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Evidence of in vivo basophil activation in chronic idiopathic urticaria

Clinical and experimental allergy, 2006-06, Vol.36 (6), p.770-776

2006

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Differential expression of RNA-binding proteins in bronchial epithelium of stable COPD patients

International journal of chronic obstructive pulmonary disease, 2018-01, Vol.13, p.3173-3190

2018

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New concepts in chronic urticaria

Current opinion in immunology, 2008-12, Vol.20 (6), p.709-716

2008

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Src homology 2 domain–containing inositol 5' phosphatase is negatively associated with histamine release to human recombinant histamine-releasing factor in human basophils

Journal of allergy and clinical immunology, 2001-11, Vol.108 (5), p.822-831

2001

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The Unique Domain as the Site on Lyn Kinase for Its Constitutive Association with the High Affinity Receptor for IgE

The Journal of biological chemistry, 1997-09, Vol.272 (38), p.24072-24080

1997

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Inhibition of Cytokine Gene Transcription by the Human Recombinant Histamine-Releasing Factor in Human T Lymphocytes

The Journal of immunology (1950), 2003-10, Vol.171 (7), p.3742-3750

2003

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Basophil FcεRI histamine release parallels expression of Src-homology 2–containing inositol phosphatases in chronic idiopathic urticaria

Journal of Allergy and Clinical Immunology, 2007-02, Vol.119 (2), p.441-448

2007

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Interaction between the Unphosphorylated Receptor with High Affinity for IgE and Lyn Kinase

The Journal of biological chemistry, 2001-01, Vol.276 (2), p.1041-1050

2001

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Cultured peripheral blood mast cells from chronic idiopathic urticaria patients spontaneously degranulate upon IgE sensitization: Relationship to expression of Syk and SHIP-2

Clinical immunology (Orlando, Fla.), 2009-09, Vol.132 (3), p.342-348

2009

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One Lyn Molecule Is Sufficient to Initiate Phosphorylation of Aggregated High-Affinity IgE Receptors

Proceedings of the National Academy of Sciences - PNAS, 1999-07, Vol.96 (15), p.8615-8620

1999

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Simultaneous determination of hydroxyeicosanoid (HETE) binding to cells and its cellular metabolism

Journal of lipid research, 1993-05, Vol.34 (5), p.853-858

1993

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Association of the Src homology 2 domain-containing inositol 5′ phosphatase (SHIP) to releasability in human basophils

Molecular Immunology, 2002-09, Vol.38 (16), p.1323-1327

2002

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15-Hydroxyeicosatetraenoic acid (15-HETE) receptors. Involvement in the 15-HETE-induced stimulation of the cryptic 5-lipoxygenase in PT-18 mast/basophil cells

The Journal of biological chemistry, 1992-11, Vol.267 (33), p.23625-23631

1992

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Role of Lyn Kinase in Leukotriene C4 (LTC4) Secretion from Mast Cells upon High Affinity IgE Receptor (FceRI) Stimulation

Journal of allergy and clinical immunology, 2009-02, Vol.123 (2), p.S119-S119

2009

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Identification of the interaction between the human recombinant histamine releasing factor/translationally controlled tumor protein and elongation factor-1 delta (also known as eElongation factor-1B beta)

Biochimica et biophysica acta, 2004-04, Vol.1688 (3), p.232-236

2004

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Histamine-releasing factor/translationally controlled tumor protein (HRF/TCTP)-lnduced histamine release is enhanced with SHIP-1 krockdown in cultured human mast cell and basophil models

Journal of leukocyte biology, 2008-10, Vol.84 (4), p.1151-1158

2008

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LynB Kinase Plays a Distinct Role in RBL-2H3 Mast Cell Spreading and a Redundant Role with LynA Kinase in FceRI-mediated LTC4 Secretion

Journal of allergy and clinical immunology, 2008-02, Vol.121 (2), p.S48-S48

2008

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A calcium-independent 5-lipoxygenase system in mast/basophil PT-18 cells

Biochimica et biophysica acta, 1990-07, Vol.1045 (2), p.142-146

1990

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ORIGINAL PAPER: Evidence of in vivo basophil activation in chronic idiopathic urticaria

Clinical and experimental allergy, 2006-06, Vol.36 (6), p.770-776

2006

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Constitutive Association of Lyn A and B Kinases with the High Affinity IgE Receptor (FceRI) Positively Regulates Mast Cell Secretion

Journal of allergy and clinical immunology, 2007, Vol.119 (1), p.S287-S287

2007

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Human recombinant Histamine Releasing Factor (HrHRF) Induced Histamine Release (HR) is Associated with SHIP Knockdown (k/d) in both the Cultured Human Mast Cell (MC) and Basophil Models

Journal of allergy and clinical immunology, 2007, Vol.119 (1), p.S313-S313

2007

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Longitudinal Examination of Basophil Functional Phenotypes and Disease Activity in Chronic Idiopathic Urticaria (CIU)

Journal of allergy and clinical immunology, 2007, Vol.119 (1), p.S312-S312

2007

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Regulation of Rat Basophilic Leukemia-2H3 Mast Cell Secretion by a Constitutive Lyn Kinase Interaction with the High Affinity IgE Receptor (Fc{epsilon}RI)

Journal of Immunology, 2005-10, Vol.175 (7), p.4543-4554

2005

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Basophils and mast cells in chronic idiopathic urticaria

Current allergy and asthma reports, 2005-07, Vol.5 (4), p.270-276

2005

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Distinct Basophil Functional Phenotypes in Chronic Idiopathic Urticaria (CIU)

Journal of allergy and clinical immunology, 2006-02, Vol.117 (2), p.S67-S67

2006

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Evidence of in vivo basophil activation in chronic idiopathic urticaria

Differential expression of RNA-binding proteins in bronchial epithelium of stable COPD patients

New concepts in chronic urticaria

Src homology 2 domain–containing inositol 5' phosphatase is negatively associated with histamine release to human recombinant histamine-releasing factor in human basophils

The Unique Domain as the Site on Lyn Kinase for Its Constitutive Association with the High Affinity Receptor for IgE

Inhibition of Cytokine Gene Transcription by the Human Recombinant Histamine-Releasing Factor in Human T Lymphocytes

Basophil FcεRI histamine release parallels expression of Src-homology 2–containing inositol phosphatases in chronic idiopathic urticaria

Interaction between the Unphosphorylated Receptor with High Affinity for IgE and Lyn Kinase

Cultured peripheral blood mast cells from chronic idiopathic urticaria patients spontaneously degranulate upon IgE sensitization: Relationship to expression of Syk and SHIP-2

One Lyn Molecule Is Sufficient to Initiate Phosphorylation of Aggregated High-Affinity IgE Receptors

Simultaneous determination of hydroxyeicosanoid (HETE) binding to cells and its cellular metabolism

Association of the Src homology 2 domain-containing inositol 5′ phosphatase (SHIP) to releasability in human basophils

15-Hydroxyeicosatetraenoic acid (15-HETE) receptors. Involvement in the 15-HETE-induced stimulation of the cryptic 5-lipoxygenase in PT-18 mast/basophil cells

Role of Lyn Kinase in Leukotriene C4 (LTC4) Secretion from Mast Cells upon High Affinity IgE Receptor (FceRI) Stimulation

Identification of the interaction between the human recombinant histamine releasing factor/translationally controlled tumor protein and elongation factor-1 delta (also known as eElongation factor-1B beta)

Histamine-releasing factor/translationally controlled tumor protein (HRF/TCTP)-lnduced histamine release is enhanced with SHIP-1 krockdown in cultured human mast cell and basophil models

LynB Kinase Plays a Distinct Role in RBL-2H3 Mast Cell Spreading and a Redundant Role with LynA Kinase in FceRI-mediated LTC4 Secretion

A calcium-independent 5-lipoxygenase system in mast/basophil PT-18 cells

ORIGINAL PAPER: Evidence of in vivo basophil activation in chronic idiopathic urticaria

Constitutive Association of Lyn A and B Kinases with the High Affinity IgE Receptor (FceRI) Positively Regulates Mast Cell Secretion

Human recombinant Histamine Releasing Factor (HrHRF) Induced Histamine Release (HR) is Associated with SHIP Knockdown (k/d) in both the Cultured Human Mast Cell (MC) and Basophil Models

Longitudinal Examination of Basophil Functional Phenotypes and Disease Activity in Chronic Idiopathic Urticaria (CIU)

Regulation of Rat Basophilic Leukemia-2H3 Mast Cell Secretion by a Constitutive Lyn Kinase Interaction with the High Affinity IgE Receptor (Fc{epsilon}RI)

Basophils and mast cells in chronic idiopathic urticaria

Distinct Basophil Functional Phenotypes in Chronic Idiopathic Urticaria (CIU)

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