suppression: characterization and inverse association with preoperative cognitive function in older adults

Expression and functional characterization of gfpmut3.1 and its unstable variants in Staphylococcus epidermidis

The structure of bovine rhodopsin

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