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We're Not "DON" Yet: Optimal Dosing and Prodrug Delivery of 6-Diazo-5-oxo-L-norleucine

Molecular cancer therapeutics, 2018-09, Vol.17 (9), p.1824-1832

2018

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Selective inhibition of NAALADase, which converts NAAG to glutamate, reduces ischemic brain injury

Nature medicine, 1999-12, Vol.5 (12), p.1396-1402

1999

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The orally active glutamate carboxypeptidase II inhibitor E2072 exhibits sustained nerve exposure and attenuates peripheral neuropathy

The Journal of pharmacology and experimental therapeutics, 2012-12, Vol.343 (3), p.746

2012

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Looking for Drugs in All the Wrong Places: Use of GCPII Inhibitors Outside the Brain

Neurochemical research, 2020-06, Vol.45 (6), p.1256-1267

2020

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Still NAAG'ing After All These Years: The Continuing Pursuit of GCPII Inhibitors

Advances in pharmacology (1990), 2016, Vol.76, p.215-255

2016

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Sustained Accumulation of Microtubule-Binding Chemotherapy Drugs in the Peripheral Nervous System: Correlations with Time Course and Neurotoxic Severity

Cancer research (Chicago, Ill.), 2016-06, Vol.76 (11), p.3332-3339

2016

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Preclinical pharmacology and pharmacokinetics of CERC‐301, a GluN2B‐selective N‐methyl‐D‐aspartate receptor antagonist

Pharmacology research & perspectives, 2015-12, Vol.3 (6), p.e00198-n/a

2015

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FOLH1 /GCPII is elevated in IBD patients, and its inhibition ameliorates murine IBD abnormalities

JCI insight, 2016-08, Vol.1 (12)

2016

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Ghrelin agonist HM01 attenuates chemotherapy-induced neurotoxicity in rodent models

European journal of pharmacology, 2018-12, Vol.840, p.89-103

2018

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The central nervous system effects, pharmacokinetics and safety of the NAALADase‐inhibitor GPI 5693

British journal of clinical pharmacology, 2005-08, Vol.60 (2), p.128-136

2005

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Protective Effects of Extracellular Acidosis and Blockade of Sodium/Hydrogen Ion Exchange During Recovery from Metabolic Inhibition in Neuronal Tissue Culture

Journal of neurochemistry, 1996-12, Vol.67 (6), p.2379-2389

1996

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Pharmacokinetics and pharmacodynamics of the glutamate carboxypeptidase II inhibitor 2-MPPA show prolonged alleviation of neuropathic pain through an indirect mechanism

The Journal of pharmacology and experimental therapeutics, 2013-09, Vol.346 (3), p.406

2013

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Sedation with GPI 15715, a water-soluble prodrug of propofol, using target-controlled infusion in volunteers

Anesthesia and analgesia, 2005-03, Vol.100 (3), p.701-706

2005

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Regional Vulnerability to Endogenous and Exogenous Oxidative Stress in Organotypic Hippocampal Culture

Experimental neurology, 1998-01, Vol.149 (1), p.109-122

1998

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The regional vulnerability to hypoglycemia-induced neurotoxicity in organotypic hippocampal culture: protection by early tetrodotoxin or delayed MK-801

The Journal of neuroscience, 1992-11, Vol.12 (11), p.4298-4308

1992

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Gastrointestinal delivery of propofol from fospropofol: its bioavailability and activity in rodents and human volunteers

Journal of translational medicine, 2015-05, Vol.13 (1), p.170-170, Article 170

2015

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Direct observation of the agonist-specific regional vulnerability to glutamate, NMDA, and kainate neurotoxicity in organotypic hippocampal cultures

Experimental neurology, 1991-10, Vol.114 (1), p.11-22

1991

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Toxic NMDA‐Receptor Activation Occurs During Recovery in a Tissue Culture Model of Ischemia

Journal of neurochemistry, 1995-10, Vol.65 (4), p.1681-1691

Vornov, James J.

1995

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Pharmacokinetics and pharmacodynamics of the new propofol prodrug GPI 15715 in rats

European journal of anaesthesiology, 2003-03, Vol.20 (3), p.182-190

2003

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N-Acetylated alpha-linked acidic dipeptidase converts N-acetylaspartylglutamate from a neuroprotectant to a neurotoxin

The Journal of pharmacology and experimental therapeutics, 2000-10, Vol.295 (1), p.16

2000

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Neurotoxicity of Acute Glutamate Transport Blockade Depends on Coactivation of Both NMDA and AMPA/Kainate Receptors in Organotypic Hippocampal Cultures

Experimental neurology, 1995-05, Vol.133 (1), p.7-17

1995

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Comparative pharmacokinetics and pharmacodynamics of the new propofol prodrug GPI 15715 and propofol emulsion

Anesthesiology (Philadelphia), 2004-09, Vol.101 (3), p.626-639

2004

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Toxicity induced by a polyglutamated folate analog is attenuated by NAALADase inhibition

Brain research, 1999-10, Vol.843 (1), p.48-52

1999

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Pharmacokinetics and clinical pharmacodynamics of the new propofol prodrug GPI 15715 in volunteers

Anesthesiology (Philadelphia), 2003-08, Vol.99 (2), p.303-313

2003

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Ion channels and exchangers that mediate ischemic neuronal injury

Current opinion in neurology, 1998-02, Vol.11 (1), p.39-43

Vornov, J J

1998

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We're Not "DON" Yet: Optimal Dosing and Prodrug Delivery of 6-Diazo-5-oxo-L-norleucine

Selective inhibition of NAALADase, which converts NAAG to glutamate, reduces ischemic brain injury

The orally active glutamate carboxypeptidase II inhibitor E2072 exhibits sustained nerve exposure and attenuates peripheral neuropathy

Looking for Drugs in All the Wrong Places: Use of GCPII Inhibitors Outside the Brain

Still NAAG'ing After All These Years: The Continuing Pursuit of GCPII Inhibitors

Sustained Accumulation of Microtubule-Binding Chemotherapy Drugs in the Peripheral Nervous System: Correlations with Time Course and Neurotoxic Severity

Preclinical pharmacology and pharmacokinetics of CERC‐301, a GluN2B‐selective N‐methyl‐D‐aspartate receptor antagonist

FOLH1 /GCPII is elevated in IBD patients, and its inhibition ameliorates murine IBD abnormalities

Ghrelin agonist HM01 attenuates chemotherapy-induced neurotoxicity in rodent models

The central nervous system effects, pharmacokinetics and safety of the NAALADase‐inhibitor GPI 5693

Protective Effects of Extracellular Acidosis and Blockade of Sodium/Hydrogen Ion Exchange During Recovery from Metabolic Inhibition in Neuronal Tissue Culture

Pharmacokinetics and pharmacodynamics of the glutamate carboxypeptidase II inhibitor 2-MPPA show prolonged alleviation of neuropathic pain through an indirect mechanism

Sedation with GPI 15715, a water-soluble prodrug of propofol, using target-controlled infusion in volunteers

Regional Vulnerability to Endogenous and Exogenous Oxidative Stress in Organotypic Hippocampal Culture

The regional vulnerability to hypoglycemia-induced neurotoxicity in organotypic hippocampal culture: protection by early tetrodotoxin or delayed MK-801

Gastrointestinal delivery of propofol from fospropofol: its bioavailability and activity in rodents and human volunteers

Direct observation of the agonist-specific regional vulnerability to glutamate, NMDA, and kainate neurotoxicity in organotypic hippocampal cultures

Toxic NMDA‐Receptor Activation Occurs During Recovery in a Tissue Culture Model of Ischemia

Pharmacokinetics and pharmacodynamics of the new propofol prodrug GPI 15715 in rats

N-Acetylated alpha-linked acidic dipeptidase converts N-acetylaspartylglutamate from a neuroprotectant to a neurotoxin

Neurotoxicity of Acute Glutamate Transport Blockade Depends on Coactivation of Both NMDA and AMPA/Kainate Receptors in Organotypic Hippocampal Cultures

Comparative pharmacokinetics and pharmacodynamics of the new propofol prodrug GPI 15715 and propofol emulsion

Toxicity induced by a polyglutamated folate analog is attenuated by NAALADase inhibition

Pharmacokinetics and clinical pharmacodynamics of the new propofol prodrug GPI 15715 in volunteers

Ion channels and exchangers that mediate ischemic neuronal injury

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