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Identification of PKC-isoform-specific biological actions using pharmacological approaches

Trends in Pharmacological Sciences, 2000-05, Vol.21 (5), p.181-187

2000

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Activation of Vascular Protein Kinase C-β Inhibits Akt-Dependent Endothelial Nitric Oxide Synthase Function in Obesity-Associated Insulin Resistance

Diabetes (New York, N.Y.), 2006-03, Vol.55 (3), p.691-698

2006

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The generation and properties of human macrophage populations from hemopoietic stem cells

Journal of leukocyte biology, 2009-05, Vol.85 (5), p.766-778

2009

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Expression of Connective Tissue Growth Factor Is Increased in Injured Myocardium Associated With Protein Kinase C β2 Activation and Diabetes

Diabetes (New York, N.Y.), 2002-09, Vol.51 (9), p.2709-2718

2002

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Differential Regulation of Angiotensin II-induced Expression of Connective Tissue Growth Factor by Protein Kinase C Isoforms in the Myocardium

The Journal of biological chemistry, 2005-04, Vol.280 (16), p.15719-15726

2005

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Decreased cardiac expression of vascular endothelial growth factor and its receptors in insulin-resistant and diabetic states: A possible explanation for impaired collateral formation in cardiac tissue

Circulation (New York, N.Y.), 2002-01, Vol.105 (3), p.373-379

2002

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The effect of tissue type-plasminogen activator deletion and associated fibrin(ogen) deposition on macrophage localization in peritoneal inflammation

Thrombosis and haemostasis, 2006-04, Vol.95 (4), p.659

2006

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Urokinase-type plasminogen activator and arthritis progression: role in systemic disease with immune complex involvement

Arthritis research & therapy, 2010-01, Vol.12 (2), p.R37-R37

2010

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Applications of inhibitors for protein kinase C and their isoforms

Methods in molecular biology (Clifton, N.J.), 2003, Vol.233, p.397

2003

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The effects of diabetes on nitric oxide-mediated responses in rat corpus cavernosum

European journal of pharmacology, 1999-07, Vol.376 (1), p.73-82

1999

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The aldose reductase inhibitor sorbinil does not prevent the impairment in nitric oxide-mediated neurotransmission in anococcygeus muscle from diabetic rats

European journal of pharmacology, 1996-12, Vol.318 (1), p.101-108

1996

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Insulin Reversal of Impaired Nitrergic Transmission in the Anococcygeus Muscle from Diabetic Rats

Pharmacology, 1999-09, Vol.59 (3), p.115-126

1999

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Decreased cardiac expression of vascular endothelial growth factor and its receptors in insulin-resistant and diabetic states: A possible explanation for impaired

Circulation (New York, N.Y.), 2002-01, Vol.105 (3), p.373

2002

Volltextzugriff (PDF)

Activation of vascular protein kinase C-[beta] inhibits Akt-dependent endothelial nitric oxide synthase function in obesity-associated insulin resistance

Diabetes (New York, N.Y.), 2006-03, Vol.55 (3), p.691

2006

Volltextzugriff (PDF)

Expression of connective tissue growth factor is increased in injured myocardium associated with protein kinase C beta2 activation and diabetes

Diabetes (New York, N.Y.), 2002-09, Vol.51 (9), p.2709-2718

2002

Volltextzugriff (PDF)

Effect of diabetes and elevated glucose on nitric oxide‐mediated neurotransmission in rat anococcygeus muscle

British journal of pharmacology, 1995-06, Vol.115 (3), p.409-414

1995

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Diabetes does not alter the activity and localisation of nitric oxide synthase in the rat anococcygeus muscle

Journal of the autonomic nervous system, 1999-04, Vol.76 (1), p.35-44

1999

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Inhibition of protein kinase C reduces left ventricular fibrosis and dysfunction following myocardial infarction

Journal of molecular and cellular cardiology, 2005-08, Vol.39 (2), p.213-221

2005

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Additive effects of endothelial progenitor cells combined with ACE inhibition and β-blockade on left ventricular function following acute myocardial infarction

Journal of the renin-angiotensin-aldosterone system, 2005-03, Vol.6 (1), p.33-37

2005

Volltextzugriff (PDF)

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Identification of PKC-isoform-specific biological actions using pharmacological approaches

Activation of Vascular Protein Kinase C-β Inhibits Akt-Dependent Endothelial Nitric Oxide Synthase Function in Obesity-Associated Insulin Resistance

The generation and properties of human macrophage populations from hemopoietic stem cells

Expression of Connective Tissue Growth Factor Is Increased in Injured Myocardium Associated With Protein Kinase C β2 Activation and Diabetes

Differential Regulation of Angiotensin II-induced Expression of Connective Tissue Growth Factor by Protein Kinase C Isoforms in the Myocardium

Decreased cardiac expression of vascular endothelial growth factor and its receptors in insulin-resistant and diabetic states: A possible explanation for impaired collateral formation in cardiac tissue

The effect of tissue type-plasminogen activator deletion and associated fibrin(ogen) deposition on macrophage localization in peritoneal inflammation

Urokinase-type plasminogen activator and arthritis progression: role in systemic disease with immune complex involvement

Applications of inhibitors for protein kinase C and their isoforms

The effects of diabetes on nitric oxide-mediated responses in rat corpus cavernosum

The aldose reductase inhibitor sorbinil does not prevent the impairment in nitric oxide-mediated neurotransmission in anococcygeus muscle from diabetic rats

Insulin Reversal of Impaired Nitrergic Transmission in the Anococcygeus Muscle from Diabetic Rats

Decreased cardiac expression of vascular endothelial growth factor and its receptors in insulin-resistant and diabetic states: A possible explanation for impaired

Activation of vascular protein kinase C-[beta] inhibits Akt-dependent endothelial nitric oxide synthase function in obesity-associated insulin resistance

Expression of connective tissue growth factor is increased in injured myocardium associated with protein kinase C beta2 activation and diabetes

Effect of diabetes and elevated glucose on nitric oxide‐mediated neurotransmission in rat anococcygeus muscle

Diabetes does not alter the activity and localisation of nitric oxide synthase in the rat anococcygeus muscle

Inhibition of protein kinase C reduces left ventricular fibrosis and dysfunction following myocardial infarction

Additive effects of endothelial progenitor cells combined with ACE inhibition and β-blockade on left ventricular function following acute myocardial infarction

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