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Activation of the Proapoptotic Unfolded Protein Response in Plaques of the Human Carotid Artery

European journal of vascular and endovascular surgery, 2014-09, Vol.48 (3), p.248-257

2014

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Activation of the Proapoptotic Unfolded Protein Response in Plaques of the Human Carotid Artery

Journal of vascular surgery, 2014-09, Vol.60 (3), p.807-807

2014

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Inflammation and atherosclerosis – activation of proapoptotic stress-pathway "unfolded protein response" in plaques of the internal carotid artery

The Thoracic and cardiovascular surgeon, 2013, Vol.61 (S 01)

2013

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Surface-attached polymer monolayers for the control of endothelial cell adhesion

Colloids and surfaces. A, Physicochemical and engineering aspects, 2002-02, Vol.198, p.519-526

2002

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Ultrathin polymer monolayers for promotion of cell growth on bioprosthetic materials -- evolution of a new concept to improve long term performance of biologic heart vales

Bio-medical materials and engineering, 2004, Vol.14 (4), p.419

2004

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Mutant lamins cause nuclear envelope rupture and DNA damage in skeletal muscle cells

Nature materials, 2020-04, Vol.19 (4), p.464-473

2020

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Myopathic lamin mutations cause reductive stress and activate the nrf2/keap-1 pathway

PLoS genetics, 2015-05, Vol.11 (5), p.e1005231-e1005231

2015

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Myopathic lamin mutations impair nuclear stability in cells and tissue and disrupt nucleo-cytoskeletal coupling

Human molecular genetics, 2013-06, Vol.22 (12), p.2335-2349

2013

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Integration of MILP and discrete-event simulation for flow shop scheduling using Benders cuts

Computers & chemical engineering, 2024-10, Vol.189, p.108809, Article 108809

2024

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Mutant lamins cause nuclear envelope rupture and DNA damage in skeletal muscle cells

Nature materials, 2020-04, Vol.19 (4), p.464-473

2020

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Linking Heterochromatin Protein 1 (HP1) to cancer progression

Mutation research, 2008-12, Vol.647 (1), p.13-20

2008

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The Influence of a Genetic Variant in CCDC78 on LMNA -Associated Skeletal Muscle Disease

International journal of molecular sciences, 2024-05, Vol.25 (9), p.4930

2024

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Nuclear organization: taking a position on gene expression

Current opinion in cell biology, 2011-06, Vol.23 (3), p.354-359

2011

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Bottom‐up or top‐down?: The role of child and parent chronic pain and anxiety in the context of parental catastrophizing and solicitousness

European journal of pain, 2020-03, Vol.24 (3), p.604-616

2020

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In Silico and In Vivo Analysis of Amino Acid Substitutions That Cause Laminopathies

International journal of molecular sciences, 2021-10, Vol.22 (20), p.11226

2021

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Human neutrophil elastase induces endothelial cell apoptosis by activating the PERK‐CHOP branch of the unfolded protein response

The FASEB journal, 2017-09, Vol.31 (9), p.3868-3881

2017

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Connections between epigenetic gene silencing and human disease

Mutation Research-Fundamental and Molecular Mechanisms of Mutagenesis, 2007-05, Vol.618 (1), p.163-174

2007

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Motion Compensation in Six Degrees of Freedom for a MIMO Radar Mounted on a Hovering UAV

IEEE transactions on aerospace and electronic systems, 2023-10, Vol.59 (5), p.1-10

2023

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The role of Drosophila Lamin C in muscle function and gene expression

Development (Cambridge), 2010-09, Vol.137 (18), p.3067-3077

2010

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Cross talk between the cytoplasm and nucleus during development and disease

Current opinion in genetics & development, 2016-04, Vol.37, p.129-136

2016

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Shallow Thermal Anomalies and Their Role in the Breakup Evolution Along the Conjugate Margins of the Fram Strait (Svalbard and Eastern North Greenland), Indicated by Low‐Temperature Thermochronology

Geochemistry, geophysics, geosystems : G3, 2024-03, Vol.25 (3), p.n/a

2024

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Mechanisms of HP1-mediated gene silencing in Drosophila

Development (Cambridge), 2004-08, Vol.131 (15), p.3571-3580

2004

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Shining Light on the Dark Side of the Genome

Cells (Basel, Switzerland), 2022-01, Vol.11 (3), p.330

2022

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Comparative Study of Drosophila and Human A-Type Lamins

PloS one, 2009-10, Vol.4 (10), p.e7564

2009

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Nuclear damage in LMNA mutant iPSC-derived cardiomyocytes is associated with impaired lamin localization to the nuclear envelope

Molecular biology of the cell, 2023-11, Vol.34 (12), p.mbcE21100527

2023

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Activation of the Proapoptotic Unfolded Protein Response in Plaques of the Human Carotid Artery

Activation of the Proapoptotic Unfolded Protein Response in Plaques of the Human Carotid Artery

Inflammation and atherosclerosis – activation of proapoptotic stress-pathway "unfolded protein response" in plaques of the internal carotid artery

Surface-attached polymer monolayers for the control of endothelial cell adhesion

Ultrathin polymer monolayers for promotion of cell growth on bioprosthetic materials -- evolution of a new concept to improve long term performance of biologic heart vales

Mutant lamins cause nuclear envelope rupture and DNA damage in skeletal muscle cells

Myopathic lamin mutations cause reductive stress and activate the nrf2/keap-1 pathway

Myopathic lamin mutations impair nuclear stability in cells and tissue and disrupt nucleo-cytoskeletal coupling

Integration of MILP and discrete-event simulation for flow shop scheduling using Benders cuts

Mutant lamins cause nuclear envelope rupture and DNA damage in skeletal muscle cells

Linking Heterochromatin Protein 1 (HP1) to cancer progression

The Influence of a Genetic Variant in CCDC78 on LMNA -Associated Skeletal Muscle Disease

Nuclear organization: taking a position on gene expression

Bottom‐up or top‐down?: The role of child and parent chronic pain and anxiety in the context of parental catastrophizing and solicitousness

In Silico and In Vivo Analysis of Amino Acid Substitutions That Cause Laminopathies

Human neutrophil elastase induces endothelial cell apoptosis by activating the PERK‐CHOP branch of the unfolded protein response

Connections between epigenetic gene silencing and human disease

Motion Compensation in Six Degrees of Freedom for a MIMO Radar Mounted on a Hovering UAV

The role of Drosophila Lamin C in muscle function and gene expression

Cross talk between the cytoplasm and nucleus during development and disease

Shallow Thermal Anomalies and Their Role in the Breakup Evolution Along the Conjugate Margins of the Fram Strait (Svalbard and Eastern North Greenland), Indicated by Low‐Temperature Thermochronology

Mechanisms of HP1-mediated gene silencing in Drosophila

Shining Light on the Dark Side of the Genome

Comparative Study of Drosophila and Human A-Type Lamins

Nuclear damage in LMNA mutant iPSC-derived cardiomyocytes is associated with impaired lamin localization to the nuclear envelope

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