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Deconstructing GSK-3 : The Fine Regulation of Its Activity

International journal of alzheimer's disease, 2011, Vol.2011 (2011), p.1-12

2011

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Neuronal and glial purinergic receptors functions in neuron development and brain disease

Frontiers in cellular neuroscience, 2013-10, Vol.7, p.197-197

2013

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Amyloid-β impairs mitochondrial dynamics and autophagy in Alzheimer’s disease experimental models

Scientific reports, 2022-06, Vol.12 (1), p.10092-15, Article 10092

2022

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Editorial: Neuroprotection in Synaptic Signalling During Neurological Disorders

Frontiers in synaptic neuroscience, 2021-09, Vol.13, p.746487-746487

2021

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Sex steroid hormones as neuroprotective elements in ischemia models

Journal of endocrinology, 2018-05, Vol.237 (2), p.R65-R81

2018

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R-Ras GTPases Signaling Role in Myelin Neurodegenerative Diseases

International journal of molecular sciences, 2020-08, Vol.21 (16), p.5911

2020

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Specific roles of Akt iso forms in apoptosis and axon growth regulation in neurons

PloS one, 2012-04, Vol.7 (4), p.e32715-e32715

2012

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Modulation of GSK-3 as a Therapeutic Strategy on Tau Pathologies

Frontiers in molecular neuroscience, 2011-01, Vol.4, p.24-24

2011

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Stroke and Neuroinflamation: Role of Sexual Hormones

Current pharmaceutical design, 2016-03, Vol.22 (10), p.1334-1349

2016

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Nanoliposomes as a Therapeutic Tool for Alzheimer’s Disease

Frontiers in synaptic neuroscience, 2020-05, Vol.12, p.20

2020

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Role of mTORC1 Controlling Proteostasis after Brain Ischemia

Frontiers in neuroscience, 2018-02, Vol.12, p.60-60

2018

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Pathways Involved in Remyelination after Cerebral Ischemia

Current neuropharmacology, 2022-01, Vol.20 (4), p.751-765

2022

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Diets with Higher ω-6/ω-3 Ratios Show Differences in Ceramides and Fatty Acid Levels Accompanied by Increased Amyloid-Beta in the Brains of Male APP/PS1 Transgenic Mice

International journal of molecular sciences, 2021-10, Vol.22 (20), p.10907

2021

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Author Correction: Amyloid-β impairs mitochondrial dynamics and autophagy in Alzheimer’s disease experimental models

Scientific reports, 2023-11, Vol.13 (1), p.19303-19303, Article 19303

2023

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Cancer Stem Cell‐Like Phenotype and Survival Are Coordinately Regulated by Akt/FoxO/Bim Pathway

Stem cells (Dayton, Ohio), 2015-03, Vol.33 (3), p.646-660

2015

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Impaired function of HDAC6 slows down axonal growth and interferes with axon initial segment development

PloS one, 2010-09, Vol.5 (9), p.e12908-e12908

2010

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WIP Drives Tumor Progression through YAP/TAZ-Dependent Autonomous Cell Growth

Cell reports (Cambridge), 2016-11, Vol.17 (8), p.1962-1977

2016

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Altered Clock Gene Expression in Female APP/PS1 Mice and Aquaporin-Dependent Amyloid Accumulation in the Retina

International journal of molecular sciences, 2023-11, Vol.24 (21), p.15679

2023

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R-Ras1 and R-Ras2 Expression in Anatomical Regions and Cell Types of the Central Nervous System

International journal of molecular sciences, 2022-01, Vol.23 (2), p.978

2022

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Repeated intraperitoneal injections of liposomes containing phosphatidic acid and cardiolipin reduce amyloid-β levels in APP/PS1 transgenic mice

Nanomedicine, 2015-02, Vol.11 (2), p.421-430

2015

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Pharmacological inhibition of mTORC1 reduces neural death and damage volume after MCAO by modulating microglial reactivity

Biology direct, 2024-04, Vol.19 (1), p.26-26, Article 26

2024

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WIP, YAP/TAZ and Actin Connections Orchestrate Development and Transformation in the Central Nervous System

Frontiers in cell and developmental biology, 2021-06, Vol.9, p.673986-673986

2021

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Editorial: Neuroprotection in Brain Hypoxia

Frontiers in neuroscience, 2019-03, Vol.13, p.212-212

2019

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Energy-Sensing Pathways in Ischemia: The Counterbalance Between AMPK and mTORC

Current pharmaceutical design, 2019-01, Vol.25 (45), p.4763-4770

2019

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Trehalose Reduces the Secreted Beta-Amyloid Levels in Primary Neurons Independently of Autophagy Induction

Metabolites, 2021-06, Vol.11 (7), p.421

2021

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Deconstructing GSK-3 : The Fine Regulation of Its Activity

Neuronal and glial purinergic receptors functions in neuron development and brain disease

Amyloid-β impairs mitochondrial dynamics and autophagy in Alzheimer’s disease experimental models

Editorial: Neuroprotection in Synaptic Signalling During Neurological Disorders

Sex steroid hormones as neuroprotective elements in ischemia models

R-Ras GTPases Signaling Role in Myelin Neurodegenerative Diseases

Specific roles of Akt iso forms in apoptosis and axon growth regulation in neurons

Modulation of GSK-3 as a Therapeutic Strategy on Tau Pathologies

Stroke and Neuroinflamation: Role of Sexual Hormones

Nanoliposomes as a Therapeutic Tool for Alzheimer’s Disease

Role of mTORC1 Controlling Proteostasis after Brain Ischemia

Pathways Involved in Remyelination after Cerebral Ischemia

Diets with Higher ω-6/ω-3 Ratios Show Differences in Ceramides and Fatty Acid Levels Accompanied by Increased Amyloid-Beta in the Brains of Male APP/PS1 Transgenic Mice

Author Correction: Amyloid-β impairs mitochondrial dynamics and autophagy in Alzheimer’s disease experimental models

Cancer Stem Cell‐Like Phenotype and Survival Are Coordinately Regulated by Akt/FoxO/Bim Pathway

Impaired function of HDAC6 slows down axonal growth and interferes with axon initial segment development

WIP Drives Tumor Progression through YAP/TAZ-Dependent Autonomous Cell Growth

Altered Clock Gene Expression in Female APP/PS1 Mice and Aquaporin-Dependent Amyloid Accumulation in the Retina

R-Ras1 and R-Ras2 Expression in Anatomical Regions and Cell Types of the Central Nervous System

Repeated intraperitoneal injections of liposomes containing phosphatidic acid and cardiolipin reduce amyloid-β levels in APP/PS1 transgenic mice

Pharmacological inhibition of mTORC1 reduces neural death and damage volume after MCAO by modulating microglial reactivity

WIP, YAP/TAZ and Actin Connections Orchestrate Development and Transformation in the Central Nervous System

Editorial: Neuroprotection in Brain Hypoxia

Energy-Sensing Pathways in Ischemia: The Counterbalance Between AMPK and mTORC

Trehalose Reduces the Secreted Beta-Amyloid Levels in Primary Neurons Independently of Autophagy Induction

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