UB Paderborn / Katalog / Suche / in CDI

1 – 25 von 683

Sortiert nach

Chromatin crosstalk in development and disease: lessons from REST

Nature reviews. Genetics, 2007-07, Vol.8 (7), p.544-554

2007

Link zum Volltext

Verfügbarkeit prüfen

The Contribution and Therapeutic Potential of Epigenetic Modifications in Alzheimer's Disease

Frontiers in neuroscience, 2018-09, Vol.12, p.649-649

Wood, Ian C

2018

Link zum Volltext

Verfügbarkeit prüfen

Transcriptional repression of the M channel subunit Kv7.2 in chronic nerve injury

Pain (Amsterdam), 2011-04, Vol.152 (4), p.742-754

2011

Link zum Volltext

Verfügbarkeit prüfen

Regulation of gene expression in the nervous system

Biochemical journal, 2008-09, Vol.414 (3), p.327-341

2008

Link zum Volltext

Verfügbarkeit prüfen

Genome-Wide Analysis of Repressor Element 1 Silencing Transcription Factor/Neuron-Restrictive Silencing Factor (REST/NRSF) Target Genes

Proceedings of the National Academy of Sciences - PNAS, 2004-07, Vol.101 (28), p.10458-10463

2004

Link zum Volltext

Verfügbarkeit prüfen

Epigenetic mechanisms in development and disease

Biochemical Society transactions, 2013-06, Vol.41 (3), p.697

2013

Link zum Volltext

Verfügbarkeit prüfen

BRG1 Chromatin Remodeling Activity Is Required for Efficient Chromatin Binding by Repressor Element 1-silencing Transcription Factor (REST) and Facilitates REST-mediated Repression

The Journal of biological chemistry, 2006-12, Vol.281 (51), p.38974-38980

2006

Link zum Volltext

Verfügbarkeit prüfen

Inhibition of histone deacetylase 1 or 2 reduces induced cytokine expression in microglia through a protein synthesis independent mechanism

Journal of neurochemistry, 2017-10, Vol.143 (2), p.214-224

2017

Link zum Volltext

Verfügbarkeit prüfen

Efficient infection of organotypic hippocampal slice cultures with adenovirus carrying the transgene REST/NRSF

Journal of neuroscience methods, 2021-05, Vol.356, p.109147-109147, Article 109147

2021

Link zum Volltext

Verfügbarkeit prüfen

Histone deacetylase inhibitors induce medulloblastoma cell death independent of HDACs recruited in REST repression complexes

Molecular genetics & genomic medicine, 2020-10, Vol.8 (10), p.e1429-n/a

2020

Link zum Volltext

Verfügbarkeit prüfen

Downregulated REST Transcription Factor Is a Switch Enabling Critical Potassium Channel Expression and Cell Proliferation

Molecular cell, 2005-10, Vol.20 (1), p.45-52

2005

Link zum Volltext

Verfügbarkeit prüfen

The functions of repressor element 1-silencing transcription factor in models of epileptogenesis and post-ischemia

Metabolic brain disease, 2021-08, Vol.36 (6), p.1135-1150

2021

Link zum Volltext

Verfügbarkeit prüfen

The Repressor Element 1-Silencing Transcription Factor Regulates Heart-Specific Gene Expression Using Multiple Chromatin-Modifying Complexes

Molecular and Cellular Biology, 2007-06, Vol.27 (11), p.4082-4092

2007

Link zum Volltext

Verfügbarkeit prüfen

Repressor element 1-silencing transcription factor drives the development of chronic pain states

Pain (Amsterdam), 2019-10, Vol.160 (10), p.2398-2408

2019

Link zum Volltext

Verfügbarkeit prüfen

Hypoxia-inducible Factor-1α (HIF1α) Switches on Transient Receptor Potential Ankyrin Repeat 1 (TRPA1) Gene Expression via a Hypoxia Response Element-like Motif to Modulate Cytokine Release

The Journal of biological chemistry, 2012-09, Vol.287 (38), p.31962-31972

2012

Link zum Volltext

Verfügbarkeit prüfen

Blocking of NF‑kB/p38 MAPK pathways mitigates oligodendrocyte pathology in a model of neonatal white matter injury

Acta neurobiologiae experimentalis, 2022-01, Vol.82 (1), p.52-64

2022

Link zum Volltext

Verfügbarkeit prüfen

Alzheimer’s disease: the potential of epigenetic treatments and current clinical candidates

Neurodegenerative disease management, 2020-06, Vol.10 (3), p.543-558, Article nmt-2019-0034

2020

Link zum Volltext

Verfügbarkeit prüfen

From beads on a string to the pearls of regulation: the structure and dynamics of chromatin

Biochemical Society transactions, 2012-04, Vol.40 (2), p.331

2012

Link zum Volltext

Verfügbarkeit prüfen

Chromatin switching and transcriptional regulation in disease

Biochemical Society transactions, 2008-08, Vol.36 (Pt 4), p.599

2008

Link zum Volltext

Verfügbarkeit prüfen

Gene expression in neuronal disease

Biochemical Society transactions, 2009-12, Vol.37 (Pt 6), p.1261

2009

Link zum Volltext

Verfügbarkeit prüfen

Transcriptional control of KCNQ channel genes and the regulation of neuronal excitability

The Journal of neuroscience, 2010-10, Vol.30 (40), p.13235-13245

2010

Link zum Volltext

Verfügbarkeit prüfen

High levels of HDAC expression correlate with microglial aging

Expert opinion on therapeutic targets, 2022-10, Vol.26 (10), p.911-922

2022

Link zum Volltext

Verfügbarkeit prüfen

Sharp Wave-Like Activity in the Hippocampus In Vitro in Mice Lacking the Gap Junction Protein Connexin 36

Journal of neurophysiology, 2003-04, Vol.89 (4), p.2046-2054

2003

Link zum Volltext

Verfügbarkeit prüfen

Inhibition of ionotropic GluR signaling preserves oligodendrocyte lineage and myelination in an ex vivo rat model of white matter ischemic injury

Acta neurobiologiae experimentalis, 2021-01, Vol.81 (3), p.233-248

2021

Link zum Volltext

Verfügbarkeit prüfen

MicroRNA‐21 drives the switch to a synthetic phenotype in human saphenous vein smooth muscle cells

IUBMB life, 2018-07, Vol.70 (7), p.649-657

2018

Link zum Volltext

Verfügbarkeit prüfen

1 – 25 von 683

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(622)
Mit Volltextzugriff
(593)

Publikationsform

3 weitere anzeigen...

Weniger anzeigen...

Sprache

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 30 Tagen
(1)
... seit 90 Tagen
(9)

Menü

Weitere Dienste

Einstellungen

Chromatin crosstalk in development and disease: lessons from REST

The Contribution and Therapeutic Potential of Epigenetic Modifications in Alzheimer's Disease

Transcriptional repression of the M channel subunit Kv7.2 in chronic nerve injury

Regulation of gene expression in the nervous system

Genome-Wide Analysis of Repressor Element 1 Silencing Transcription Factor/Neuron-Restrictive Silencing Factor (REST/NRSF) Target Genes

Epigenetic mechanisms in development and disease

BRG1 Chromatin Remodeling Activity Is Required for Efficient Chromatin Binding by Repressor Element 1-silencing Transcription Factor (REST) and Facilitates REST-mediated Repression

Inhibition of histone deacetylase 1 or 2 reduces induced cytokine expression in microglia through a protein synthesis independent mechanism

Efficient infection of organotypic hippocampal slice cultures with adenovirus carrying the transgene REST/NRSF

Histone deacetylase inhibitors induce medulloblastoma cell death independent of HDACs recruited in REST repression complexes

Downregulated REST Transcription Factor Is a Switch Enabling Critical Potassium Channel Expression and Cell Proliferation

The functions of repressor element 1-silencing transcription factor in models of epileptogenesis and post-ischemia

The Repressor Element 1-Silencing Transcription Factor Regulates Heart-Specific Gene Expression Using Multiple Chromatin-Modifying Complexes

Repressor element 1-silencing transcription factor drives the development of chronic pain states

Hypoxia-inducible Factor-1α (HIF1α) Switches on Transient Receptor Potential Ankyrin Repeat 1 (TRPA1) Gene Expression via a Hypoxia Response Element-like Motif to Modulate Cytokine Release

Blocking of NF‑kB/p38 MAPK pathways mitigates oligodendrocyte pathology in a model of neonatal white matter injury

Alzheimer’s disease: the potential of epigenetic treatments and current clinical candidates

From beads on a string to the pearls of regulation: the structure and dynamics of chromatin

Chromatin switching and transcriptional regulation in disease

Gene expression in neuronal disease

Transcriptional control of KCNQ channel genes and the regulation of neuronal excitability

High levels of HDAC expression correlate with microglial aging

Sharp Wave-Like Activity in the Hippocampus In Vitro in Mice Lacking the Gap Junction Protein Connexin 36

Inhibition of ionotropic GluR signaling preserves oligodendrocyte lineage and myelination in an ex vivo rat model of white matter ischemic injury

MicroRNA‐21 drives the switch to a synthetic phenotype in human saphenous vein smooth muscle cells

Suchergebnisse filtern